J. Ma

发表

Antitumor Activity of TGF-β, Inhibitor is Dependent on the Microenvironment

Lingyun Li, G. Schreiner, S. Medicherla, 2007 .

myelodysplastic syndrome progenitors Inhibition of overactivated p38 MAPK can restore hematopoiesis in

A. Verma, S. Parmar, L. Platanias, 2013 .

From bloodjournal.hematologylibrary.org at PENN STATE UNIVERSITY on February 21, 2013. For personal use only.

A. Verma, S. Parmar, L. Platanias, 2005 .

A Selective p 38 Mitogen-Activated Protein Kinase Inhibitor Reverses Cartilage and Bone Destruction in Mice with Collagen-Induced Arthritis

H. Genant, Yebin Jiang, S. Medicherla, 2006 .

Selective inhibition of p38alpha MAPK improves cardiac function and reduces myocardial apoptosis in rat model of myocardial injury.

G. Schreiner, A. Protter, S. Chakravarty, 2006, American journal of physiology. Heart and circulatory physiology.

In vivo and In vitro Cells Enhances Immunogenicity of Murine and Human Glioma Kinase Inhibitor , Inhibits Growth and Invasiveness and Receptor I β SD-208 , a Novel Transforming Growth Factor

M. Weller, W. Wick, M. Platten, 2004 .

NXY-059, a novel free radical trapping compound, reduces cortical infarction after permanent focal cerebral ischemia in the rat

A. Buchan, K. R. Maples, J. Ma, 2001, Brain Research.

Neuroprotective and anti-inflammatory effects of myricetin 3-glucoside in a rat model of cerebral ischemia

J. Ma, C. Ye, Shengshou Ye, 2020, Tropical Journal of Pharmaceutical Research.

p38α Mitogen-Activated Protein Kinase Inhibition Improves Cardiac Function and Reduces Myocardial Damage in Isoproterenol-Induced Acute Myocardial Injury in Rats

G. Schreiner, A. Protter, A. Kapoun, 2004, Journal of cardiovascular pharmacology.

Cyanidin-3-galactoside from Aronia melanocarpa ameliorates PM10 induced pulmonary injury by modulating M1/M2 macrophage polarization and NRF2/Sirt1 MAPK signaling

Xing He, Xianjun Meng, J. Ma, 2021 .

Transforming growth factor-beta receptor type 1 (TGFbetaRI) kinase activity but not p38 activation is required for TGFbetaRI-induced myofibroblast differentiation and profibrotic gene expression.

S. Rennard, Ying Wang, F. Wen, 2006, Molecular Pharmacology.

A Selective p38α Mitogen-Activated Protein Kinase Inhibitor Reverses Cartilage and Bone Destruction in Mice with Collagen-Induced Arthritis

H. Genant, Yebin Jiang, S. Medicherla, 2006, Journal of Pharmacology and Experimental Therapeutics.

A selective p38 alpha mitogen-activated protein kinase inhibitor reverses cartilage and bone destruction in mice with collagen-induced arthritis.

H. Genant, Yebin Jiang, S. Medicherla, 2006, The Journal of pharmacology and experimental therapeutics.

The presence of B-type natriuretic peptide in burns and the responsiveness of fibroblasts to BNP: proof of principle.

A. Singer, H. Soroff, A. Protter, 2007, Academic emergency medicine : official journal of the Society for Academic Emergency Medicine.

Preventive and Therapeutic Potential of p38α-Selective Mitogen-Activated Protein Kinase Inhibitor in Nonobese Diabetic Mice with Type 1 Diabetes

G. Schreiner, S. Medicherla, A. Protter, 2006, Journal of Pharmacology and Experimental Therapeutics.

Preventive and Therapeutic Potential of p 38-Selective Mitogen-Activated Protein Kinase Inhibitor in Nonobese Diabetic Mice with Type 1 Diabetes

G. Schreiner, S. Medicherla, A. Protter, 2006 .

Selective p38α mitogen-activated protein kinase inhibitor attenuates lung inflammation and fibrosis in IL-13 transgenic mouse model of asthma

S. Medicherla, A. Protter, L. Higgins, 2008, Journal of asthma and allergy.

Selective p 38 α mitogen-activated protein kinase inhibitor attenuates lung in fl ammation and fi brosis in IL-13 transgenic mouse model of asthma

S. Medicherla, A. Protter, L. Higgins, 2008 .

Topical alpha-selective p38 MAP kinase inhibition reduces acute skin inflammation in guinea pig

M. Reddy, S. Medicherla, A. Protter, 2010, Journal of inflammation research.

SD-208, a Novel Transforming Growth Factor β Receptor I Kinase Inhibitor, Inhibits Growth and Invasiveness and Enhances Immunogenicity of Murine and Human Glioma Cells In vitro and In vivo

M. Weller, W. Wick, M. Platten, 2004, Cancer Research.

Genetic deletion of Fatty Acid Amide Hydrolase results in improved long-term outcome in chronic autoimmune encephalitis

