M. Stoner

发表

CD28 down-regulation on CD4 T cells is a marker for graft dysfunction in lung transplant recipients.

Xuehai Zhu, S. Studer, V. Valentine, 2008, American journal of respiratory and critical care medicine.

Diet-induced obese mice are resistant to improvements in cardiac function resulting from short-term adropin treatment

I. Scott, M. Jurczak, M. Stoner, 2022, Current research in physiology.

Idiopathic Pulmonary Fibrosis Cellular and Humoral Autoreactivity in Zhang

N. Kaminski, A. Choi, C. Feghali-Bostwick, 2007 .

Cellular and Humoral Autoreactivity in Idiopathic Pulmonary Fibrosis1

N. Kaminski, A. Choi, J. Pilewski, 2007, The Journal of Immunology.

Cardiac-specific deletion of GCN5L1 restricts recovery from ischemia-reperfusion injury.

I. Scott, C. McTiernan, S. Shiva, 2019, Journal of molecular and cellular cardiology.

Cardiomyocyte-Specific Deletion of GCN5L1 in Mice Limits Ex Vivo Cardiac Functional Decline in Response to a High Fat Diet

I. Scott, M. Sack, M. Stoner, 2019 .

Cardiomyocyte-Specific Deletion of GCN5L1 Limits Cardiac Contractile Decline in Obese Pre-Diabetic Mice

I. Scott, M. Sack, M. Stoner, 2019, bioRxiv.

Acetylation of mitochondrial proteins by GCN5L1 promotes enhanced fatty acid oxidation in the heart.

I. Scott, S. Shiva, R. O’Doherty, 2017, American journal of physiology. Heart and circulatory physiology.

GPER-dependent estrogen signaling increases cardiac GCN5L1 expression and MCAD activity

M. Rojas, I. Scott, J. Sembrat, 2021, bioRxiv.

A human-mouse chimeric model of obliterative bronchiolitis after lung transplantation.

Xuehai Zhu, M. Myerburg, J. Xue, 2011, The American journal of pathology.

The protein acetylase GCN5L1 modulates hepatic fatty acid oxidation activity via acetylation of the mitochondrial β-oxidation enzyme HADHA

Yong Chen, M. Gucek, N. Mehta, 2018, The Journal of Biological Chemistry.

Autoantibodies in patients with chronic obstructive pulmonary disease.

F. Sciurba, L. Otterbein, J. Pilewski, 2008, American journal of respiratory and critical care medicine.

GCN5L1 impairs diastolic function in mice exposed to a high fat diet by restricting cardiac pyruvate oxidation

N. Yates, I. Scott, M. Jurczak, 2022, Physiological reports.

GCN 5 L 1 promotes diastolic dysfunction by inhibiting cardiac pyruvate oxidation 1 2

N. Yates, I. Scott, M. Jurczak, 2022 .

Inhibitory Acetylation of Pyruvate Dehydrogenase Promotes Diastolic Dysfunction in Mice

I. Scott, M. Jurczak, M. Stoner, 2021 .

Loss of GCN5L1 in cardiac cells disrupts glucose metabolism and promotes cell death via reduced Akt/mTORC2 signaling.

I. Scott, S. Shiva, C. Corey, 2019, The Biochemical journal.

α-Lipoic acid promotes α-tubulin hyperacetylation and blocks the turnover of mitochondria through mitophagy.

Michael J. Calderon, I. Scott, C. S. St. Croix, 2016, The Biochemical journal.

GCN5L1 promotes diastolic dysfunction by inhibiting cardiac pyruvate oxidation

N. Yates, I. Scott, M. Jurczak, 2022, bioRxiv.

Reduced acetylation of TFAM promotes bioenergetic dysfunction in the failing heart

B. Kaufman, I. Scott, C. McTiernan, 2022, bioRxiv.

Adropin regulates pyruvate dehydrogenase in cardiac cells via a novel GPCR-MAPK-PDK4 signaling pathway

I. Scott, S. Shiva, M. Jurczak, 2018, Redox biology.

G-PROTEIN COUPLED RECEPTOR 19 (GPR19) KNOCKOUT MICE DISPLAY SEX-DEPENDENT METABOLIC DYSFUNCTION

I. Scott, M. Jurczak, M. Stoner, 2022, bioRxiv.

Adropin treatment restores cardiac glucose oxidation in pre-diabetic obese mice.

I. Scott, C. McTiernan, M. Jurczak, 2019, Journal of molecular and cellular cardiology.

Peripheral T Cell Functions Correlate with the Severity of Chronic Obstructive Pulmonary Disease1

F. Sciurba, Xuehai Zhu, R. Givelber, 2009, The Journal of Immunology.

Pulmonary Disease the Severity of Chronic Obstructive Peripheral T Cell Functions Correlate with

F. Sciurba, Xuehai Zhu, R. Givelber, 2009 .

Loss of GCN5L1 in cardiac cells limits mitochondrial respiratory capacity under hyperglycemic conditions

I. Scott, S. Shiva, Y. Lai, 2019, Physiological Reports.

Validation of GCN5L1/BLOC1S1/BLOS1 Antibodies Using Knockout Cells and Tissue

I. Scott, M. Stoner, Dharendra Thapa, 2023, bioRxiv.

Cardiomyocyte-specific deletion of GCN5L1 in mice restricts mitochondrial protein hyperacetylation in response to a high fat diet

I. Scott, M. Stoner, Bingxian Xie, 2020, Scientific Reports.

The occurrence patterns of eye-hand preference in athletes and non-athletes

M. Stoner, M. W. Stoner, Jill M Hlavac, 1991 .