Norina M. Tang

Control of PKR Protein Kinase by Hepatitis C Virus Nonstructural 5A Protein: Molecular Mechanisms of Kinase Regulation

Michael G. Katze, Michael Gale, Seng-Lai Tan, 1998, Molecular and Cellular Biology.

Shiga-like toxin-producing Escherichia coli in Seattle children: a prospective study.

N. Tang, S. Moseley, P. Tarr, 1993, Gastroenterology.

Evidence that hepatitis C virus resistance to interferon is mediated through repression of the PKR protein kinase by the nonstructural 5A protein.

M. Katze, D. Hopkins, S. Tan, 1997, Virology.

The 58,000-dalton cellular inhibitor of the interferon-induced double-stranded RNA-activated protein kinase (PKR) is a member of the tetratricopeptide repeat family of proteins

M. Katze, N. Tang, T. Lee, 1994, Molecular and cellular biology.

Inhibition of Double-Stranded RNA- and Tumor Necrosis Factor Alpha-Mediated Apoptosis by Tetratricopeptide Repeat Protein and Cochaperone P58IPK

M. Katze, B. Williams, M. J. Korth, 1999, Molecular and Cellular Biology.

The 58-kDa Cellular Inhibitor of the Double Stranded RNA-dependent Protein Kinase Requires the Tetratricopeptide Repeat 6 and DnaJ Motifs to Stimulate Protein Synthesis in Vivo*

M. Katze, N. Tang, C. Ho, 1996, The Journal of Biological Chemistry.

The P58 Cellular Inhibitor Complexes with the Interferon-induced, Double-stranded RNA-dependent Protein Kinase, PKR, to Regulate Its Autophosphorylation and Activity (*)

M. Katze, G. Barber, S. Polyak, 1996, The Journal of Biological Chemistry.