J. J. Out-Luiting

发表

Exon skipping of TGFβRI affects signalling and ECM expression in hypertrophic scar-derived fibroblasts

P. V. van Zuijlen, R. van Doorn, Marianna Kruithof-de Julio, 2020, Scars, burns & healing.

Reduced IL‐1Ra/IL‐1 ratio in ultraviolet B‐exposed skin of patients with polymorphic light eruption

F. D. de Gruijl, R. Willemze, J. J. Out-Luiting, 2009, Experimental dermatology.

GNAQ and GNA11 mutations and downstream YAP activation in choroidal nevi

M. Madigan, W. Bergman, R. Verdijk, 2017, British Journal of Cancer.

Reduced filaggrin expression is accompanied by increased Staphylococcus aureus colonization of epidermal skin models

P. Nibbering, E. M. Haisma, A. El Ghalbzouri, 2014, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

Presence of immunoreactive β‐endorphin in human skin

S. V. van Duinen, S. Duinen, J. J. Out-Luiting, 2001 .

Birth cohort-specific trends of sun-related behaviors among individuals from an international consortium of melanoma-prone families

S. Puig, D. Bishop, D. Elder, 2021, BMC Public Health.

Deep-sequencing analysis reveals that the miR-199a2/214 cluster within DNM3os represents the vast majority of aberrantly expressed microRNAs in Sézary syndrome.

H. Buermans, L. van der Fits, R. Willemze, 2012, The Journal of investigative dermatology.

Normalized ultraviolet (UV) induction of Langerhans cell depletion and neutrophil infiltrates after artificial UVB hardening of patients with polymorphic light eruption

F. D. de Gruijl, R. Willemze, J. J. Out-Luiting, 2005, The British journal of dermatology.

A novel mouse model for Sézary Syndrome using xenotransplantation of Sézary cells into immunodeficient RAG2−/− γc−/− mice

M. Vermeer, J. J. Out-Luiting, C. Tensen, 2012, Experimental dermatology.

The effect of TGFβRI inhibition on fibroblast heterogeneity in hypertrophic scar 2D in vitro models.

P. V. van Zuijlen, R. van Doorn, Marianna Kruithof-de Julio, 2021, Burns : journal of the International Society for Burn Injuries.

A meta-analysis of gene expression data identifies a molecular signature characteristic for tumor-stage mycosis fungoides.

R. Willemze, R. van Doorn, M. Vermeer, 2012, The Journal of investigative dermatology.

Evaluation of Immunophenotypic and Molecular Biomarkers for Sézary Syndrome Using Standard Operating Procedures: A Multicenter Study of 59 Patients.

M. Felcht, C. Klemke, L. van der Fits, 2016, The Journal of investigative dermatology.

MicroRNA-21 expression in CD4+ T cells is regulated by STAT3 and is pathologically involved in Sézary syndrome.

L. van der Fits, R. Willemze, Yongjun Qin, 2011, The Journal of investigative dermatology.

Aberrant Expression of the Tyrosine Kinase Receptor EphA4 and the Transcription Factor Twist in Sézary Syndrome Identified by Gene Expression Analysis

R. Willemze, R. van Doorn, M. Vermeer, 2004, Cancer Research.

Novel and highly recurrent chromosomal alterations in Sézary syndrome.

Remco Dijkman, Karl-Johan Leuchowius, Ulf Landegren, 2008, Cancer research.

Accurate Quantification of T Cells by Measuring Loss of Germline T-Cell Receptor Loci with Generic Single Duplex Droplet Digital PCR Assays.

A. Langerak, P. A. van der Velden, M. Versluis, 2017, The Journal of molecular diagnostics : JMD.

Deregulation of JAK2 signaling underlies primary cutaneous CD8+ aggressive epidermotropic cytotoxic T-cell lymphoma

H. Mei, D. Fanoni, E. Berti, 2021, Haematologica.

Lgr6+ stem cells and their progeny in mouse epidermis under regimens of exogenous skin carcinogenesis, and their absence in ensuing skin tumors

F. D. de Gruijl, J. J. Out-Luiting, W. Zoutman, 2016, Oncotarget.

Genome-Wide Analysis of Gene and Protein Expression of Dysplastic Naevus Cells

N. Gruis, W. Bergman, P. Hensbergen, 2012, Journal of skin cancer.

Autocrine IL-21 stimulation is involved in the maintenance of constitutive STAT3 activation in Sézary syndrome.

L. van der Fits, R. Willemze, J. Samsom, 2012, The Journal of investigative dermatology.

Cucurbitacin E and I target the JAK/STAT pathway and induce apoptosis in Sézary cells

M. Vermeer, J. J. Out-Luiting, C. Tensen, 2020, Biochemistry and biophysics reports.

Exploring the IL-21-STAT3 axis as therapeutic target for Sézary syndrome.

L. van der Fits, M. Vermeer, J. J. Out-Luiting, 2014, The Journal of investigative dermatology.

Cucurbitacin I inhibits Stat3 and induces apoptosis in Sézary cells.

R. Willemze, P. A. von dem Borne, M. Vermeer, 2008, The Journal of investigative dermatology.

Presence of immunoreactive beta-endorphin in human skin.

S. V. van Duinen, J. J. Out-Luiting, B. Vermeer, 2001, Experimental dermatology.

EPHA4 is overexpressed but not functionally active in Sézary syndrome

G. Siegal, I. D. de Esch, L. van der Fits, 2015, Oncotarget.

Improved Sézary cell detection and novel insights into immunophenotypic and molecular heterogeneity in Sézary syndrome.

A. Órfão, J. V. van Dongen, C. Teodosio, 2021, Blood.

NOTCH1 signaling as a therapeutic target in Sézary syndrome.

J. van Es, L. van der Fits, S. Whittaker, 2012, The Journal of investigative dermatology.

MicroRNA-21 expression in CD4+ T cells is regulated by STAT3 and is pathologically involved in Sézary syndrome.

L. van der Fits, R. Willemze, Yongjun Qin, 2011, The Journal of investigative dermatology.

NOTCH1 signaling as a therapeutic target in Sézary syndrome.

J. van Es, L. van der Fits, S. Whittaker, 2012, The Journal of investigative dermatology.