P. Barrionuevo

发表

Bacterial RNA Contributes to the Down-Modulation of MHC-II Expression on Monocytes/Macrophages Diminishing CD4+ T Cell Responses

S. Oliveira, F. V. Marinho, L. Balboa, 2019, Front. Immunol..

B. abortus RNA is the component involved in the down-modulation of MHC-I expression on human monocytes via TLR8 and the EGFR pathway

G. Fernández, S. Oliveira, F. V. Marinho, 2017, PLoS pathogens.

Brucella abortus Invasion of Synoviocytes Inhibits Apoptosis and Induces Bone Resorption through RANKL Expression

M. Delpino, C. A. Fossati, P. C. Arriola Benitez, 2013, Infection and Immunity.

Brucella abortus induces intracellular retention of MHC‐I molecules in human macrophages down‐modulating cytotoxic CD8+ T cell responses

M. Delpino, G. Giambartolomei, P. Barrionuevo, 2013, Cellular microbiology.

Brucella abortus induces the secretion of proinflammatory mediators from glial cells leading to astrocyte apoptosis.

P. Mathieu, F. Pitossi, M. Delpino, 2010, The American journal of pathology.

Brucella-infected hepatocytes mediate potentially tissue-damaging immune responses.

M. Delpino, C. A. Fossati, P. Baldi, 2010, Journal of hepatology.

Brucella abortus Inhibits Major Histocompatibility Complex Class II Expression and Antigen Processing through Interleukin-6 Secretion via Toll-Like Receptor 2

M. Delpino, C. A. Fossati, G. Giambartolomei, 2007, Infection and Immunity.

An Oral Vaccine Based on U-Omp19 Induces Protection against B. abortus Mucosal Challenge by Inducing an Adaptive IL-17 Immune Response in Mice

Fernanda S. Oliveira, S. Oliveira, H. Warzecha, 2011, PloS one.

Immunization with Recombinant Brucella Species Outer Membrane Protein Omp16 or Omp19 in Adjuvant Induces Specific CD4+ and CD8+ T Cells as Well as Systemic and Oral Protection against Brucella abortus Infection

S. Estein, C. A. Fossati, G. Giambartolomei, 2008, Infection and Immunity.

Brucella abortus–infected platelets modulate the activation of neutrophils

G. Fernández, M. Delpino, G. Giambartolomei, 2020, Immunology and cell biology.

Platelets Promote Brucella abortus Monocyte Invasion by Establishing Complexes With Monocytes

M. Delpino, G. Giambartolomei, M. A. Milillo, 2018, Front. Immunol..

Brucella abortus‐activated microglia induce neuronal death through primary phagocytosis

Sergio C Oliveira, V. Dennis, S. Oliveira, 2017, Glia.

Infected Alveolar Macrophages Downregulation in Brucella Histocompatibility Complex Class II Inflammatory Response and Major Key Role of Toll-Like Receptor 2 in the

S. Oliveira, P. P. Corsetti, P. Baldi, 2014 .

Key Role of Toll-Like Receptor 2 in the Inflammatory Response and Major Histocompatibility Complex Class II Downregulation in Brucella abortus-Infected Alveolar Macrophages

S. Oliveira, P. P. Corsetti, P. Baldi, 2013, Infection and Immunity.

The Protein Moiety of Brucella abortus Outer Membrane Protein 16 Is a New Bacterial Pathogen-Associated Molecular Pattern That Activates Dendritic Cells In Vivo, Induces a Th1 Immune Response, and Is a Promising Self-Adjuvanting Vaccine against Systemic and Oral Acquired Brucellosis

Fernanda S. Oliveira, J. Borkowski, S. Oliveira, 2010, The Journal of Immunology.

The Formyl Peptide N‐Formyl‐methionyl‐leucyl‐phenylalanine Downregulates the Expression of FcγRs in Interferon‐γ‐Activated Monocytes/Macrophages In Vitro and In Vivo

C. Rubel, M. Isturiz, M. Palermo, 2003, Scandinavian journal of immunology.

Inhibition of allospecific response in pancreatic islet transplantation: the glycan approach.

