Y Kitamura

Activating mutation in the catalytic domain of c-kit elicits hematopoietic transformation by receptor self-association not at the ligand-induced dimerization site.

T. Kaisho, H. Kitayama, Y. Kitamura, 1999, Blood.

Decreased production of mast cells in S1/S1d anemic mice.

Y. Kitamura, Y Kitamura, S Go, 1979, Blood.

A truncated isoform of the PP2A B56 subunit promotes cell motility through paxillin phosphorylation

H Nojima, H. Nojima, H. Sabe, 2000, The EMBO journal.

Distribution of mast-cell precursors in hematopoeitic and lymphopoietic tissues of mice

Y. Kitamura, H. Matsuda, Y Kitamura, 1979, The Journal of experimental medicine.

Impairment of spatial learning and hippocampal synaptic potentiation in c-kit mutant rats.

S. Hirota, Y. Kitamura, Y Kitamura, 2000, Learning & memory.

A loss-of-function mutation of c-kit results in depletion of mast cells and interstitial cells of Cajal, while its gain-of-function mutation results in their oncogenesis.

S. Hirota, T. Nishida, Y. Kitamura, 2001, Mutation research.

Constitutively activating mutations of c-kit receptor tyrosine kinase confer factor-independent growth and tumorigenicity of factor-dependent hematopoietic cell lines.

H. Kitayama, Y. Kitamura, Y. Kanakura, 1995, Blood.