M. Wambach

发表

Inhibition of Double-Stranded RNA- and Tumor Necrosis Factor Alpha-Mediated Apoptosis by Tetratricopeptide Repeat Protein and Cochaperone P58IPK

M. Katze, B. Williams, M. J. Korth, 1999, Molecular and Cellular Biology.

Translational regulation by the interferon-induced double-stranded-RNA-activated 68-kDa protein kinase.

M. Katze, A. Hinnebusch, G. Barber, 1993, Proceedings of the National Academy of Sciences of the United States of America.

Nontranslated cellular mRNAs are associated with the cytoskeletal framework in influenza virus or adenovirus infected cells.

M. Katze, M. Wambach, J. Lara, 1989, Virology.

Cloning, expression, and cellular localization of the oncogenic 58-kDa inhibitor of the RNA-activated human and mouse protein kinase.

M. Katze, M. J. Korth, M. Wambach, 1996, Gene.

Functional expression and RNA binding analysis of the interferon-induced, double-stranded RNA-activated, 68,000-Mr protein kinase in a cell-free system

M. Katze, B. Williams, G. Barber, 1991, Molecular and cellular biology.

Regulation of Interferon-Induced Protein Kinase PKR: Modulation of P58IPK Inhibitory Function by a Novel Protein, P52rIPK

M. Katze, P. Romano, M. Gale, 1998, Molecular and Cellular Biology.

Binding of the influenza virus NS1 protein to double-stranded RNA inhibits the activation of the protein kinase that phosphorylates the elF-2 translation initiation factor.

M. Katze, R. Krug, M G Katze, 1995, Virology.

Interaction of the interferon-induced PKR protein kinase with inhibitory proteins P58IPK and vaccinia virus K3L is mediated by unique domains: implications for kinase regulation

M. Katze, S. Tan, M. Gale, 1996, Molecular and cellular biology.

Mutants of the RNA-dependent protein kinase (PKR) lacking double-stranded RNA binding domain I can act as transdominant inhibitors and induce malignant transformation

M. Katze, G. Barber, R. Jagus, 1995, Molecular and cellular biology.

The Cellular Inhibitor of the PKR Protein Kinase, P58IPK, Is an Influenza Virus-activated Co-chaperone That Modulates Heat Shock Protein 70 Activity*

M. Katze, Seng-Lai Tan, R. Morimoto, 1999, The Journal of Biological Chemistry.

Katze domains : implications for kinase regulation . and vaccinia virus K 3 L is mediated by unique protein kinase with inhibitory proteins P 58 IPK Interaction of the interferon-induced PKR

M. Katze, Seng-Lai Tan, M. Gale, 1996 .

Expression of cellular genes in CD4 positive lymphoid cells infected by the human immunodeficiency virus, HIV-1: evidence for a host protein synthesis shut-off induced by cellular mRNA degradation.

M. Katze, M. Wambach, M. Agy, 1990, Virology.

The P58 Cellular Inhibitor Complexes with the Interferon-induced, Double-stranded RNA-dependent Protein Kinase, PKR, to Regulate Its Autophosphorylation and Activity (*)

M. Katze, G. Barber, S. Polyak, 1996, The Journal of Biological Chemistry.