Hai-lan Yao

Coxsackievirus B3 induces crosstalk between autophagy and apoptosis to benefit its release after replicating in autophagosomes through a mechanism involving caspase cleavage of autophagy-related proteins.

Le Xin, Zonghui Xiao, Xiao-Lin Ma, 2014, Infection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases.

Coxsackievirus B3 induces autophagy in HeLa cells via the AMPK/MEK/ERK and Ras/Raf/MEK/ERK signaling pathways.

Le Xin, Zonghui Xiao, Xiao-Lin Ma, 2015, Infection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases.

Coxsackievirus B3 infection induces changes in the expression of numerous piRNAs

Juan Song, Qinqin Song, Jun Han, 2019, Archives of Virology.

Construction of miRNA-target networks using microRNA profiles of CVB3-infected HeLa cells

Juan Song, Qinqin Song, Mi Liu, 2019, Scientific Reports.

Gene expression analysis during recovery process indicates the mechanism for innate immune injury and repair from Coxsackievirus B3-induced myocarditis.

Juan Song, Qinqin Song, Jun Han, 2016, Virus research.

Coxsackievirus B3 2A protease promotes encephalomyocarditis virus replication.

Juan Song, Qinqin Song, Xiaonuan Luo, 2015, Virus research.

Single‐cell transcriptomic atlas reveals distinct immunological responses between COVID‐19 vaccine and natural SARS‐CoV‐2 infection

Chongzhen Wang, Jieqiong Li, Xiaolan Huang, 2022, Journal of medical virology.

Engineered coxsackievirus B3 containing multiple organ-specific miRNA targets showed attenuated viral tropism and protective immunity.

Zhuo Liu, Jianxin Wu, Wei Wang, 2022, Infection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases.