Luyun Zou

发表

Assessing bioenergetic function in response to oxidative stress by metabolic profiling.

A. Landar, Jianhua Zhang, J. Chatham, 2011, Free radical biology & medicine.

Adult rat cardiomyocytes exhibit capacitative calcium entry.

R. Marchase, D. Hunton, Y. Pang, 2004, American journal of physiology. Heart and circulatory physiology.

Acute increases in O-GlcNAc indirectly impair mitochondrial bioenergetics through dysregulation of LonP1-mediated mitochondrial protein complex turnover.

Jianhua Zhang, W. Wani, J. Chatham, 2019, American journal of physiology. Cell physiology.

THE PROTECTIVE EFFECTS OF PUGNAC ON CARDIAC FUNCTION AFTER TRAUMA-HEMORRHAGE ARE MEDIATED VIA INCREASED PROTEIN O-GlcNAc LEVELS

R. Marchase, I. Chaudry, J. Chatham, 2007, Shock.

Importance of the bioenergetic reserve capacity in response to cardiomyocyte stress induced by 4-hydroxynonenal.

J. Chatham, V. Darley-Usmar, B. Hill, 2009, The Biochemical journal.

The Identification of a Novel Calcium-Dependent Link Between NAD+ and Glucose Deprivation-Induced Increases in Protein O-GlcNAcylation and ER Stress

Jianhua Zhang, J. Chatham, V. Darley-Usmar, 2021, Frontiers in Molecular Biosciences.

Aging Influences Cardiac Mitochondrial Gene Expression and Cardiovascular Function following Hemorrhage Injury

I. Chaudry, J. Chatham, Bixi Jian, 2011, Molecular medicine.

Novel role of the ER/SR Ca2+ sensor STIM1 in the regulation of cardiac metabolism.

S. Litovsky, J. Chatham, M. Young, 2019, American journal of physiology. Heart and circulatory physiology.

Stromal interaction molecule 1 is essential for normal cardiac homeostasis through modulation of ER and mitochondrial function.

R. Marchase, S. Litovsky, J. Chatham, 2014, American journal of physiology. Heart and circulatory physiology.

High-fat, low-carbohydrate diet promotes arrhythmic death and increases myocardial ischemia-reperfusion injury in rats.

L. Dell’Italia, S. Lloyd, S. Litovsky, 2014, American journal of physiology. Heart and circulatory physiology.

Glucose Deprivation-induced Increase in Protein O-GlcNAcylation in Cardiomyocytes Is Calcium-dependent*

R. Marchase, A. Paterson, J. Chatham, 2012, The Journal of Biological Chemistry.

GLUCOSAMINE ADMINISTRATION DURING RESUSCITATION IMPROVES ORGAN FUNCTION AFTER TRAUMA HEMORRHAGE

R. Marchase, I. Chaudry, J. Chatham, 2006, Shock.

O-GlcNAcylation, Novel Post-Translational Modification Linking Myocardial Metabolism and Cardiomyocyte Circadian Clock*

J. Dyck, K. Gamble, M. Bray, 2011, The Journal of Biological Chemistry.

Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O-GlcNAcylation and attenuation of NF-{kappa}B signaling.

R. Marchase, I. Chaudry, J. Chatham, 2009, American journal of physiology. Heart and circulatory physiology.

Modification of STIM1 by O-linked N-Acetylglucosamine (O-GlcNAc) Attenuates Store-operated Calcium Entry in Neonatal Cardiomyocytes*

R. Marchase, J. Chatham, Luyun Zou, 2012, The Journal of Biological Chemistry.

In isolated cardiomyocytes NF‐κB activation is modulated by alterations in protein O‐GlcNAc levels

R. Marchase, J. Chatham, V. Champattanachai, 2008 .

The Protective Effect of Glucosamine on Cardiac Function Following Trauma Hemorrhage: Downregulation of cardiac NF‐κB signaling

R. Marchase, I. Chaudry, J. Chatham, 2007 .

Aging Influences Cardiac Mitochondrial Gene Expression and Cardiovascular Function following Hemorrhage Injury

I. Chaudry, J. Chatham, Bixi Jian, 2011, Molecular medicine.

Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O-GlcNAcylation and attenuation of NF-{kappa}B signaling.

R. Marchase, I. Chaudry, J. Chatham, 2009, American journal of physiology. Heart and circulatory physiology.

Sustained Increases in Cardiomyocyte Protein O‐Linked β‐N‐Acetylglucosamine Levels Lead to Cardiac Hypertrophy and Reduced Mitochondrial Function Without Systolic Contractile Impairment

Chae-Myeong Ha, Jianhua Zhang, J. Chatham, 2023, Journal of the American Heart Association.