M. Gray

发表

Cardioprotection from ischemia by a brief exposure to physiological levels of ethanol: role of epsilon protein kinase C.

D. Mochly‐Rosen, Che-Hong Chen, C. Chen, 1999, Proceedings of the National Academy of Sciences of the United States of America.

Sustained in vivo cardiac protection by a rationally designed peptide that causes ɛ protein kinase C translocation

G. Dorn, Guangyu Wu, J. Lorenz, 1999 .

MnSOD in mouse heart: acute responses to ischemic preconditioning and ischemia-reperfusion injury.

G. Cecchini, J. Karliner, Zhu-qiu Jin, 2005, American journal of physiology. Heart and circulatory physiology.

preconditioning and ischemia-reperfusion injury MnSOD in mouse heart: acute responses to ischemic

G. Cecchini, J. Karliner, Zhu-qiu Jin, 2016 .

Angiotensin II stimulates cardiac myocyte hypertrophy via paracrine release of TGF-beta 1 and endothelin-1 from fibroblasts.

C. Long, J. Karliner, H. T. Li, 1998, Cardiovascular research.

69 – Signaling Pathways in the Heart and Moderate Alcohol Intake

M. Gray, M. O. Gray, 2004 .

Moderate alcohol consumption induces sustained cardiac protection by activating PKC-ε and Akt

J. Karliner, M. Gray, Hui-zhong Zhou, 2002 .

Cross Talk Between Angiotensin AT1 and α1-Adrenergic Receptors : Angiotensin II Downregulates α1a-Adrenergic Receptor Subtype mRNA and Density in Neonatal Rat Cardiac Myocytes

C. Long, J. Karliner, N. Honbo, 1997 .

Neonatal mouse cardiac myocytes exhibit cardioprotection induced by hypoxic and pharmacologic preconditioning and by transgenic overexpression of human Cu/Zn superoxide dismutase.

C. Epstein, J. Karliner, Y. Lau, 2000, Journal of molecular and cellular cardiology.

Signals from type 1 sphingosine 1-phosphate receptors enhance adult mouse cardiac myocyte survival during hypoxia.

E. Goetzl, J. Karliner, K. Chatterjee, 2007, American journal of physiology. Heart and circulatory physiology.

Sustained in vivo cardiac protection by a rationally designed peptide that causes epsilon protein kinase C translocation.

G. Dorn, J. Lorenz, D. Mochly‐Rosen, 1999, Proceedings of the National Academy of Sciences of the United States of America.

Angiotensin II stimulates cardiac myocyte hypertrophy via paracrine release of TGF-b and endothelin-1 from fibroblasts 1

C. Long, J. Karliner, M. Gray, 1998 .

Transgenic MMP-2 expression induces latent cardiac mitochondrial dysfunction.

A. Baker, G. Cecchini, U. Simonis, 2007, Biochemical and biophysical research communications.

A Protein Kinase C Translocation Inhibitor as an Isozyme-selective Antagonist of Cardiac Function*

D. Mochly‐Rosen, Che-Hong Chen, M. Gray, 1996, The Journal of Biological Chemistry.

Cardioprotection mediated by sphingosine-1-phosphate and ganglioside GM-1 in wild-type and PKCε knockout mouse hearts

E. Goetzl, D. Mochly‐Rosen, J. Karliner, 2002 .

Endothelin-1 stimulates cardiac fibroblast proliferation through activation of protein kinase C.

J. Karliner, L. Piacentini, N. Honbo, 2000, Journal of molecular and cellular cardiology.

The lysophospholipids sphingosine-1-phosphate and lysophosphatidic acid enhance survival during hypoxia in neonatal rat cardiac myocytes.

E. Goetzl, J. Karliner, N. Honbo, 2001, Journal of molecular and cellular cardiology.

Preservation of Base-line Hemodynamic Function and Loss of Inducible Cardioprotection in Adult Mice Lacking Protein Kinase Cϵ*

D. Mochly‐Rosen, R. Messing, M. Gray, 2004, Journal of Biological Chemistry.

Chronic ethanol-induced myocardial protection requires activation of mitochondrial K(ATP) channels.

M. Gray, P. Zhu, H. Z. Zhou, 2000, Journal of molecular and cellular cardiology.

A selective epsilon-protein kinase C antagonist inhibits protection of cardiac myocytes from hypoxia-induced cell death.

D. Mochly‐Rosen, J. Karliner, M. Gray, 1997, The Journal of biological chemistry.

Effect of anoxia/reperfusion on the reversible active/de-active transition of NADH-ubiquinone oxidoreductase (complex I) in rat heart.

G. Cecchini, J. Karliner, Y. Sher, 2002, Biochimica et biophysica acta.

Alcohol in moderation, cardioprotection, and neuroprotection: epidemiological considerations and mechanistic studies.

E. Neafsey, D. Das, R. Korthuis, 2009, Alcoholism, clinical and experimental research.

An improved permeabilization protocol for the introduction of peptides into cardiac myocytes. Application to protein kinase C research.

D. Mochly‐Rosen, J. Karliner, Che-Hong Chen, 1996, Circulation research.

Translocation Inhibitors Define Specificity of Protein Kinase C Isoenzymes in Pancreatic β-Cells*

D. Mochly‐Rosen, E. Cerasi, D. Ron, 1997, The Journal of Biological Chemistry.

Moderate alcohol consumption induces sustained cardiac protection by activating PKC-epsilon and Akt.

J. Karliner, M. Gray, Hui-zhong Zhou, 2002, American journal of physiology. Heart and circulatory physiology.

Poly(ADP-ribose) polymerase-1 hyperactivation and impairment of mitochondrial respiratory chain complex I function in reperfused mouse hearts.

R. Swanson, G. Cecchini, U. Simonis, 2006, American journal of physiology. Heart and circulatory physiology.

Cross talk between angiotensin AT1 and alpha 1-adrenergic receptors: angiotensin II downregulates alpha 1a-adrenergic receptor subtype mRNA and density in neonatal rat cardiac myocytes.

C. Long, J. Karliner, N. Honbo, 1997, Circulation Research.

A Selective ε-Protein Kinase C Antagonist Inhibits Protection of Cardiac Myocytes from Hypoxia-induced Cell Death*

D. Mochly‐Rosen, J. Karliner, M. Gray, 1997, The Journal of Biological Chemistry.