H. Hoff

发表

Formation of nitric oxide-derived oxidants by myeloperoxidase in monocytes: pathways for monocyte-mediated protein nitration and lipid peroxidation In vivo.

S. Hazen, E. Podrez, C. Mukhopadhyay, 1999, Circulation research.

Myeloperoxidase-generated reactive nitrogen species convert LDL into an atherogenic form in vitro.

S. Hazen, E. Podrez, H F Hoff, 1999, The Journal of clinical investigation.

Plasma low density lipoprotein accumulation in aortas of hypercholesterolemic swine correlates with modifications in aortic glycosaminoglycan composition.

W D Wagner, H F Hoff, W. Wagner, 1986, Atherosclerosis.

LDL accumulation in the grossly normal human iliac bifurcation and common iliac arteries.

P. Spring, H. Hoff, 1989, Experimental and molecular pathology.

Correlation of apolipoprotein B retention with the structure of atherosclerotic plaques from human aortas.

A. Gotto, J. Gaubatz, J. Titus, 1978, Laboratory investigation; a journal of technical methods and pathology.

Accumulation of lipoproteins containing Apo B in the aorta of cholesterol-fed cynomolgus monkeys.

M. Bond, H. Hoff, 1982, Atherosclerosis.

Quantitation of apolipoprotein B in aortas of hypercholesterolemic swine.

R. Gerrity, H. Hoff, H. Naito, 1983, Laboratory investigation; a journal of technical methods and pathology.

Immunohistochemical localization of apoprotein B in aortas from hyperlipemic swine. Preferential accumulation in lesion-prone areas.

R. Gerrity, D. Feldman, H. Hoff, 1984, Archives of pathology & laboratory medicine.

Apolipoprotein B accumulation and development of foam cell lesions in coronary arteries of hypercholesterolemic swine.

H. Hoff, Y. Yamauchi, 1984, Laboratory investigation; a journal of technical methods and pathology.

Characterization of low density lipoprotein-like particle in the human aorta from grossly normal and atherosclerotic regions.

W. A. Bradley, A. Gotto, J. Gaubatz, 1979, Biochimica et biophysica acta.

Isolation, purification, and characterization of a lipoprotein containing Apo B from the human aorta.

J. Gaubatz, H. Hoff, Henry F. Hoff, 1982, Atherosclerosis.

Comparative studies on plasma low density lipoproteins from pig and man.

A. Gotto, R. Jackson, H. Hoff, 1976, Comparative biochemistry and physiology. B, Comparative biochemistry.

Contrasting Effects on Plasma Lipoproteins of Intravenous Versus Oral Administration of a Triglyceride‐Phospholipid Emulsion

A. Gotto, H. Hoff, R. Segura, 1975, European journal of clinical investigation.

Oxidized low density lipoprotein-induced inhibition of processing of photoreceptor outer segments by RPE.

G. Hoppe, A. Marmorstein, H. Hoff, 2001, Investigative ophthalmology & visual science.

Studies on the pathogenesis of atherosclerosis with experimental model systems

H. Hoff, 2004, Virchows Archiv A.

Studies on the pathogenesis of atherosclerosis with experimental model systems

R. Gottlob, H. Hoff, 2004, Virchows Archiv A.

Studies on the pathogenesis of atherosclerosis with experimental model systems

H. Hoff, 2004, Virchows Archiv A.

Studies on the pathogenesis of atherosclerosis with experimental model systems

R. Gottlob, H. Hoff, 2004, Virchows Archiv A.

A Fine Structure Study of Injury to the Endothelial Cells of the Rabbit Abdominal Aorta by Various Stimuli

R. Gottlob, H. Hoff, 1967, Angiology.

Macrophage Receptors Responsible for Distinct Recognition of Low Density Lipoprotein Containing Pyrrole or Pyridinium Adducts: Models of Oxidized Low Density Lipoprotein

E. Podrez, N. Sheibani, L. Sayre, 2022 .

Protein Adducts of Iso[4]levuglandin E2, a Product of the Isoprostane Pathway, in Oxidized Low Density Lipoprotein*

L. Roberts, K. Kaur, C. Brame, 1999, The Journal of Biological Chemistry.

Macrophage recognition of LDL modified by levuglandin E2, an oxidation product of arachidonic acid.

