K. Nagashima

发表

14-3-3 and calmodulin control subcellular distribution of Kir/Gem and its regulation of cell shape and calcium channel activity

Yuichiro Yamada, Y. Seino, W. Hunziker, 2005, Journal of Cell Science.

Lack of goal attainment regarding the low-density lipoprotein cholesterol level in the management of type 2 diabetes mellitus.

N. Inagaki, N. Harada, A. Hamasaki, 2013, Internal medicine.

Rapamycin impairs metabolism-secretion coupling in rat pancreatic islets by suppressing carbohydrate metabolism.

Hiroki Sato, Y. Seino, N. Inagaki, 2009, The Journal of endocrinology.

Autoantibodies against the exocrine pancreas in fulminant type 1 diabetes.

Yuichiro Yamada, K. Okazaki, Y. Seino, 2005, Pancreas.

High Prevalence of Autoantibodies Against Carbonic Anhydrase II and Lactoferrin in Type 1 Diabetes: Concept of Autoimmune Exocrinopathy and Endocrinopathy of the Pancreas

Yuichiro Yamada, T. Chiba, K. Uchida, 2003, Pancreas.

Sulfonylurea and non-sulfonylurea hypoglycemic agents: pharmachological properties and tissue selectivity.

Yuichiro Yamada, Y. Seino, A. Hamasaki, 2004, Diabetes research and clinical practice.

Gastric inhibitory polypeptide is the major insulinotropic factor in K(ATP) null mice.

S. Toyokuni, Yuichiro Yamada, Y. Oiso, 2004, European journal of endocrinology.

Diverse roles of K(ATP) channels learned from Kir6.2 genetically engineered mice.

T. Iwanaga, S. Seino, T. Miki, 2000, Diabetes.

PKA‐mediated phosphorylation of the human KATP channel: separate roles of Kir6.2 and SUR1 subunit phosphorylation

S. Seino, M. Nishimura, P. Béguin, 1999, The EMBO journal.

Defective insulin secretion and enhanced insulin action in KATP channel-deficient mice.

J. Miyazaki, T. Iwanaga, F. Tashiro, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Dietary habits associated with reduced insulin resistance: The Nagahama study.

A. Sekine, F. Matsuda, T. Nakayama, 2018, Diabetes research and clinical practice.

Chronic administration of apple polyphenols ameliorates hyperglycaemia in high-normal and borderline subjects: A randomised, placebo-controlled trial.

N. Inagaki, T. Shoji, M. Maeda-Yamamoto, 2017, Diabetes research and clinical practice.

Glycemic Variability Is Associated With Quality of Life and Treatment Satisfaction in Patients With Type 1 Diabetes

N. Inagaki, A. Hamasaki, K. Toyoda, 2014, Diabetes Care.

A new equation to estimate basal energy expenditure of patients with diabetes.

Takashi Kawamura, Akihiro Hamasaki, Masashi Goto, 2013, Clinical nutrition.

Src regulates insulin secretion and glucose metabolism by influencing subcellular localization of glucokinase in pancreatic β‐cells

Hiroki Sato, N. Fujita, N. Inagaki, 2015, Journal of diabetes investigation.

Gatifloxacin acutely stimulates insulin secretion and chronically suppresses insulin biosynthesis.

Yuichiro Yamada, Y. Seino, N. Inagaki, 2006, European journal of pharmacology.

Glucose-stimulated Single Pancreatic Islets Sustain Increased Cytosolic ATP Levels during Initial Ca2+ Influx and Subsequent Ca2+ Oscillations*

H. Noji, S. Takashima, Hajime Fukuoka, 2013, The Journal of Biological Chemistry.

Roles of ATP-sensitive K+ channels in cell survival and differentiation in the endocrine pancreas.

T. Iwanaga, T. Miki, K. Nagashima, 2001, Diabetes.

Abnormalities of pancreatic islets by targeted expression of a dominant-negative KATP channel.

H. Aihara, J. Miyazaki, T. Iwanaga, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Insulin secretion and differential gene expression in glucose-responsive and-unresponsive MIN 6 sublines

J. Miyazaki, S. Seino, T. Miki, 2000 .

Adult pancreatic islets require differential pax6 gene dosage.

S. Toyokuni, Yuichiro Yamada, Y. Seino, 2007, Biochemical and biophysical research communications.

Perspectives in Diabetes Diverse Roles of K AT P Channels Learned From Kir 6 . 2 Genetically Engineered Mice

T. Iwanaga, S. Seino, T. Miki, 2000 .

Molecular basis of functional diversity of ATP-sensitive K+ channels.

C. H. Wang, L. Aguilar-Bryan, S. Seino, 1997, The Japanese journal of physiology.

Palmitate induces reactive oxygen species production and β‐cell dysfunction by activating nicotinamide adenine dinucleotide phosphate oxidase through Src signaling

Hiroki Sato, N. Inagaki, E. Mukai, 2013, Journal of diabetes investigation.

