I. Khaliulin

发表

Acute regional changes in myocardial strain may predict ventricular remodelling after myocardial infarction in a large animal model

K. Fraser, A. Chiribiri, E. Sammut, 2021, Scientific Reports.

Cardioprotective and Antiarrhythmic Drugs Based on Opioid Receptor

Yury, J. Downey, P. Oeltgen, 2016 .

Prospects for Creation of Cardioprotective and Antiarrhythmic Drugs Based on Opioid Receptor Agonists

J. Downey, P. Oeltgen, J. Pei, 2016, Medicinal research reviews.

Mimicking the Phenomenon of Ischemic Preconditioning by Actions on Cannabinoid Receptors: The Roles of Protein Kinase C and NO Synthase

I. Khaliulin, L. Maslov, Y. Lishmanov, 2016, Neuroscience and Behavioral Physiology.

Prospects for Creation of Cardioprotective Drugs Based on Cannabinoid Receptor Agonists

J. Downey, R. Mechoulam, L. De Petrocellis, 2016, Journal of cardiovascular pharmacology and therapeutics.

The Question of the End Effector of Ischemic Postconditioning of the Heart

J. Pei, I. Khaliulin, L. Maslov, 2015, Neuroscience and Behavioral Physiology.

[MIMICKING ISCHEMIC PRECONDITIONING PHENOMENON THROUGH THE IMPACT ON THE CANNABINOID RECEPTORS: ROLE OF PROTEIN KINASE AND NO-SYNTHASE].

I. Khaliulin, L. Maslov, Y. Lishmanov, 2015, Rossiiskii fiziologicheskii zhurnal imeni I.M. Sechenova.

Temperature preconditioning of isolated rat hearts – a potent cardioprotective mechanism involving a reduction in oxidative stress and inhibition of the mitochondrial permeability transition pore

A. Halestrap, S. Clarke, M. Suleiman, 2007, The Journal of physiology.

Prevention of ischemic disturbances of contractile function of isolated heart by short-term total ischemia episodes

I. Khaliulin, B. Laptev, D. V. Ushcheko, 1997, Bulletin of Experimental Biology and Medicine.

Hypoxic Preconditioning – a Phenomenon Increasing the Tolerance of Cardiomyocytes to Hypoxia/Reoxygenation

J. Pei, I. Khaliulin, F. Kolář, 2010, Neuroscience and Behavioral Physiology.

Inhibition of Mitochondrial Permeability Transition Pore Opening by Ischemic Preconditioning Is Probably Mediated by Reduction of Oxidative Stress Rather Than Mitochondrial Protein Phosphorylation

K. Heesom, A. Halestrap, S. Clarke, 2008, Circulation research.

Role of the prostacycline-thromboxane system in mechanisms preventing arrhythmias induced by coronary occlusion in adapted rats

I. Khaliulin, L. Maslov, Y. Lishmanov, 2004, Bulletin of Experimental Biology and Medicine.

Preconditioning or Postconditioning with 8-Br-cAMP-AM Protects the Heart against Regional Ischemia and Reperfusion: A Role for Mitochondrial Permeability Transition

M. Suleiman, R. Ascione, I. Khaliulin, 2021, Cells.

Consecutive Isoproterenol and Adenosine Treatment Confers Marked Protection against Reperfusion Injury in Adult but Not in Immature Heart: A Role for Glycogen

C. Orchard, M. Suleiman, E. Griffiths, 2018, International journal of molecular sciences.

Cardioprotection of Immature Heart by Simultaneous Activation of PKA and Epac: A Role for the Mitochondrial Permeability Transition Pore

M. Suleiman, I. Khaliulin, Martin Lewis, 2022, International journal of molecular sciences.

[Hypoxic preconditioning as novel approach to prophylaxis of ischemic and reperfusion damage of brain and heart].

J. Pei, I. Khaliulin, F. Kolář, 2011, Angiologiia i sosudistaia khirurgiia = Angiology and vascular surgery.

Activation of Glibenclamide-Sensitive ATP-Sensitive K+ Channels During &bgr;-Adrenergically Induced Metabolic Stress Produces a Substrate for Atrial Tachyarrhythmia

J. Hancox, Shang-Jin Kim, A. James, 2012, Circulation. Arrhythmia and electrophysiology.

Remote ischemic preconditioning triggers changes in autonomic nervous system activity

G. Angelini, M. Suleiman, M. Meloni, 2017 .

Urocortin prevents mitochondrial permeability transition in response to reperfusion injury indirectly by reducing oxidative stress.