J. O'Neil, G. Hoppe, R. G. Salomon, 1997, Biochimica et biophysica acta.

Non-conventional modification of low density lipoproteins: chemical models for macrophage recognition of oxidized LDL.

L. Sayre, J. O'Neil, G. Hoppe, 1997, Biochimica et biophysica acta.

Erratum: Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species (Journal of Clinical Investigation (2000) 105 (1095-1108))

S. Hazen, E. Podrez, N. Sheibani, 2000 .

LDL and HDL in Human Arteries — Role in Atherogenesis

J. Gaubatz, H. Hoff, C. Heideman, 1983 .

Lipoproteins containing apolipoprotein A-I extracted from human aortas.

H. Hoff, C. Heideman, 1982, Biochimica et biophysica acta.

Ultrastructural localization of apolipoprotein B in human aortic and coronary atherosclerotic plaques.

J. Gaubatz, H. Hoff, 1977, Experimental and molecular pathology.

Apolipoprotein B (ApoB) Retention in Atherosclerotic Intracranial Arteries

J. Gaubatz, H. Hoff, C. Heideman, 1977, Stroke.

Phospholipids in oxidized LDL not adducted to apoB are recognized by the CD36 scavenger receptor.

E. Podrez, G. Hoppe, H. Hoff, 2003, Free radical biology & medicine.

Correlation in the human aorta of APO B fractions with tissue cholesterol and collagen content.

A. Gotto, J. Gaubatz, H. Hoff, 1979, Atherosclerosis.

Oxidation of LDL: Role in atherogenesis

H. Hoff, J. O'neil, 1991, Klinische Wochenschrift.

Lipoproteins Containing Apo B Extracted from Human Aortas Structure and Function a

H. Hoff, R. Morton, 1985, Annals of the New York Academy of Sciences.

Structural and functional changes in LDL after modification with both 4-hydroxynonenal and malondialdehyde.

H. Hoff, J. O'neil, 1993, Journal of lipid research.

Filipin staining of lipoproteins in polyacrylamide gels: Sensitivity and photobleaching of the fluorophore and its use in a double staining method

G. Smejkal, H. Hoff, 1994, Electrophoresis.

Apo B concentration in the normal human aorta.

A. Gotto, J. Gaubatz, H. Hoff, 1978, Biochemical and biophysical research communications.

Regulation of CD36 by PPARγ: Pro- or Antiatherogenic?

S. Hazen, E. Podrez, A. Gotto, 2002 .

Interaction of phosphatidylcholine and apolipoprotein-alanine: electron microscopic studies.

A. Gotto, J. Morrisett, H. Hoff, 1973, Biochimica et biophysica acta.

Modification of human serum low density lipoprotein by oxidation--characterization and pathophysiological implications.

G. Chisolm, H. Esterbauer, G. Jürgens, 1987, Chemistry and physics of lipids.

Macrophage degradation of LDL extracted from human aortic plaques: effect of isolation conditions.

H. Hoff, J. O'neil, T. Cole, 1991, Experimental and molecular pathology.

Cholesterol esterification in macrophages. Stimulation by lipoproteins containing apo B isolated from human aortas.

B. Clevidence, H. Hoff, R. Morton, 1984, Arteriosclerosis.

Stimulation of Cholesteryl Ester Synthesis in Macrophages by Extracts of Atherosclerotic Human Aortas and Complexes of Albumin/Cholesteryl Esters

M. Brown, J. Goldstein, Michael S. Brown, 1981, Arteriosclerosis.

Uptake of LDL -Sized Particles Extracted from Human Aortic Lesions by Macrophages in Culture

H. Hoff, R. Morton, 1987 .

Reduction in tissue LDL accumulation during coronary artery regression in cynomolgus macaques.

M. Bond, H. Hoff, Y. Yamauchi, 1985, Atherosclerosis.

Low Density Lipoproteins in the Aorta: Relation to Atherosclerosis

J. Gaubatz, H. Hoff, C. Heideman, 1980 .

Localization of low-density lipoproteins in atherosclerotic lesions from human normolipemics employing a purified fluorescent-labeled antibody.

