K. B. Bourloud

发表

Hypermethylation‐mediated regulation of CD44 gene expression in human neuroblastoma

P. Yan, N. Gross, Annick Mühlethaler, 2003, Genes, chromosomes & cancer.

Wild-type ALK and activating ALK-R1275Q and ALK-F1174L mutations upregulate Myc and initiate tumor formation in murine neural crest progenitor cells

O. Delattre, L. Sommer, M. Peuchmaur, 2014, Oncotarget.

Loss of caspase-8 expression in highly malignant human neuroblastoma cells correlates with resistance to tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis.

J. Tschopp, J. Bodmer, J. Tschopp, 2000, Cancer research.

Complex molecular mechanisms cooperate to mediate histone deacetylase inhibitors anti-tumour activity in neuroblastoma cells

J. Joseph, R. Meier, N. Gross, 2008, Molecular Cancer.

Persistent inhibition of FLIPL expression by lentiviral small hairpin RNA delivery restores death-receptor-induced apoptosis in neuroblastoma cells

M. Popovic, J. Joseph, R. Meier, 2006, Apoptosis.

The noradrenergic profile of plasma metanephrine in neuroblastoma patients is reproduced in xenograft mice models and arise from PNMT downregulation

K. Abid, J. Schoumans, M. Popovic, 2021, Oncotarget.

Advances in Brief Loss of Caspase-8 Expression in Highly Malignant Human Neuroblastoma Cells Correlates with Resistance to Tumor Necrosis Factor-related Apoptosis-inducing Ligand-induced Apoptosis 1

J. Tschopp, J. Bodmer, N. Gross, 2000 .

Loss of caspase-8 expression in neuroblastoma is related to malignancy and resistance to TRAIL-induced apoptosis.

J. Bodmer, J. Tschopp, N. Gross, 2000, Medical and pediatric oncology.

Individual caspase-10 isoforms play distinct and opposing roles in the initiation of death receptor-mediated tumour cell apoptosis

M. Thome, N. Gross, K. B. Bourloud, 2011, Cell Death and Disease.

Drug-mediated sensitization to TRAIL-induced apoptosis in caspase-8-complemented neuroblastoma cells proceeds via activation of intrinsic and extrinsic pathways and caspase-dependent cleavage of XIAP, Bcl-xL and RIP

J. Joseph, N. Gross, K. B. Bourloud, 2004, Oncogene.

Doxorubicin-induced death in neuroblastoma does not involve death receptors in S-type cells and is caspase-independent in N-type cells

J. Bodmer, P. Yan, N. Gross, 2002, Oncogene.

TWIST1 expression is associated with high-risk neuroblastoma and promotes primary and metastatic tumor growth

Nicolò Riggi, V. Praz, J. Scoazec, 2021, Communications Biology.

Metastatic Tumor Growth

V. Praz, H. Sartelet, J. Joseph, 2021 .

Histone deacetylase inhibitors strongly sensitise neuroblastoma cells to TRAIL-induced apoptosis by a caspases-dependent increase of the pro- to anti-apoptotic proteins ratio

J. Joseph, R. Meier, N. Gross, 2006, BMC Cancer.

The Chemokine Receptor CXCR4 Strongly Promotes Neuroblastoma Primary Tumour and Metastatic Growth, but not Invasion

Nicole Gross, Curzio Ruegg, Gilles Vassal, 2007, PloS one.

Molecular cytogenetic characterization of doxorubicin‐resistant neuroblastoma cell lines: Evidence that acquired multidrug resistance results from a unique large amplification of the 7q21 region

O. Delattre, J. Joseph, D. Martinet, 2006, Genes, chromosomes & cancer.

MYCN-related suppression of functional CD44 expression enhances tumorigenic properties of human neuroblastoma cells.

N. Gross, K. B. Bourloud, C. B. Brognara, 2000, Experimental cell research.

Complex molecular mechanisms cooperate to mediate histone deacetylase inhibitors anti-tumour activity in neuroblastoma cells

J. Joseph, R. Meier, N. Gross, 2008, Molecular Cancer.