E. Akirav

发表

Effects of insulin treatment without and with recurrent hypoglycemia on hypoglycemic counterregulation and adrenal catecholamine-synthesizing enzymes in diabetic rats.

M. Vranic, M. Riddell, K. Inouye, 2006, Endocrinology.

Partial leptin restoration increases hypothalamic-pituitary-adrenal activity while diminishing weight loss and hyperphagia in streptozotocin diabetic rats.

M. Vranic, M. Riddell, K. Inouye, 2004, Metabolism: clinical and experimental.

Resident B cells regulate thymic expression of myelin oligodendrocyte glycoprotein

Yan Xu, E. Akirav, N. Ruddle, 2011, Journal of Neuroimmunology.

The role of AIRE in human autoimmune disease

K. Herold, E. Akirav, N. Ruddle, 2011, Nature Reviews Endocrinology.

Secondary Lymphoid Organs: Responding to Genetic and Environmental Cues in Ontogeny and the Immune Response1

E. Akirav, N. Ruddle, 2009, The Journal of Immunology.

β-Cell Mass and Type 1 Diabetes

J. Kushner, K. Herold, E. Akirav, 2008, Diabetes.

RAGE Expression in Human T Cells: A Link between Environmental Factors and Adaptive Immune Responses

K. Herold, A. Schmidt, P. Preston-Hurlburt, 2012, PloS one.

RAGE Ligation Affects T Cell Activation and Controls T Cell Differentiation1

K. Herold, Yali Chen, A. Schmidt, 2008, The Journal of Immunology.

Insulin alone increases hypothalamo-pituitary-adrenal activity, and diabetes lowers peak stress responses.

M. Vranic, M. Riddell, K. Inouye, 2005, Endocrinology.

Hyperglycemia does not increase basal hypothalamo-pituitary-adrenal activity in diabetes but it does impair the HPA response to insulin-induced hypoglycemia.

M. Vranic, M. Riddell, K. Inouye, 2005, American journal of physiology. Regulatory, integrative and comparative physiology.

Human EndoC-βH1 β-cells form pseudoislets with improved glucose sensitivity and enhanced GLP-1 signaling in the presence of islet-derived endothelial cells.

Michael G. Spelios, E. Akirav, Lauren Afinowicz, 2018, American journal of physiology. Endocrinology and metabolism.

Human EndoC- (cid:2) H1 (cid:2) -cells form pseudoislets with improved glucose sensitivity and enhanced GLP-1 signaling in the presence of islet-derived endothelial cells

Michael G. Spelios, E. Akirav, Lauren Afinowicz, 2018 .

Controls T Cell Differentiation RAGE Ligation Affects T Cell Activation and

K. Herold, Yali Chen, A. Schmidt, 2008 .

Islet Endothelial Cells Induce Glycosylation and Increase Cell-surface Expression of Integrin β1 in β Cells*

John A. Olsen, Michael G. Spelios, E. Akirav, 2015, The Journal of Biological Chemistry.

Immune Therapy and β-Cell Death in Type 1 Diabetes

J. Bluestone, S. Gitelman, K. Herold, 2013, Diabetes.

Immune Therapy and B-cell Death in Type 1 Diabetes Research Design and Methods

J. Bluestone, S. Gitelman, K. Herold, 2022 .

Immunological biomarkers: Catalysts for translational advances in autoimmune diabetes

E. McKinney, S. Ahmed, F. Waldron-Lynch, 2013, Clinical and experimental immunology.

A Minimally-invasive Blood-derived Biomarker of Oligodendrocyte Cell-loss in Multiple Sclerosis

M. Stecker, Michael G. Spelios, E. Akirav, 2016, EBioMedicine.

Remyelination in multiple sclerosis: Cellular mechanisms and novel therapeutic approaches

E. Akirav, J. Olsen, 2015, Journal of neuroscience research.

Detection of β cell death in diabetes using differentially methylated circulating DNA

P. Lizardi, K. Herold, O. Henegariu, 2011, Proceedings of the National Academy of Sciences.

The Receptor for Advanced Glycation End Products (RAGE) Affects T Cell Differentiation in OVA Induced Asthma

Ann Marie Schmidt, Kevan C. Herold, K. Herold, 2014, PloS one.

A composite immune signature parallels disease progression across T1D subjects.

P. Linsley, R. Gottardo, M. Hessner, 2019, JCI insight.

Beta-cell mass and type 1 diabetes: going, going, gone?

J. Kushner, K. Herold, E. Akirav, 2008, Diabetes.

Circulating Differentially Methylated Amylin DNA as a Biomarker of β-Cell Loss in Type 1 Diabetes

M. Hessner, Michael G. Spelios, E. Akirav, 2016, PloS one.

Glucose and inflammation control islet vascular density and beta-cell function in NOD mice: control of islet vasculature and vascular endothelial growth factor by glucose.

D. Rimm, J. Kushner, K. Herold, 2011, Diabetes.

β-Cell death is decreased in women with gestational diabetes mellitus

Michael G. Spelios, E. Akirav, M. Radin, 2016, Diabetology & Metabolic Syndrome.

Prevention of Islet Graft Rejection and Recipient Tolerization

K. Herold, E. Akirav, 2010 .

Depletion of CD4+CD25+ T cells exacerbates experimental autoimmune encephalomyelitis induced by mouse, but not rat, antigens

E. Akirav, N. Ruddle, C. Bergman, 2009, Journal of neuroscience research.

Mechanisms of impaired hypothalamic-pituitary-adrenal (HPA) function in diabetes: reduced counterregulatory responsiveness to hypoglycaemia.

M. Vranic, M. Riddell, K. Inouye, 2002, Diabetes, Nutrition and Metabolism. Clinical and Experimental.

In Vitro Formation of β Cell Pseudoislets Using Islet-Derived Endothelial Cells

Michael G. Spelios, E. Akirav, Lauren A. Kenna, 2013, PloS one.

Depletion of CD4+CD25+ T cells exacerbates experimental autoimmune encephalomyelitis induced by mouse, but not rat, antigens

E. Akirav, N. Ruddle, C. Bergman, 2009, Journal of neuroscience research.

Insulin alone increases hypothalamo-pituitary-adrenal activity, and diabetes lowers peak stress responses.

M. Vranic, M. Riddell, K. Inouye, 2005, Endocrinology.