Insulinoma with a History of Epilepsy: Still a Possible Misleading Factor in the Early Diagnosis of Insulinoma

T. Masui, D. Yabe, N. Inagaki, 2017, Internal medicine.

Mutations in KCNJ11 are associated with the development of autosomal dominant, early-onset type 2 diabetes

K. Xiang, T. Friedman, K. Nagashima, 2013, Diabetologia.

Gastric inhibitory polypeptide is the major insulinotropic factor in K(ATP) null mice.

S. Toyokuni, Yuichiro Yamada, Y. Oiso, 2004, European journal of endocrinology.

Dietary Sodium Restriction Reduces Nocturnal Urine Volume and Nocturnal Polyuria Index in Renal Allograft Recipients With Nocturnal Polyuria.

O. Ogawa, H. Negoro, M. Yanagita, 2017, Urology.

[ATP-sensitive K+ channel].

K. Nagashima, 2002, Nihon rinsho. Japanese journal of clinical medicine.

The spontaneously diabetic Torii rat with gastroenteropathy.

Yuichiro Yamada, Y. Seino, N. Inagaki, 2007, Diabetes research and clinical practice.

Epidemiology, clinical characteristics, and genetic etiology of neonatal diabetes in Japan

N. Inagaki, K. Nagashima, D. Tanaka, 2017, Pediatrics international : official journal of the Japan Pediatric Society.

Essential Role of Ubiquitin-Proteasome System in Normal Regulation of Insulin Secretion*

S. Seino, K. Minami, K. Nagashima, 2006, Journal of Biological Chemistry.

GCKR mutations in Japanese families with clustered type 2 diabetes.

A. Koizumi, Kouji H. Harada, N. Inagaki, 2011, Molecular genetics and metabolism.

Chapter 20 Structure and Function of ATP-Sensitive Potassium Channels

S. Seino, T. Miki, N. Inagaki, 1999 .

The structure and function of the ATP-sensitive K channel in insulin-secreting pancreatic â-cells

T. Miki, K. Nagashima, S. Seino, 1999 .

Exome sequencing identifies a new candidate mutation for susceptibility to diabetes in a family with highly aggregated type 2 diabetes.

A. Koizumi, N. Inagaki, K. Nagashima, 2013, Molecular genetics and metabolism.

The C42R mutation in the Kir6.2 (KCNJ11) gene as a cause of transient neonatal diabetes, childhood diabetes, or later-onset, apparently type 2 diabetes mellitus.

Yuichiro Yamada, T. Nakahata, N. Inagaki, 2005, The Journal of clinical endocrinology and metabolism.

The Na+-driven Cl−/HCO3 −Exchanger

S. Seino, H. Yano, S Seino, 2000, The Journal of Biological Chemistry.

BARP suppresses voltage-gated calcium channel activity and Ca2+-evoked exocytosis

Y. Mori, S. Seino, T. Launey, 2014, The Journal of cell biology.

JCB_201304101 1..17

Y. Mori, S. Seino, T. Launey, 2014 .

Regulation of Ca2+ channel expression at the cell surface by the small G-protein kir/Gem

K. Geering, T. Shibasaki, Y. Kashima, 2001, Nature.

The Na+-driven Cl-/HCO3 − exchanger: cloning, tissue distribution, and functional characterization. J Biol Chem

S. Seino, H. Yano, K. Nagashima, 2000 .

Insulin secretion and differential gene expression in glucose-responsive and -unresponsive MIN6 sublines.

J. Miyazaki, S. Seino, T. Miki, 2000, American journal of physiology. Endocrinology and metabolism.

Cdk5-dependent regulation of glucose-stimulated insulin secretion

K. Mikoshiba, K. Tomizawa, M. Matsushita, 2005, Nature Medicine.

Title Transcriptional regulatory factor X6 (Rfx6) increases gastric inhibitory polypeptide (GIP) expression in enteroendocrine K- cells and is involved in GIP hypersecretion in high fat diet- induced obesity

N. Inagaki, N. Harada, S. Yamane, 2016 .

Transcriptional Regulatory Factor X6 (Rfx6) Increases Gastric Inhibitory Polypeptide (GIP) Expression in Enteroendocrine K-cells and Is Involved in GIP Hypersecretion in High Fat Diet-induced Obesity*

N. Inagaki, N. Harada, S. Yamane, 2012, The Journal of Biological Chemistry.

Rfx6 increases GIP mRNA expression in K-cells

N. Inagaki, N. Harada, S. Yamane, 2012 .

Gastric inhibitory polypeptide modulates adiposity and fat oxidation under diminished insulin action.

Yuichiro Yamada, T. Kadowaki, Y. Seino, 2005, Biochemical and biophysical research communications.

