K. Katsaros

发表

Association of Small Dense LDL Serum Levels and Circulating Monocyte Subsets in Stable Coronary Artery Disease

G. Maurer, K. Huber, J. Wojta, 2015, PloS one.

In Human Macrophages the Complement Component C5a Induces the Expression of Oncostatin M via AP-1 Activation

G. Maurer, K. Huber, T. Afonyushkin, 2008, Arteriosclerosis, thrombosis, and vascular biology.

Monocyte subset distribution in patients with stable atherosclerosis and elevated levels of lipoprotein(a)

G. Maurer, K. Huber, J. Wojta, 2015, Journal of clinical lipidology.

An increase of VEGF plasma levels is associated with restenosis of drug-eluting stents.

G. Maurer, K. Huber, J. Wojta, 2014, EuroIntervention : journal of EuroPCR in collaboration with the Working Group on Interventional Cardiology of the European Society of Cardiology.

The complement component C5a is present in human coronary lesions in vivo and induces the expression of MMP‐1 and MMP‐9 in human macrophages in vitro

J. Badimón, G. Maurer, K. Huber, 2011, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Clopidogrel pretreatment abolishes increase of PAI-1 after coronary stent implantation.

G. Maurer, K. Huber, J. Wojta, 2008, Thrombosis research.

Glycoprotein 130 polymorphism predicts soluble glycoprotein 130 levels.

G. Maurer, K. Huber, J. Wojta, 2014, Metabolism: clinical and experimental.

Monday, 27 August 2012

V. V. Ivanenko, E. A. Medvedeva, Michail I. Papafaklis, 2012 .

The adipokine vaspin is associated with decreased coronary in-stent restenosis in vivo and inhibits migration of human coronary smooth muscle cells in vitro

K. Huber, J. Wojta, C. Kaun, 2020, PloS one.

Thrombin induces the expression of oncostatin M via AP-1 activation in human macrophages: a link between coagulation and inflammation.

G. Maurer, K. Huber, J. Wojta, 2009, Blood.

Oncostatin M-enhanced vascular endothelial growth factor expression in human vascular smooth muscle cells involves PI3K-, p38 MAPK-, Erk1/2- and STAT1/STAT3-dependent pathways and is attenuated by interferon-γ

G. Maurer, K. Huber, T. Afonyushkin, 2011, Basic Research in Cardiology.

Coronary late lumen loss of drug eluting stents is associated with increased serum levels of the complement components C3a and C5a.

G. Maurer, K. Huber, J. Wojta, 2010, Atherosclerosis.

G-CSF Predicts Cardiovascular Events in Patients with Stable Coronary Artery Disease

G. Maurer, K. Huber, S. Farhan, 2015, PloS one.

Premature myocardial infarction is associated with low serum levels of Wnt-1.

G. Maurer, K. Huber, J. Wojta, 2012, Atherosclerosis.

Small high-density lipoprotein is associated with monocyte subsets in stable coronary artery disease

G. Maurer, K. Huber, J. Wojta, 2014, Atherosclerosis.

An increase of interleukin-33 serum levels after coronary stent implantation is associated with coronary in-stent restenosis

K. Huber, S. Farhan, J. Wojta, 2014, Cytokine.

Increased restenosis rate after implantation of drug-eluting stents in patients with elevated serum activity of matrix metalloproteinase-2 and -9.

G. Maurer, K. Huber, J. Wojta, 2010, JACC. Cardiovascular interventions.

High soluble Fas and soluble Fas Ligand serum levels before stent implantation are protective against restenosis

G. Maurer, K. Huber, D. Glogar, 2011, Thrombosis and Haemostasis.

Soluble ST2 and Interleukin-33 Levels in Coronary Artery Disease: Relation to Disease Activity and Adverse Outcome

K. Huber, S. Farhan, J. Wojta, 2014, PloS one.

Plasminogen activator inhibitor‐1 predicts coronary in‐stent restenosis of drug‐eluting stents

G. Maurer, J. Wojta, D. Glogar, 2008, Journal of thrombosis and haemostasis : JTH.