R. Knight, D. Latchman, P. Townsend, 2007, American journal of physiology. Heart and circulatory physiology.

Reactive oxygen species, but not Ca overloading, trigger pH- and mitochondrial permeability transition-dependent death of adult rat myocytes after ischemia-reperfusion

A. Halestrap, P. Townsend, A. Stephanou, 2006 .

Problem of heart salvation during reperfusion. Opioid receptor agonists as a possible solution

I. Khaliulin, L. Maslov, 2017 .

The regulation of necroptosis and perspectives for the development of new drugs preventing ischemic/reperfusion of cardiac injury

A. Degterev, Nirmal Singh, A. Boshchenko, 2022, Apoptosis.

Activation of Peripheral Opioid κ 1 Receptor Prevents Cardiac Reperfusion Injury

P. Oeltgen, I. Khaliulin, L. Maslov, 2021 .

Hypoxic Preconditioning – a Phenomenon Increasing the Tolerance of Cardiomyocytes to Hypoxia/Reoxygenation

J. Pei, I. Khaliulin, F. Kolář, 2010, Neuroscience and Behavioral Physiology.

Remote ischemic preconditioning triggers changes in autonomic nervous system activity: implications for cardioprotection

G. Angelini, M. Suleiman, M. Meloni, 2017, Physiological reports.

The role of mitochondria in protection of the heart by preconditioning

A. Halestrap, S. Clarke, I. Khaliulin, 2007, Biochimica et biophysica acta.

Functional and cardioprotective effects of simultaneous and individual activation of protein kinase A and Epac

Jason L. Johnson, A. James, M. Suleiman, 2017, British journal of pharmacology.

Comparative Analysis of Early and Delayed Cardioprotective and Antiarrhythmic Efficacy of Hypoxic Preconditioning

I. Khaliulin, L. Maslov, Y. Lishmanov, 2014, Bulletin of Experimental Biology and Medicine.

Role of MEK, PI3, p38, Tyrosine, and mTOR Kinases in Regulation of Heart Resistance to the Arrhythmogenic Action of Short-Term Ischemia and Reperfusion

I. Khaliulin, L. Maslov, A. G. Portnichenko, 2015, Bulletin of Experimental Biology and Medicine.

Neuro-autonomic changes induced by remote ischemic preconditioning (RIPC) in healthy young adults: Implications for stress

M. Suleiman, R. Ascione, I. Khaliulin, 2019, Neurobiology of Stress.

Consecutive pharmacological activation of PKA and PKC mimics the potent cardioprotection of temperature preconditioning

A. Halestrap, I. Khaliulin, Joanne E. Parker, 2010, Cardiovascular research.

Clinically-relevant consecutive treatment with isoproterenol and adenosine protects the failing heart against ischaemia and reperfusion

S. Bryant, A. Halestrap, A. James, 2014, Journal of Translational Medicine.

Reperfusion Cardiac Injury: Receptors and the Signaling Mechanisms

A. Boshchenko, S. Popov, V. Ryabov, 2022, Current cardiology reviews.

Protection Ion Transport and Energetics During Cell

Rackley, L. Opie, J. Ingwall, 2008 .

[Perspective of use of agonists of adenosine and opioid receptors for prevention of reperfusion damages of heart. Analysis of experimental and clinical data].

I. Khaliulin, A. Mrochek, L. Maslov, 2014, Vestnik Rossiiskoi akademii meditsinskikh nauk.

[Role of thyroid system in adaptation to cold].

I. Khaliulin, L. Maslov, A. Gorbunov, 2014, Rossiiskii fiziologicheskii zhurnal imeni I.M. Sechenova.

Temperature preconditioning is optimal at 26°C and confers additional protection to hypothermic cardioplegic ischemic arrest

A. Halestrap, M. Suleiman, I. Khaliulin, 2011, Experimental biology and medicine.

The Question of the End Effector of Ischemic Postconditioning of the Heart

J. Pei, I. Khaliulin, L. Maslov, 2015, Neuroscience and Behavioral Physiology.

Regional Changes in Myocardial Strain Predict Ventricular Remodelling after Myocardial Infarction in a Large Animal Model

K. Fraser, H. Gill, A. Chiribiri, 2021 .

[Uncoupling proteins and their role in the regulation of brain and heart tolerance to impact of ischemia and reperfusion].

J. Pei, E. Griffiths, I. Khaliulin, 2011, Rossiiskii fiziologicheskii zhurnal imeni I.M. Sechenova.

Activation of peripheral δ2-opioid receptor prevents reperfusion heart injury.

P. Oeltgen, M. Suleiman, I. Khaliulin, 2021, European journal of pharmacology.

Cardioprotection of Immature Heart by Simultaneous Activation of PKA and Epac: A Role for the Mitochondrial Permeability Transition Pore

I. Khaliulin, Martin Lewis, Katie Hall, 2022, International journal of molecular sciences.