A. da Costa

Development of an automated malaria discriminant factor using VCS technology.

Ilse Aucamp, S. Machin, C. Briggs, 2006, American journal of clinical pathology.

GLUT4, AMP kinase, but not the insulin receptor, are required for hepatoportal glucose sensor-stimulated muscle glucose utilization.

C. Kahn, M. Birnbaum, P. Vollenweider, 2003, The Journal of clinical investigation.

Glut9 is a major regulator of urate homeostasis and its genetic inactivation induces hyperuricosuria and urate nephropathy

B. Thorens, D. Firsov, O. Bonny, 2009, Proceedings of the National Academy of Sciences.

Glucose competence of the hepatoportal vein sensor requires the presence of an activated glucagon-like peptide-1 receptor.

D. Drucker, R. Burcelin, B. Thorens, 2001, Diabetes.

Hepatic glucose sensing is required to preserve β cell glucose competence.

F. Schütz, M. Foretz, B. Thorens, 2013, The Journal of clinical investigation.

Urate-induced acute renal failure and chronic inflammation in liver-specific Glut9 knockout mice.

M. Daudon, Dominique Bazin, A. Dessombz, 2013, American journal of physiology. Renal physiology.