A. Warbritton

Protective Effect of Type 2 Diabetes on Acetaminophen-Induced Hepatotoxicity in Male Swiss-Webster Mice

Sharmilee P. Sawant, A. Dnyanmote, H. Mehendale, 2006, Journal of Pharmacology and Experimental Therapeutics.

Prior administration of a low dose of thioacetamide protects type 1 diabetic rats from subsequent administration of lethal dose of thioacetamide.

H. Mehendale, J. Latendresse, A. Warbritton, 2006, Toxicology.

Tolerance of aged Fischer 344 rats against chlordecone-amplified carbon tetrachloride toxicity

H. Mehendale, J. Latendresse, A. Warbritton, 2004, Mechanisms of Ageing and Development.

Role of CYP2E1 and saturation kinetics in the bioactivation of thioacetamide: Effects of diet restriction and phenobarbital.

K. Shankar, H. Mehendale, J. Latendresse, 2007, Toxicology and applied pharmacology.

Toxicokinetics and toxicity of thioacetamide sulfoxide: a metabolite of thioacetamide.

H. Mehendale, J. Latendresse, J. Chilakapati, 2007, Toxicology.

Calpastatin overexpression prevents progression of S-1,2-dichlorovinyl-l-cysteine (DCVC)-initiated acute renal injury and renal failure (ARF) in diabetes.

E. Lock, Sharmilee P. Sawant, A. Dnyanmote, 2006, Toxicology and Applied Pharmacology.

Pancreatic Islet Cells in Preobese Yellow Avy/- Mice: Relation to Adult Hyperinsulinemia and Obesity

G. Wolff, A. Warbritton, T. Bucci, 1994, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

Stimulated hepatic tissue repair underlies heteroprotection by thioacetamide against acetaminophen‐induced lethality

H. Mehendale, R. Mangipudy, A. Warbritton, 1995, Hepatology.

Diabetic mice are protected from normally lethal nephrotoxicity of S-1,2-dichlorovinyl-L-cysteine (DCVC): role of nephrogenic tissue repair.

E. Lock, Sharmilee P. Sawant, A. Dnyanmote, 2006, Toxicology and applied pharmacology.

Type 2 diabetic rats are sensitive to thioacetamide hepatotoxicity.

Sharmilee P. Sawant, A. Dnyanmote, H. Mehendale, 2006, Toxicology and applied pharmacology.

Preplaced cell division: a critical mechanism of autoprotection against S-1,2-dichlorovinyl-L-cysteine-induced acute renal failure and death in mice.

E. Lock, H. Mehendale, J. Latendresse, 2006, American journal of physiology. Renal physiology.