A. Gotto, S. Mao, R. Jackson, 1974, Biochimica et biophysica acta.

Contrasting Effects on Plasma Lipoproteins of Intravenous Versus Oral Administration of a Triglyceride‐Phospholipid Emulsion

A. Gotto, G. Thompson, H. Hoff, 1975 .

Detergent extraction of tightly-bound apoB from extracts of normal aortic intima and plaques.

A. Gotto, J. Gaubatz, H. Hoff, 1978, Experimental and molecular pathology.

Quantitation of apo B in human aortic fatty streaks. A comparison with grossly normal intima and fibrous plaques.

A. Gotto, J. Gaubatz, J. Titus, 1978, Atherosclerosis.

Phospholipids and oxophospholipids in atherosclerotic plaques at different stages of plaque development

A. Kuksis, A. Ravandi, G. Hoppe, 2004, Lipids.

Modification of low density lipoprotein with 4-hydroxynonenal induces uptake by macrophages.

O. Quehenberger, G. Chisolm, H. Esterbauer, 1989, Arteriosclerosis.

Inactivation of cathepsin B by oxidized LDL involves complex formation induced by binding of putative reactive sites exposed at low pH to thiols on the enzyme.

L. Sayre, G. Hoppe, H. Hoff, 1997, Free radical biology & medicine.

Lp(a) and premature mortality during chronic hemodialysis treatment.

H. Hoff, Doris A. Abood, M. Cressman, 1994, Chemistry and physics of lipids.

Modified low density lipoprotein isolated from atherosclerotic lesions does not cause lipid accumulation in aortic smooth muscle cells.

H. Hoff, R. Morton, J. Pepin, 1991, Journal of lipid research.

Quantification of apo[a] and apoB in human atherosclerotic lesions.

H. Hoff, J. Pepin, J. O'neil, 1991, Journal of lipid research.

Products of lipid peroxidation induce missorting of the principal lysosomal protease in retinal pigment epithelium.

J. Sears, G. Hoppe, H. Hoff, 2004, Biochimica et biophysica acta.

Accumulation of oxidized lipid-protein complexes alters phagosome maturation in retinal pigment epithelium

J. Sears, G. Hoppe, H. Hoff, 2004, Cellular and Molecular Life Sciences CMLS.

Phospholipids in oxidized low density lipoproteins perturb the ability of macrophages to degrade internalized macromolecules and reduce intracellular cathepsin B activity.

G. Hoppe, H. Hoff, J. O'neil, 2003, Atherosclerosis.

Inactivation of lysosomal proteases by oxidized low density lipoprotein is partially responsible for its poor degradation by mouse peritoneal macrophages.

G. Hoppe, H. Hoff, J. O'neil, 1994, The Journal of clinical investigation.

Extracts of human atherosclerotic lesions can modify low density lipoproteins leading to enhanced uptake by macrophages.

H. Hoff, J. O'neil, 1988, Atherosclerosis.

Uptake by mouse peritoneal macrophages of large cholesteryl ester-rich particles isolated from human atherosclerotic lesions.

B. Clevidence, H. Hoff, 1987, Experimental and molecular pathology.

Effect of hypoxia on phosphoesterases, oxidative and glycolytic enzymes in the rabbit common carotid artery

H. Hoff, 1970, Histochemie.

A histoenzymatic study of human intracranial atherosclerosis.

H. Hoff, 1972, The American journal of pathology.

Localization of LDL in Arteries: Improvements in Immunofluorescence Procedures a

R. Gerrity, D. Feldman, H. Hoff, 1983, Annals of the New York Academy of Sciences.

Partial e haracterizat ion of I i poprotei ns containing apo [ a ] in human atherosclerotic lesions

H. Hoff, A. Yashiro, '. J. O'Neil, 2002 .

Partial characterization of lipoproteins containing apo[a] in human atherosclerotic lesions.

H. Hoff, A. Yashiro, J. O'neil, 1993, Journal of lipid research.

Apolipoprotein Localization in Human Cranial Arteries, Coronary Arteries, and the Aorta

H. Hoff, 1976, Stroke.

Hydroxynonenal inactivates cathepsin B by forming Michael adducts with active site residues

M. Miyagi, J. Crabb, K. West, 2002, Protein science : a publication of the Protein Society.

Measurement of oxidation in plasma Lp(a) in CAPD patients using a novel ELISA.

