K J Pienta

发表

Cell motility as a chemotherapeutic target.

K. Pienta, D. S. Coffey, K J Pienta, 1991, Cancer surveys.

The tissue matrix: cell dynamics and hormone action.

K. Pienta, D. S. Coffey, R. Getzenberg, 1990, Endocrine reviews.

Cellular harmonic information transfer through a tissue tensegrity-matrix system.

K. Pienta, D. S. Coffey, K J Pienta, 1991, Medical hypotheses.

Novel molecular targets for prostate cancer therapy.

K. Pienta, K J Pienta, J M Kamradt, 1999, Seminars in oncology.

the PHSCN Sequence in Prostate Carcinoma Anti-invasive , Antitumorigenic , and Antimetastatic Activities of Updated Version

K. Kurachi, K. Pienta, D. Allen, 2000 .

Cancer as a complex adaptive system.

K. Pienta, K J Pienta, E D Schwab, 1996, Medical hypotheses.

Nuclear‐Cytoskeletal interactions: Evidence for physical connections between the nucleus and cell periphery and their alteration by transformation

K. Pienta, D. S. Coffey, K J Pienta, 1992, Journal of cellular biochemistry.

Correlation of nuclear morphometry with progression of breast cancer

K. Pienta, D. S. Coffey, K J Pienta, 1991, Cancer.

VCaP, a cell-based model system of human prostate cancer.

K. Pienta, D L Lin, L. McLean, 2001, In vivo.

Inhibition of spontaneous metastasis in a rat prostate cancer model by oral administration of modified citrus pectin.

K. Pienta, V. Hogan, A. Raz, 1995, Journal of the National Cancer Institute.

Modeling signal transduction in normal and cancer cells using complex adaptive systems.

K. Pienta, K J Pienta, E D Schwab, 1997, Medical hypotheses.