N. Dalla Venezia

发表

Molecular genetics of hereditary elliptocytosis and hereditary spherocytosis.

P. Maillet, L. Morlé, J. Delaunay, 1996, Annales de genetique.

Cell cycle regulation of the BRCA1/acetyl-CoA-carboxylase complex.

H. Ray, F. Suau, N. Dalla Venezia, 2009, Biochemical and biophysical research communications.

Caspase-dependent BRCA1 cleavage facilitates chemotherapy-induced apoptosis

Eva Dizin, T. Voeltzel, H. Ray, 2008, Apoptosis.

Studies on the hexose region of the lipopolysaccharide from a low virulence strain of Salmonella typhimurium.

P. Binder, R. Fontanges, N. Dalla Venezia, 1988, FEMS microbiology immunology.

Lipopolysaccharides from Yersinia pestis. Studies on lipid A of lipopolysaccharides I and II.

H. Mayer, S. Minka, N. Dalla Venezia, 1985, European journal of biochemistry.

Serine79-phosphorylated acetyl-CoA carboxylase, a downstream target of AMPK, localizes to the mitotic spindle poles and the cytokinesis furrow

J. Menéndez, A. Vázquez-Martín, S. Cufí, 2013, Cell cycle.

Isolation of an enzyme catalysing nitrosamine formation in Pseudomonas aeruginosa and Neisseria mucosae.

H. Bartsch, N. Dalla Venezia, S. Calmels, 1990, Biochemical and biophysical research communications.

Protein 4.1 deficiency associated with an altered binding to the spectrin-actin complex of the red cell membrane skeleton.

J. Delaunay, N. Alloisio, F. Baklouti, 1994, The Journal of clinical investigation.

Differential expression and subcellular localization of murine BRCA1 and BRCA1‐δ11 isoforms in murine and human cell lines

G. Lenoir, S. Mazoyer, R. Bachelier, 2000 .

Evidence that red blood cell protein p55 may participate in the skeleton-membrane linkage that involves protein 4.1 and glycophorin C.

A. Chishti, K. Andrabi, J. Delaunay, 1993, Blood.

Emerging Role of Eukaryote Ribosomes in Translational Control

V. Marcel, F. Catez, N. Dalla Venezia, 2019, International journal of molecular sciences.

Ribosome biogenesis: An emerging druggable pathway for cancer therapeutics

D. Lafontaine, V. Marcel, F. Catez, 2019, Biochemical pharmacology.

Alteration of ribosome function upon 5-fluorouracil treatment favors cancer cell drug-tolerance

J. Saurin, M. Bertrand, V. Marcel, 2022, Nature Communications.

Alteration of ribosome function upon 5-fluorouracil treatment favours cancer cell drug-tolerance

J. Saurin, V. Marcel, J. Guitton, 2020, bioRxiv.

An alpha-spectrin mutation responsible for hereditary elliptocytosis associated in cis with the alpha v/41 polymorphism.

J. Delaunay, D. Dhermy, M. Garbarz, 1993, Human genetics.

Elliptocytosis in patients with C-terminal domain mutations of protein 4.1 correlates with encoded messenger RNA levels rather than with alterations in primary protein structure.

F. Delhommeau, L. Ribeiro, P. Maillet, 2000, Blood.

Influence of Nucleoshuttling of the ATM Protein in the Healthy Tissues Response to Radiation Therapy: Toward a Molecular Classification of Human Radiosensitivity.

J. Bourhis, D. Stoppa-Lyonnet, A. Puisieux, 2016, International journal of radiation oncology, biology, physics.

Translational reprogramming of colorectal cancer cells induced by 5-fluorouracil through a miRNA-dependent mechanism

Didier Auboeuf, Julien Textoris, Alexandre David, 2017, Oncotarget.

Protein 4.1R expression in normal and dystrophic skeletal muscle.

F. Delhommeau, M. Fardeau, P. Leclerc, 2005, Comptes rendus biologies.

Low glucose microenvironment of normal kidney cells stabilizes a subset of messengers involved in angiogenesis

B. Eymin, X. Leroy, Elodie de Laplanche, 2015, Physiological reports.

Homozygous 4.1(-) hereditary elliptocytosis associated with a point mutation in the downstream initiation codon of protein 4.1 gene.

E. Benz, J. Delaunay, F. Gilsanz, 1992, The Journal of clinical investigation.

Influence of Nucleoshuttling of the ATM Protein in the Healthy Tissues Response to Radiation Therapy: Toward a Molecular Classification of Human Radiosensitivity.

J. Bourhis, D. Stoppa-Lyonnet, A. Puisieux, 2016, International journal of radiation oncology, biology, physics.