E. Podrez, R. G. Salomon, H. Hoff, 1998, Kidney international.

Further studies on the ultrastructural localization of a magnesium dependant neutral ATPase in arteries

H. Hoff, 2004, Histochemie.

AN ELECTRON MICROSCOPY STUDY OF PHOSPHATASE ACTIVITY IN THE ENDOTHELIAL CELLS OF RABBIT AORTA

J. Graf, H. Hoff, 1966, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

Apo-lipoprotein localization in human atherosclerotic arteries.

A. Gotto, R. Jackson, H. Hoff, 1976, Advances in experimental medicine and biology.

Transverse pore gradient gel electrophoresis of lipoprotein [a] using methylenebisacrylamide gradients

G. Smejkal, H. Hoff, 1992, Electrophoresis.

Insoluble complex formation of lipoprotein (a) with low density lipoprotein in the presence of calcium ions.

H. Hoff, A. Yashiro, J. O'neil, 1993, The Journal of biological chemistry.

(Carboxyalkyl)pyrroles in human plasma and oxidized low-density lipoproteins.

R. G. Salomon, H. Hoff, K. Kaur, 1997, Chemical research in toxicology.

Phospholipid Hydroxyalkenals: Biological and Chemical Properties of Specific Oxidized Lipids Present in Atherosclerotic Lesions

G. Hoppe, H. Hoff, Zhiping Wu, 2003, Arteriosclerosis, thrombosis, and vascular biology.

Exchange and mass efflux of cholesterol in macrophages. Evidence for a common mechanism and a role for plasma membrane proteins.

H. Hoff, R. K. Randolph, R. Randolph, 1986, Journal of lipid research.

Immunochemically detectable lipid-free apo(a) in plasma and in human atherosclerotic lesions.

G. Smejkal, H. Hoff, A. Yashiro, 1994, Chemistry and physics of lipids.

Localization of Apo‐Lipoproteins in Human Carotid Artery Plaques

G. Noon, J. Meyer, J. Meyer, 1975, Stroke.

Differential interference contrast microscopy of human atherosclerotic lesions.

H. Hoff, 1973, The American journal of pathology.

Levuglandin E2-protein adducts in human plasma and vasculature.

M. Smith, V. Monnier, R. G. Salomon, 1997, Chemical research in toxicology.

Macrophage uptake of cholesterol-containing particles derived from LDL and isolated from atherosclerotic lesions.

H. Hoff, J. Pepin, J. O'neil, 1990, European heart journal.

Macrophage uptake of low-density lipoprotein modified by 4-hydroxynonenal. An ultrastructural study.

H. Hoff, T. Cole, 1991, Laboratory investigation; a journal of technical methods and pathology.

Apolipoprotein B localization in coronary atherosclerotic plaques from cynomolgus monkeys.

M. Bond, H. Hoff, 1983, Artery.

Oxidation products of cholesteryl linoleate are resistant to hydrolysis in macrophages, form complexes with proteins, and are present in human atherosclerotic lesions.

A. Kuksis, A. Ravandi, G. Hoppe, 1997, Journal of lipid research.

An electron microscopy study of fat uptake by endothelial cells of doubly-ligated carotid artery segments

R. Gottlob, H. Hoff, 1968, Experientia.

A comparison of the fine-structural localization of nucleoside phosphatase activity in large intracranial blood vessels and the thoracic aorta of rabbits

H. Hoff, 1968, Histochemie.

Human intracranial atherosclerosis

H. Hoff, 1973, Virchows Archiv A.

Aggregation as well as chemical modification of LDL during oxidation is responsible for poor processing in macrophages.

H. Hoff, D. Armstrong, J. O'neil, 1993, Journal of lipid research.

A modified alcian blue-periodic acid Schiff stain for epoxy-embedded atherosclerotic arteries.

H. Hoff, C. Rayburn, 1974, Stain technology.

Identification of a Novel Family of Oxidized Phospholipids That Serve as Ligands for the Macrophage Scavenger Receptor CD36*

Yijun Deng, Paul L. Fox, Stanley L. Hazen, 2002, The Journal of Biological Chemistry.