P. Argibay, M. Isturiz, P. Barrionuevo, 2005, Transplantation proceedings.

Adrenal Steroids Modulate the Immune Response during Brucella abortus Infection by a Mechanism That Depends on the Regulation of Cytokine Production

M. Delpino, P. C. Arriola Benitez, G. Giambartolomei, 2015, Infection and Immunity.

Brucella abortus induces TNF-α-dependent astroglial MMP-9 secretion through mitogen-activated protein kinases

M. Delpino, P. Baldi, G. Giambartolomei, 2013, Journal of Neuroinflammation.

Potential Role of Fibroblast-Like Synoviocytes in Joint Damage Induced by Brucella abortus Infection through Production and Induction of Matrix Metalloproteinases

S. Vanzulli, M. Delpino, C. A. Fossati, 2011, Infection and Immunity.

Granulocyte-Macrophage Colony-Stimulating Factor- and Tumor Necrosis Factor Alpha-Mediated Matrix Metalloproteinase Production by Human Osteoblasts and Monocytes after Infection with Brucella abortus

M. Delpino, C. A. Fossati, P. Baldi, 2010, Infection and Immunity.

SLAMF1 signaling induces Mycobacterium tuberculosis uptake leading to endolysosomal maturation in human macrophages

V. García, L. Balboa, A. Trotta, 2020, Journal of leukocyte biology.

Specific lytic activity against mycobacterial antigens is inversely correlated with the severity of tuberculosis

M. Franco, M. Sasiain, P. Barrionuevo, 2003, Clinical and experimental immunology.

Macrophage‐elicited osteoclastogenesis in response to Brucella abortus infection requires TLR2/MyD88‐dependent TNF‐α production

S. Oliveira, M. Delpino, C. A. Fossati, 2012, Journal of leukocyte biology.

Microbial RNA, the New PAMP of Many Faces

M. A. Milillo, P. Barrionuevo, L. Velásquez, 2022, Frontiers in Tropical Diseases.

Impaired neutrophils in children with the typical form of hemolytic uremic syndrome

G. Fernández, C. Rubel, S. Gómez, 2005, Pediatric Nephrology.

Glial Cell–Elicited Activation of Brain Microvasculature in Response to Brucella abortus Infection Requires ASC Inflammasome–Dependent IL-1β Production

K. Kim, C. Ferrari, F. Pitossi, 2016, The Journal of Immunology.

A Brucella spp. Protease Inhibitor Limits Antigen Lysosomal Proteolysis, Increases Cross-Presentation, and Enhances CD8+ T Cell Responses

M. Tkach, R. Schillaci, A. Trevani, 2016, The Journal of Immunology.

Brucella abortus-Stimulated Platelets Activate Brain Microvascular Endothelial Cells Increasing Cell Transmigration through the Erk1/2 Pathway

K. Kim, M. Delpino, G. Giambartolomei, 2020, Pathogens.

Brucella abortus Traverses Brain Microvascular Endothelial Cells Using Infected Monocytes as a Trojan Horse

K. Kim, V. Dennis, Julia Rodríguez, 2018, Front. Cell. Infect. Microbiol..

Endogenous glucocorticoids modulate neutrophil function in a murine model of haemolytic uraemic syndrome

G. Fernández, S. Gómez, M. Isturiz, 2005, Clinical and experimental immunology.

Phenotype markers and function of neutrophils in children with hemolytic uremic syndrome

G. Fernández, C. Rubel, M. Isturiz, 2002, Pediatric Nephrology.

Brucella abortus induces apoptosis of human T lymphocytes.

M. Delpino, G. Giambartolomei, P. Barrionuevo, 2012, Microbes and infection.

Tolerance to lipopolysaccharide (LPS) regulates the endotoxin effects on Shiga toxin-2 lethality.

G. Fernández, M. Isturiz, M. Palermo, 2001, Immunology letters.

Brucella abortus activates human neutrophils.

M. Delpino, G. Giambartolomei, P. Barrionuevo, 2009, Microbes and infection.

Monocytes and neutrophils from tuberculosis patients are insensitive to anti‐inflammatory effects triggered by the prototypic formyl peptide N‐formyl‐methionyl‐leucyl‐phenylalanine (FMLP)

C. Rubel, M. Isturiz, M. Palermo, 2003, Clinical and experimental immunology.

N-Formyl-Methionyl-Leucyl-Phenylalanine Inhibits both Gamma Interferon- and Interleukin-10-Induced Expression of FcγRI on Human Monocytes

C. Rubel, M. Isturiz, M. Palermo, 2001, Clinical Diagnostic Laboratory Immunology.

A bacterial protease inhibitor protects antigens delivered in oral vaccines from digestion while triggering specific mucosal immune responses.

F. Frank, G. Briones, F. Goldbaum, 2015, Journal of controlled release : official journal of the Controlled Release Society.

Inhibition of antigen presentation by Brucella: many more than many ways.

