Diana C. Albarado

发表

Reduced adipose tissue oxygenation in human obesity evidence for rarefaction, macrophage chemotaxis, and inflammation without an angiogenic response

Leanne M. Redman, Jennifer C. Rood, J. Rood, 2008 .

FGF21 Signals Protein Status to the Brain and Adaptively Regulates Food Choice and Metabolism

Stephen J. Simpson, H. Berthoud, H. Münzberg, 2019, Cell reports.

Insights into transcription enhancer factor 1 (TEF-1) activity from the solution structure of the TEA domain

Xiaolian Gao, G. Halder, Xiaolian Gao, 2006, Proceedings of the National Academy of Sciences.

Reduced Adipose Tissue Oxygenation in Human Obesity

J. Rood, L. Redman, Steven R Smith, 2009, Diabetes.

FGF21, not GCN2, influences bone morphology due to dietary protein restrictions

T. Schulz, A. Schürmann, Brad A. Goupil, 2019, Bone reports.

FGF21 is an endocrine signal of protein restriction.

G. Bray, L. Redman, H. Münzberg, 2014, The Journal of clinical investigation.

Activation of hepatic estrogen receptor-α increases energy expenditure by stimulating the production of fibroblast growth factor 21 in female mice

K. Korach, J. Lefante, F. Bonnet, 2019, Molecular metabolism.

Low protein-induced increases in FGF21 drive UCP1-dependent metabolic but not thermoregulatory endpoints

H. Berthoud, H. Münzberg, D. McDougal, 2017, Scientific Reports.

Metabolic Responses to Dietary Protein Restriction Require an Increase in FGF21 that Is Delayed by the Absence of GCN2.

H. Berthoud, H. Münzberg, T. Gettys, 2016, Cell reports.

Inactivation of C/ebp Homologous Protein-driven Immune-Metabolic Interactions Exacerbate Obesity and Adipose Tissue Leukocytosis*

V. Dixit, Y. Youm, K. Nguyen, 2014, The Journal of Biological Chemistry.

FGF21 is required for protein restriction to extend lifespan and improve metabolic health in male mice

H. Berthoud, H. Münzberg, A. Bartke, 2022, Nature communications.

Canonical Nlrp3 inflammasome links systemic low-grade inflammation to functional decline in aging.

D. Ingram, H. Münzberg, P. Pistell, 2013, Cell metabolism.

Hepatic autophagy contributes to the metabolic response to dietary protein restriction.

C. Morrison, T. Laeger, Diana C. Albarado, 2016, Metabolism: clinical and experimental.

Quantifying Biochemical Alterations in Brown and Subcutaneous White Adipose Tissues of Mice Using Fourier Transform Infrared Widefield Imaging

Owen Carmichael, P. He, C. Hirschmugl, 2017, Front. Endocrinol..

Skeletal muscle NAMPT is induced by exercise in humans.

T. Church, K. Conley, Steven R Smith, 2010, American journal of physiology. Endocrinology and metabolism.

Impaired coordination of nutrient intake and substrate oxidation in melanocortin-4 receptor knockout mice.

W. Richards, J. Stephens, Jianping Ye, 2004, Endocrinology.

Low protein-induced increases in FGF21 drive UCP1-dependent metabolic but not thermoregulatory endpoints

H. Berthoud, H. Münzberg, D. McDougal, 2017, Scientific Reports.

Molecular basis for proline- and arginine-rich peptide inhibition of proteasome.

M. Simons, Diana C. Albarado, A. Anbanandam, 2008, Journal of molecular biology.

Dynamic effects of dietary protein restriction on body weights, food consumption, and protein preference in C57BL/6J and Fgf21-KO mice.

C. Morrison, Redin A. Spann, S. Fernandez-Kim, 2022, Journal of the experimental analysis of behavior.

A Potential Structural Switch for Regulating DNA-Binding by TEAD Transcription Factors.

C. Vonrhein, C. Raman, Dong-Sun Lee, 2016, Journal of molecular biology.

Canonical Nlrp3 inflammasome links systemic low-grade inflammation to functional decline in aging.

D. Ingram, H. Münzberg, P. Pistell, 2013, Cell metabolism.

FGF21 is an endocrine signal of protein restriction.

G. Bray, L. Redman, H. Münzberg, 2014, The Journal of clinical investigation.

A Potential Structural Switch for Regulating DNA-Binding by TEAD Transcription Factors.

C. Vonrhein, C. Raman, Dong-Sun Lee, 2016, Journal of molecular biology.