J. Ma, C. Tham, Michael Webb, 2008, Neuroscience Letters.

p38α-Selective Mitogen-Activated Protein Kinase Inhibitor SD-282 Reduces Inflammation in a Subchronic Model of Tobacco Smoke-Induced Airway Inflammation

S. Medicherla, A. Protter, A. Kapoun, 2008, Journal of Pharmacology and Experimental Therapeutics.

Transforming Growth Factor-β Receptor Type 1 (TGFβRI) Kinase Activity but Not p38 Activation Is Required for TGFβRI-Induced Myofibroblast Differentiation and Profibrotic Gene Expression

S. Rennard, Ying Wang, F. Wen, 2006, Molecular Pharmacology.

Antagonism of the histamine H4 receptor reduces LPS-induced TNF production in vivo

R. Thurmond, Fuqu Yu, W. Fung-Leung, 2013, Inflammation Research.

Inhibiting TGF-beta signaling restores immune surveillance in the SMA-560 glioma model.

M. Weller, W. Wick, A. Protter, 2007, Neuro-oncology.

NXY-059 maintains Akt activation and inhibits release of cytochrome C after focal cerebral ischemia

B. Siesjö, K. R. Maples, P. Kanakaraj, 2002, Brain Research.

Role of p38 mitogen activated protein kinase in a model of osteosarcoma-induced pain

C. Svensson, T. Yaksh, Yebin Jiang, 2008, Pharmacology Biochemistry and Behavior.

Recombinant Vascular Endothelial Growth Factor 121 Attenuates Hypertension and Improves Kidney Damage in a Rat Model of Preeclampsia

N Stephen Pollitt, Ying Zhang, Frederick Sannajust, 2007, Hypertension.

Histamine H4 receptor antagonism diminishes existing airway inflammation and dysfunction via modulation of Th2 cytokines

R. Thurmond, J. Ma, J. Riley, 2010, Respiratory research.

Histamine H 4 receptor antagonism diminishes existing airway inflammation and dysfunction via modulation of Th 2 cytokines

R. Thurmond, Jason P. Riley, J. Ma, 2010 .

Inhibition of the TGF-β receptor I kinase promotes hematopoiesis in MDS. Commentary

M. Bitzer, A. Verma, S. Kambhampati, 2008 .

Normalizing the bone marrow microenvironment with p38 inhibitor reduces multiple myeloma cell proliferation and adhesion and suppresses osteoclast formation.

G. Roodman, N. Kurihara, G. Schreiner, 2006, Experimental cell research.

Antitumor activity of TGF-beta inhibitor is dependent on the microenvironment.

Lingyun Li, G. Schreiner, S. Medicherla, 2007, Anticancer research.

Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy

S. Medicherla, A. Protter, B. Cullen, 2009 .

p38 MAPK inhibition reduces diabetes-induced impairment of wound healing

S. Medicherla, A. Protter, B. Cullen, 2009, Diabetes, metabolic syndrome and obesity : targets and therapy.

Preventive and therapeutic potential of p38 alpha-selective mitogen-activated protein kinase inhibitor in nonobese diabetic mice with type 1 diabetes.

G. Schreiner, S. Medicherla, A. Protter, 2006, The Journal of pharmacology and experimental therapeutics.

Cyanidin-3-glucoside attenuates silica-induced pulmonary inflammatory responses by modulating T cell immune responses and STAT1/STAT3 signaling

Xianjun Meng, Qi Zhang, J. Ma, 2020 .

Antagonism of the histamine H4 receptor reduces LPS-induced TNF production in vivo

R. Thurmond, Fuqu Yu, W. Fung-Leung, 2013, Inflammation Research.

Transforming Growth Factor-β Receptor Type 1 (TGFβRI) Kinase Activity but Not p38 Activation Is Required for TGFβRI-Induced Myofibroblast Differentiation and Profibrotic Gene Expression

S. Rennard, Ying Wang, F. Wen, 2006, Molecular Pharmacology.

induced myofibroblast differentiation and pro-fibrotic gene expression

S. Rennard, Ying Wang, F. Wen, 2006 .

Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy

S. Medicherla, A. Protter, B. Cullen, 2009 .

DMSOTT-5859-p38 MAPK inhibition reduces diabetes-induced impair

S. Medicherla, A. Protter, B. Cullen, 2009 .

Topical alpha-selective p 38 MaP kinase inhibition reduces acute skin inflammation in guinea pig satyanarayana

M. Reddy, A. Protter, L. Higgins, 2010 .

p38α Mitogen-Activated Protein Kinase Inhibition Improves Cardiac Function and Reduces Myocardial Damage in Isoproterenol-Induced Acute Myocardial Injury in Rats

A. Protter, A. Kapoun, S. Chakravarty, 2004, Journal of cardiovascular pharmacology.

Inhibition of overactivated p38 MAPK can restore hematopoiesis in myelodysplastic syndrome progenitors.

A. Verma, S. Parmar, L. Platanias, 2006, Blood.