S. Banerjee

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Caspase-1 activation of IL-1beta and IL-18 are essential for Shigella flexneri-induced inflammation.

S. Akira, P. Sansonetti, K. Takeda, 2000, Immunity.

Caspase-1 processes IFN-gamma-inducing factor and regulates LPS-induced IFN-gamma production.

R. Kamen, T. Ghayur, D. Butler, 1997, Nature.

Shigella-induced Apoptosis Is Dependent on Caspase-1 Which Binds to IpaB*

Junying Yuan, P. Sansonetti, R. Flavell, 1998, The Journal of Biological Chemistry.

Restricted Expression of T Cell Receptor Vβ and Lymphokine Genes in Arthritic Joints of a TCR VβA (H-2Q) Mouse Strain-BUB/BnJ-with Collagen-Induced Arthritis

M. Sy, S. Banerjee, T. Haqqi, 1995 .

Mice deficient in IL-1 beta-converting enzyme are defective in production of mature IL-1 beta and resistant to endotoxic shock.

M. Paskind, J. Salfeld, S. Banerjee, 1995, Cell.

IL-17A is Essential for Cell Activation and Inflammatory Gene Circuits in Psoriasis

P. Haslett, Artemis, G. Cameron, 2012 .

The induction of arthritis in mice by the cartilage proteoglycan aggrecan: roles of CD4+ and CD8+ T cells.

A. Poole, S. Banerjee, C. Webber, 1992, Cellular immunology.

Restricted heterogeneity in T-cell antigen receptor V beta gene usage in the lymph nodes and arthritic joints of mice.

C. David, S. Banerjee, T. Haqqi, 1992, Proceedings of the National Academy of Sciences of the United States of America.

Autoimmunity to cartilage link protein in patients with rheumatoid arthritis and ankylosing spondylitis.

J. Esdaile, A. Poole, M. Fitzcharles, 1998, The Journal of rheumatology.

Immunosuppression of collagen-induced arthritis in mice with an anti-IL-2 receptor antibody.

H. Luthra, C. David, S. Banerjee, 1988, Journal of immunology.

Cellular immunity to the G1 domain of cartilage proteoglycan aggrecan is enhanced in patients with rheumatoid arthritis but only after removal of keratan sulfate.

J. Esdaile, A. Poole, M. Fitzcharles, 1998, Arthritis and rheumatism.

Immunity to the G1 globular domain of the cartilage proteoglycan aggrecan can induce inflammatory erosive polyarthritis and spondylitis in BALB/c mice but immunity to G1 is inhibited by covalently bound keratan sulfate in vitro and in vivo.

A. Poole, L. Rosenberg, S. Banerjee, 1996, The Journal of clinical investigation.

Suppression of renal disease and arthritis, and prolongation of survival in MRL-lpr mice treated with an extract of Tripterygium wilfordii Hook f.

S. Banerjee, W. Gu, S. Brandwein, 1992, Arthritis and rheumatism.

Characterization of mice deficient in interleukin-1 beta converting enzyme.

S. Banerjee, H. Allen, T. Seshadri, 1997, Journal of cellular biochemistry.

RIII S/J (H-2r). An inbred mouse strain with a massive deletion of T cell receptor V beta genes

C. David, S. Banerjee, T. Haqqi, 1989, The Journal of experimental medicine.

Inhibition of type II collagen induced arthritis in mice by an immunosuppressive extract of Tripterygium wilfordii Hook f.

S. Banerjee, W. Gu, S. Brandwein, 1992, The Journal of rheumatology.

MEP1A allele for meprin A metalloprotease is a susceptibility gene for inflammatory bowel disease

T. Ahmad, D. Jewell, F. Seibold, 2009, Mucosal Immunology.

Serum IgG anti-native type II collagen antibodies in rheumatoid arthritis: association with HLA DR4 and lack of clinical correlation.

W. O'Fallon, H. Luthra, S. Moore, 1988, Clinical and experimental rheumatology.

Immunity to cartilage proteoglycans.

A. Poole, S. Banerjee, 1992, The Journal of rheumatology. Supplement.

Possible role of V beta T cell receptor genes in susceptibility to collagen-induced arthritis in mice

H. Luthra, C. David, S. Banerjee, 1988, The Journal of experimental medicine.

Influence of complement C5 and V beta T cell receptor mutations on susceptibility to collagen-induced arthritis in mice.

H. Luthra, C. David, S. Banerjee, 1989, Journal of immunology.

T cell receptor genes and disease susceptibility.

C. David, S. Banerjee, 1989, Arthritis and rheumatism.

Role of MHC Genes and T Cell Receptor Genes in Collagen Induced Arthritis in Mice

H. Luthra, C. David, S. Banerjee, 1987 .

Induction of Host Protective Th1 Immune Response by Chemokines in Leishmania donovani‐infected BALB/c Mice

R. Dey, S. Bhattacharjee, S. Banerjee, 2007, Scandinavian journal of immunology.

BUB/BnJ (H-2q) is a TCR deletion mutant mouse strain (TCR V beta a, KJ16-) that is susceptible to type II collagen-induced arthritis.

S. Banerjee, T. Haqqi, X. Qu, 1994, Journal of Immunology.

Limited Heterogeneity in T‐Cell Receptor Vβ Chain Gene Expression in Arthritic Joints of BUB/BnJ (H‐2q) Mice—A T‐Cell Receptor Vβa Strain a

S. Banerjee, T. Haqqi, X. Qu, 1995 .

Role of Mls-1 locus and clonal deletion of T cells in susceptibility to collagen-induced arthritis in mice.

H. Luthra, C. David, S. Banerjee, 1991, Journal of immunology.

MHC class II A alpha and E alpha molecules determine the clonal deletion of V beta 6+ T cells. Studies with recombinant and transgenic mice.

C. David, S. Banerjee, G. Anderson, 1989, Journal of immunology.