François Houle

p38 MAP kinase activation by vascular endothelial growth factor mediates actin reorganization and cell migration in human endothelial cells

Jacques Landry, J. Landry, J. Huot, 1997, Oncogene.

SAPK2/p38-dependent F-Actin Reorganization Regulates Early Membrane Blebbing during Stress-induced Apoptosis

Jacques Landry, G. Shah, J. Landry, 1998, The Journal of cell biology.

Vascular Endothelial Growth Factor (VEGF)-driven Actin-based Motility Is Mediated by VEGFR2 and Requires Concerted Activation of Stress-activated Protein Kinase 2 (SAPK2/p38) and Geldanamycin-sensitive Phosphorylation of Focal Adhesion Kinase*

Jacques Landry, Johannes Waltenberger, J. Landry, 2000, The Journal of Biological Chemistry.

Phosphorylation of tyrosine 1214 on VEGFR2 is required for VEGF-induced activation of Cdc42 upstream of SAPK2/p38

J. Huot, F. Houle, G. Jourdan, 2004, Oncogene.

Survival advantages conferred to colon cancer cells by E-selectin-induced activation of the PI3K-NFκB survival axis downstream of Death receptor-3

R. Klinck, J. Huot, F. Houle, 2011, BMC Cancer.