S. Stetson

发表

Decreased expression of tumor necrosis factor-alpha in failing human myocardium after mechanical circulatory support : A potential mechanism for cardiac recovery.

G. Noon, M. Entman, O. Frazier, 1999, Circulation.

Regression of fibrosis and hypertrophy in failing myocardium following mechanical circulatory support.

G. Noon, O. Frazier, B. Bruckner, 2001, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

Dobutamine stress echocardiography predicts myocardial improvement in patients supported by left ventricular assist devices (LVADs): hemodynamic and histologic evidence of improvement before LVAD explantation.

T. Myers, O. Frazier, B. Radovancević, 2003, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

New strategies for the management of acute decompensated heart failure.

M. Loebe, G. Noon, G. Torre-Amione, 2001, Current opinion in cardiology.

Cellular and hemodynamics responses of failing myocardium to continuous flow mechanical circulatory support using the DeBakey-Noon left ventricular assist device: a comparative analysis with pulsatile-type devices.

M. Loebe, G. Noon, Carlos Rivas-Gotz, 2005, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

The Effect of Cytokines on Cardiac Allograft Function: Tumor Necrosis Factor α a Mediator of Chronic Injury

W. Mazur, G. Torre-Amione, M. Koerner, 2001, Heart Failure Reviews.

Mechanical Circulatory Support : A Potential Mechanism for Cardiac Recovery in Failing Human Myocardium After α Decreased Expression of Tumor Necrosis Factor

Branislav, G. Noon, M. Entman, 1999 .

The role of inflammation in the pathogenesis of heart failure

J. Farmer, G. Torre-Amione, M. Koerner, 2002, Current cardiology reports.

Evidence of improved right ventricular structure after LVAD support in patients with end-stage cardiomyopathy.

M. Loebe, G. Noon, M. Entman, 2004, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

Degree of cardiac fibrosis and hypertrophy at time of implantation predicts myocardial improvement during left ventricular assist device support.

M. Loebe, G. Noon, M. Entman, 2004, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

The implications for cardiac recovery of left ventricular assist device support on myocardial collagen content.

J. Farmer, G. Noon, M. Entman, 2000, American journal of surgery.

Molecular remodelling of dystrophin in patients with end-stage cardiomyopathies and reversal in patients on assistance-device therapy

J. Towbin, G. Noon, M. Entman, 2002, The Lancet.

olecular Normalization of Dystrophin n the Failing Left and Right Ventricle of atients Treated With Either Pulsatile or ontinuous Flow-Type Ventricular Assist Devices

J. Towbin, M. Entman, N. Bowles, 2016 .

Molecular normalization of dystrophin in the failing left and right ventricle of patients treated with either pulsatile or continuous flow-type ventricular assist devices.

J. Towbin, G. Noon, M. Entman, 2004, Journal of the American College of Cardiology.

Tumor necrosis factor-α in the failing human heart: Does it play a role in disease progression?

K. Youker, G. Torre-Amione, S. Stetson, 1999, Zeitschrift für Kardiologie.

A Mediator of Disease Progression in the Failing Human Heart

J. Farmer, G. Torre-Amione, S. Stetson, 2015 .

The role of tumor necrosis factor alpha blockade in the treatment of congestive heart failure.

J. Farmer, G. Torre-Amione, M. Koerner, 2002, Congestive heart failure.

Tumor Necrosis Factor-α: A Mediator of Disease Progression in the Failing Human Heart

J. Farmer, G. Torre-Amione, S. Stetson, 1999 .

Managing heart failure with immunomodulatory agents.

J. Farmer, G. Torre-Amione, M. Koerner, 2001, Cardiology clinics.

Simvastatin decreases myocardial tumor necrosis factor α content in heart transplant recipients

J. Farmer, G. Noon, G. Torre-Amione, 2004 .

Cardiac Hypertrophy After Transplantation Is Associated With Persistent Expression of Tumor Necrosis Factor-&agr;

M. Quiñones, J. Farmer, G. Noon, 2001, Circulation.

Results Systemic Hypertension Does Not Play a Primary Role in the Development of Cardiac Allograft Hypertrophy

M. Quiñones, J. Farmer, G. Noon, 2001 .

Tumor necrosis factor-alpha: a mediator of disease progression in the failing human heart.

J. Farmer, G. Torre-Amione, S. Stetson, 1999, Chest.

Improved myocardial structure following LVAD support: effect of unloading on dystrophin expression.

J. Towbin, M. Vatta, N. Bowles, 2001, The Journal of Heart and Lung Transplantation.

Decreased Expression of Tumor Necrosis Factor-&agr; and Regression of Hypertrophy After Nonsurgical Septal Reduction Therapy for Patients With Hypertrophic Obstructive Cardiomyopathy

M. Entman, S. Nagueh, N. Lakkis, 2001, Circulation.

The Use of Clomiphene Citrate in Natural Cycle IVF

S. Stetson, 2015 .

Simvastatin decreases myocardial tumor necrosis factor alpha content in heart transplant recipients.

J. Farmer, G. Noon, G. Torre-Amione, 2005, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.