G. Giambartolomei, P. Barrionuevo, 2019, Microbes and infection.

Inhibition of MHC‐I by Brucella abortus is an early event during infection and involves EGFR pathway

M. Mercogliano, P. Elizalde, R. Schillaci, 2017, Immunology and cell biology.

Brucella abortus RNA does not polarize macrophages to a particular profile but interferes with M1 polarization

L. Balboa, M. A. Milillo, A. Trotta, 2022, PLoS neglected tropical diseases.

Brucella abortus down‐regulates MHC class II by the IL‐6‐dependent inhibition of CIITA through the downmodulation of IFN regulatory factor‐1 (IRF‐1)

R. Lang, L. Balboa, M. Delpino, 2017, Journal of leukocyte biology.

Brucella lipoproteins mimic dendritic cell maturation induced by Brucella abortus.

M. Delpino, C. A. Fossati, G. Giambartolomei, 2008, Microbes and infection.

and Oral Acquired Brucellosis Self-Adjuvanting Vaccine against Systemic Th1 Immune Response, and Is a Promising Activates Dendritic Cells In Vivo, Induces a Pathogen-Associated Molecular Pattern That Membrane Protein 16 Is a New Bacterial Outer Brucella abortus The Protein Moiety of

Fernanda S. Oliveira, J. Borkowski, H. Warzecha, 2017 .

Immune complexes (IC) down‐regulate the basal and interferon‐γ‐induced expression of MHC Class II on human monocytes

G. Fernández, M. Isturiz, M. Palermo, 2001, Clinical and experimental immunology.

Proinflammatory response of human endothelial cells to Brucella infection.

M. Delpino, P. Baldi, G. Giambartolomei, 2011, Microbes and infection.

Brucella abortus Invasion of Osteoblasts Inhibits Bone Formation

M. Delpino, G. Giambartolomei, P. Barrionuevo, 2012, Infection and Immunity.

, Inhibits Bone Formation Brucella abortus Invasion of Osteoblasts

G. Giambartolomei, P. Barrionuevo, R. Scian, 2012 .

Fatty acid oxidation of alternatively activated macrophages prevents foam cell formation, but Mycobacterium tuberculosis counteracts this process via HIF-1α activation

G. Lugo-Villarino, O. Neyrolles, Rémi Mascarau, 2020, PLoS pathogens.

Brucella abortus inhibits IFN-γ-induced FcγRI expression and FcγRI-restricted phagocytosis via toll-like receptor 2 on human monocytes/macrophages.

M. Delpino, G. Giambartolomei, P. Barrionuevo, 2011, Microbes and infection.

Prokaryotic RNA Associated to Bacterial Viability Induces Polymorphonuclear Neutrophil Activation

G. Fernández, M. A. Milillo, P. Barrionuevo, 2017, Front. Cell. Infect. Microbiol..

A Novel Function for Galectin-1 at the Crossroad of Innate and Adaptive Immunity: Galectin-1 Regulates Monocyte/Macrophage Physiology through a Nonapoptotic ERK-Dependent Pathway1

G. Rabinovich, J. Ilarregui, M. Toscano, 2007, The Journal of Immunology.

Formation of Foamy Macrophages by Tuberculous Pleural Effusions Is Triggered by the Interleukin-10/Signal Transducer and Activator of Transcription 3 Axis through ACAT Upregulation

R. Hernández-Pando, G. Lugo-Villarino, I. Maridonneau-Parini, 2018, Front. Immunol..

Mycobacterium tuberculosis Modulates the Metabolism of Alternatively Activated Macrophages to Promote Foam Cell Formation and Intracellular Survival

G. Lugo-Villarino, O. Neyrolles, Rémi Mascarau, 2019, bioRxiv.

Immune complex–FcγR interaction modulates monocyte/macrophage molecules involved in inflammation and immune response

G. Fernández, S. Gómez, M. Palermo, 2003 .

Complete sequence of a new HLA-B35 allele found in a tribe of Mapuche Indians in the south of Argentina

P. Barrionuevo, E. Kolkowski, M. Satz, 2005, Immunogenetics.

Hypothesis: an alternative pathway for the regulation of inflammation.

M. Isturiz, M. Palermo, M. Vulcano, 2004, Medicina.

Adrenal Steroids Modulate the Immune Response during Brucella abortus Infection by a Mechanism That Depends on the Regulation of Cytokine Production

M. Delpino, P. C. Arriola Benitez, G. Giambartolomei, 2015, Infection and Immunity.

Brucella-infected hepatocytes mediate potentially tissue-damaging immune responses.

M. Delpino, P. Baldi, P. Barrionuevo, 2010, Journal of hepatology.

P25 - HLA-B35 alleles in 440 individuals from 16 different populations as assessed by DNA typing

C. Carcassi, R. Scorza, P. Stastny, 1996 .