Single-Cell Transcriptome Analysis Dissects the Replicating Process of Pancreatic Beta Cells in Partial Pancreatectomy Model

D. Yabe, N. Inagaki, E. Mukai, 2020, iScience.

Oral Administration of Apple Procyanidins Ameliorates Insulin Resistance via Suppression of Pro-Inflammatory Cytokine Expression in Liver of Diabetic ob/ob Mice.

Hiroki Sato, N. Fujita, N. Inagaki, 2016, Journal of agricultural and food chemistry.

Utility of indices using C‐peptide levels for indication of insulin therapy to achieve good glycemic control in Japanese patients with type 2 diabetes

Y. Seino, N. Inagaki, A. Hamasaki, 2011, Journal of diabetes investigation.

Nuclear Transport of Kir/Gem Requires Specific Signals and Importin α5 and Is Regulated by Calmodulin and Predicted Serine Phosphorylations

W. Hunziker, N. Inagaki, R. Mahalakshmi, 2007, Traffic.

Title:Glycemic variability is associated with quality of life and treatment satisfaction in patients with type 1 diabetes Running title: Glycemic variability and patient-reported outcomes

N. Inagaki, A. Hamasaki, K. Toyoda, 2018 .

Clinical and functional characterization of the Pro1198Leu ABCC8 gene mutation associated with permanent neonatal diabetes mellitus

K. Nanjo, M. Nishi, H. Furuta, 2013, Journal of diabetes investigation.

Whole-exome sequencing in a Japanese family with highly aggregated diabetes identifies a candidate susceptibility mutation in ADAMTSL3.

F. Matsuda, Y. Tabara, A. Koizumi, 2018, Diabetes research and clinical practice.

Nuclear sequestration of beta-subunits by Rad and Rem is controlled by 14-3-3 and calmodulin and reveals a novel mechanism for Ca2+ channel regulation.

Yuichiro Yamada, Y. Seino, W. Hunziker, 2006, Journal of molecular biology.

Roles of 14-3-3 and calmodulin binding in subcellular localization and function of the small G-protein Rem2.

Yuichiro Yamada, Y. Seino, W. Hunziker, 2005, The Biochemical journal.

Direct Inhibition of the Interaction between α-Interaction Domain and β-Interaction Domain of Voltage-dependent Ca2+ Channels by Gem*

T. Shibasaki, S. Seino, P. Béguin, 2005, Journal of Biological Chemistry.

Direct inhibition of the interaction between alpha-interaction domain and beta-interaction domain of voltage-dependent Ca2+ channels by Gem.

T. Shibasaki, S. Seino, P. Béguin, 2005, The Journal of biological chemistry.

Paternal allelic mutation at the Kcnq1 locus reduces pancreatic β-cell mass by epigenetic modification of Cdkn1c

Y. Kido, M. Kasuga, S. Seino, 2015, Proceedings of the National Academy of Sciences.

Sulfonylurea and glinide reduce insulin content, functional expression of K(ATP) channels, and accelerate apoptotic beta-cell death in the chronic phase.

Yuichiro Yamada, Y. Seino, N. Inagaki, 2007, Diabetes research and clinical practice.

GPR40 activation initiates store-operated Ca2+ entry and potentiates insulin secretion via the IP3R1/STIM1/Orai1 pathway in pancreatic β-cells

T. Manabe, Y. Baba, T. Kurosaki, 2019, Scientific Reports.

SUIT, secretory units of islets in transplantation: An index for therapeutic management of islet transplanted patients and its application to type 2 diabetes.

Yuichiro Yamada, Y. Seino, N. Inagaki, 2006, Diabetes research and clinical practice.

The structure and function of the ATP-sensitive K+ channel in insulin-secreting pancreatic beta-cells.

T. Miki, K. Nagashima, S. Seino, 1999, Journal of molecular endocrinology.

Reduction of Reactive Oxygen Species Ameliorates Metabolism-Secretion Coupling in Islets of Diabetic GK Rats by Suppressing Lactate Overproduction

N. Inagaki, E. Mukai, Y. Tahara, 2013, Diabetes.

GCKR mutations in Japanese families with clustered type 2 diabetes.

A. Koizumi, Kouji H. Harada, N. Inagaki, 2011, Molecular genetics and metabolism.

Self‐monitoring of blood glucose (SMBG) improves glycaemic control in oral hypoglycaemic agent (OHA)‐treated type 2 diabetes (SMBG‐OHA study)

Y. Seino, N. Inagaki, N. Harada, 2013, Diabetes/metabolism research and reviews.

Metformin suppresses hepatic gluconeogenesis and lowers fasting blood glucose levels through reactive nitrogen species in mice

E. Mukai, K. Toyoda, M. Ogura, 2010, Diabetologia.

Color record in self-monitoring of blood glucose improves glycemic control by better self-management.

K. Hosoda, N. Inagaki, N. Harada, 2014, Diabetes technology & therapeutics.