Scavenger Receptors Class A-I/II and CD36 Are the Principal Receptors Responsible for the Uptake of Modified Low Density Lipoprotein Leading to Lipid Loading in Macrophages*

K. Moore, M. Freeman, E. Podrez, 2002, The Journal of Biological Chemistry.

Lesion-derived low density lipoprotein and oxidized low density lipoprotein share a lability for aggregation, leading to enhanced macrophage degradation.

H F Hoff, J O'Neil, H. Hoff, 1991, Arteriosclerosis and thrombosis : a journal of vascular biology.

Targeted disruption of the class B scavenger receptor CD36 protects against atherosclerotic lesion development in mice.

S. Hazen, E. Podrez, R. Silverstein, 2000, The Journal of clinical investigation.

Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species.

S. Hazen, E. Podrez, N. Sheibani, 2000, The Journal of clinical investigation.

Lipoprotein(a) Is an Independent Risk Factor for Cardiovascular Disease in Hemodialysis Patients

R. Heyka, H. Hoff, E. Paganini, 1992, Circulation.

A Novel Family of Atherogenic Oxidized Phospholipids Promotes Macrophage Foam Cell Formation via the Scavenger Receptor CD36 and Is Enriched in Atherosclerotic Lesions*

S. Hazen, E. Podrez, Renliang Zhang, 2002, Journal of Biological Chemistry.

Oxidation of low density lipoprotein leads to particle aggregation and altered macrophage recognition.

T. E. Whitaker, H F Hoff, J O'Neil, 1992, The Journal of biological chemistry.

Structure of cholesterol-containing particles accumulating in atherosclerotic lesions and the mechanisms of their derivation

G. Hoppe, H. Hoff, 1995, Current Opinion in Lipidology.

Ultrastructural localization of plasma lipoproteins in human intracranial arteries

J. Gaubatz, H. Hoff, 1975, Virchows Archiv A.

The interaction of apolipoprotein-serine with phosphatidylcholine.

A. Gotto, H. Pownall, J. Segrest, 1974, The Journal of biological chemistry.

Serum Lp(a) level as a predictor of vein graft stenosis after coronary artery bypass surgery in patients.

G. Beck, B. Lytle, H. Hoff, 1988, Circulation.

Apolipoprotein B Retention in the Grossly Normal and Atherosclerotic Human Aorta

A. Gotto, J. Gaubatz, H. Hoff, 1977, Circulation research.

Lipoproteins in atherosclerotic lesions. Localization by immunofluorescence of apo-low density lipoproteins in human atherosclerotic arteries from normal and hyperlipoproteinemics.

M. Debakey, A. Gotto, J. Lie, 1975, Archives of pathology.

A Study of the Relation Between Endothelial Silver Lines, Medial Transverse Silver Lines, and the Ultrastructural Morphology of Blood Vessels

R. Gottlob, H. Hoff, 1967 .

Ultrastructural changes of large rabbit blood vessels following mild mechanical trauma

R. Gottlob, H. Hoff, 2004, Virchows Archiv A.

Quantification of Apolipoprotein B in Grossly Normal Human Aorta

A. Gotto, J. Gaubatz, R. Jackson, 1977, Circulation research.

HNE-derived 2-pentylpyrroles are generated during oxidation of LDL, are more prevalent in blood plasma from patients with renal disease or atherosclerosis, and are present in atherosclerotic plaques.

E. Podrez, L. Sayre, R. G. Salomon, 2000, Chemical research in toxicology.

Localization Patterns of Plasma Apolipoproteins in Human Atherosclerotic Lesions

R. Bayardo, A. Gotto, R. Jackson, 1975, Circulation research.

Oxidation of low-density lipoproteins produces levuglandin-protein adducts.

R. G. Salomon, H. Hoff, G. Subbanagounder, 1997, Chemical research in toxicology.

The uptake of colloidal particles by aortic endothelial cells

R. Gottlob, H. Hoff, 1967, Naturwissenschaften.

A low density lipoprotein-sized particle isolated from human atherosclerotic lesions is internalized by macrophages via a non-scavenger-receptor mechanism.

H F Hoff, H. Hoff, R. Morton, 1986, Journal of lipid research.

APO low density lipoprotein localization. Intracranial and extracranial atherosclerotic lesions from human normolipoproteinemics and hyperlipoproteinemics.

J. Gaubatz, H. Hoff, C. Heideman, 1975, Archives of neurology.