H. Gan

发表

A Mechanism of Virulence: Virulent Mycobacterium tuberculosis Strain H37Rv, but Not Attenuated H37Ra, Causes Significant Mitochondrial Inner Membrane Disruption in Macrophages Leading to Necrosis1

Minjian Chen, Minjian Chen, H. Gan, 2006, The Journal of Immunology.

Lysosome-Mediated Plasma Membrane Repair Is Dependent on the Small GTPase Arl8b and Determines Cell Death Type in Mycobacterium tuberculosis Infection

M. Brenner, Xavier Michelet, A. Tuli, 2018, The Journal of Immunology.

Mycobacterium tuberculosis Arl 8 b and Determines Cell Death Type in Repair Is Dependent on the Small GTPase Lysosome-Mediated Plasma Membrane

M. Brenner, Xavier Michelet, A. Tuli, 2018 .

Enhancement of antimycobacterial activity of macrophages by stabilization of inner mitochondrial membrane potential.

Lei Duan, H. Kornfeld, H. Gan, 2005, The Journal of infectious diseases.

Mycobacterium tuberculosis blocks annexin-1 crosslinking and thus apoptotic envelope completion on infected cells to maintain virulence

Minjian Chen, H. Kornfeld, H. Gan, 2008, Nature Immunology.

Lipid mediators in innate immunity against tuberculosis: opposing roles of PGE2 and LXA4 in the induction of macrophage death

Minjian Chen, C. Serhan, Song Hong, 2008, The Journal of experimental medicine.

Apoptosis is an innate defense function of macrophages against Mycobacterium tuberculosis

Xiaomin Zhao, S. Behar, H. Gan, 2011, Mucosal Immunology.

Critical Role of Mitochondrial Damage in Determining Outcome of Macrophage Infection with Mycobacterium tuberculosis1

D. Golan, Lei Duan, H. Gan, 2002, The Journal of Immunology.

The prostaglandin E2 receptor EP4 is integral to a positive feedback loop for prostaglandin E2 production in human macrophages infected with Mycobacterium tuberculosis

Xiaomin Zhao, S. Koyasu, H. Gan, 2013, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Cytosolic Phospholipase A2 Participates with TNF-a in the Induction of Apoptosis of Human Macrophages Infected with Mycobacterium tuberculosisH37Ra

J. Arm, H. Gan, H. Remold, 2001 .

Interleukin 10 produced by macrophages inoculated with Mycobacterium avium attenuates mycobacteria-induced apoptosis by reduction of TNF-alpha activity.

H. Gan, H. Remold, M. K. Balcewicz-Sablinska, 1999, The Journal of infectious diseases.

Human macrophages acquire a hyporesponsive state of tumor necrosis factor alpha production in response to successive Mycobacterium avium serovar 4 stimulation

H. Gan, H. Remold, G. Newman, 1995, Infection and immunity.

Interleukin-6 expression in primary macrophages infected with human immunodeficiency virus-1 (HIV-1).

B. F. Hall, J. Mellors, C. Ruef, 1991, AIDS research and human retroviruses.

Bcl-xL mediates RIPK3-dependent necrosis in M. tuberculosis-infected macrophages

Xiaomin Zhao, F. Tzelepis, Seung-Yeol Park, 2017, Mucosal Immunology.

NecrosisDisruption in Macrophages Leading to Significant Mitochondrial Inner Membrane but Not Attenuated H37Ra, Causes Strain H37Rv, Mycobacterium tuberculosis A Mechanism of Virulence: Virulent

Minjian Chen, H. Gan, H. Remold, 2006 .

Survival of human macrophages infected with Mycobacterium avium intracellulare correlates with increased production of tumor necrosis factor-alpha and IL-6.

P. McCarthy, H. Gan, H. Remold, 1991, Journal of immunology.

Mycobacterium tuberculosis evades macrophage defenses by inhibiting plasma membrane repair

S. Fortune, Minjian Chen, T. Hickman, 2009, Nature Immunology.

Cytosolic Phospholipase A2 Participates with TNF-α in the Induction of Apoptosis of Human Macrophages Infected with Mycobacterium tuberculosis H37Ra1

J. Arm, H. Gan, H. Remold, 2001, The Journal of Immunology.

Plasminogen activator inhibitor type 2 prevents programmed cell death of human macrophages infected with Mycobacterium avium, serovar 4.

H. Gan, H. Remold, G. Newman, 1995, Journal of immunology.

Concurrent infection of human macrophages with HIV-1 and Mycobacterium avium results in decreased cell viability, increased M. avium multiplication and altered cytokine production.

R. Rose, M. Newman, H. Gan, 1993, Journal of immunology.

TNF-alpha response of human monocyte-derived macrophages to Mycobacterium avium, serovar 4, is of brief duration and protein kinase C dependent.

P. McCarthy, H. Gan, H. Remold, 1993, Journal of immunology.

Mycobacterium tuberculosis blocks annexin-1 crosslinking and thus apoptotic envelope completion on infected cells to maintain virulence

Minjian Chen, H. Kornfeld, H. Gan, 2008, Nature Immunology.