J. Kabarowski

ts BCR-ABL kinase activation confers increased resistance to genotoxic damage via cell cycle block.

M. Greaves, M. Greaves, L. Wiedemann, 1996, Oncogene.

A temperature sensitive p210 BCR‐ABL mutant defines the primary consequences of BCR‐ABL tyrosine kinase expression in growth factor dependent cells.

L. Wiedemann, J. Kabarowski, P. B. Allen, 1994, The EMBO journal.

Novel BTB/POZ domain zinc-finger protein, LRF, is a potential target of the LAZ-3/BCL-6 oncogene

D. Leprince, A. Zelent, G. Morriss-Kay, 1999, Oncogene.

Leukemia-associated retinoic acid receptor alpha fusion partners, PML and PLZF, heterodimerize and colocalize to nuclear bodies.

J M Davies, A Zelent, J. Licht, 1997, Proceedings of the National Academy of Sciences of the United States of America.