K. B. Grandinetti

发表

Loss of one HuD allele on chromosome #1p selects for amplification of the N-myc proto-oncogene in human neuroblastoma cells

R. Ross, B. Spengler, J. L. Biedler, 2006, Oncogene.

MicroRNAs define distinct human neuroblastoma cell phenotypes and regulate their differentiation and tumorigenicity

R. Ross, B. Spengler, K. B. Grandinetti, 2014, BMC Cancer.

Specific requirement of the chromatin modifier mSin3B in cell cycle exit and cellular differentiation

Gerald C. Chu, R. DePinho, G. Chu, 2008, Proceedings of the National Academy of Sciences.

Sin3B expression is required for cellular senescence and is up-regulated upon oncogenic stress.

P. Jelinic, S. Logan, N. Bardeesy, 2009, Cancer research.

Sin3B: An essential regulator of chromatin modifications at E2F target promoters during cell cycle withdrawal

G. David, K. B. Grandinetti, 2008, Cell cycle.

Dual ALK and CDK4/6 Inhibition Demonstrates Synergy against Neuroblastoma

G. Caponigro, Nanxin Li, Jennifer L. Harris, 2016, Clinical Cancer Research.

Overexpression of TRIB2 in human lung cancers contributes to tumorigenesis through downregulation of C/EBPα

D. Tenen, V. Reddy, E. C. Peters, 2011, Oncogene.

FGFR2 Promotes Breast Tumorigenicity through Maintenance of Breast Tumor-Initiating Cells

G. Bonamy, A. Orth, C. García-echeverría, 2013, PloS one.

Glycogen synthase kinase 3 inhibitors induce the canonical WNT/β-catenin pathway to suppress growth and self-renewal in embryonal rhabdomyosarcoma

C. Keller, D. Langenau, M. DeRan, 2014, Proceedings of the National Academy of Sciences.

Sin3B expression is required for cellular senescence and is up-regulated upon oncogenic stress.

P. Jelinic, S. Logan, N. Bardeesy, 2009, Cancer research.