Kurt A. Krummel

Mouse Mutants Reveal that Putative Protein Interaction Sites in the p53 Proline-Rich Domain Are Dispensable for Tumor Suppression

G. Wahl, F. Toledo, K. Krummel, 2006, Molecular and Cellular Biology.

Gatekeepers of the Guardian: p53 Regulation by Post-Translational Modification, MDM2 and MDMX

G. Wahl, J. Stommel, M. Wade, 2007 .

A mouse p53 mutant lacking the proline-rich domain rescues Mdm4 deficiency and provides insight into the Mdm2-Mdm4-p53 regulatory network.

G. Wahl, F. Toledo, K. Krummel, 2006, Cancer cell.

The common fragile site FRA16D and its associated gene WWOX are highly conserved in the mouse at Fra8E1

E. Calhoun, K. Krummel, Kurt A. Krummel, 2002, Genes, chromosomes & cancer.

Fine-tuning p53 activity through C-terminal modification significantly contributes to HSC homeostasis and mouse radiosensitivity.

G. Wahl, A. Balmain, H. Piwnica-Worms, 2011, Genes & development.

The characterization of the common fragile site FRA16D and its involvement in multiple myeloma translocations.

L. Roberts, T. Glover, K. Krummel, 2000, Genomics.

The C-terminal lysines fine-tune P53 stress responses in a mouse model but are not required for stability control or transactivation.

G. Wahl, F. Toledo, K. Krummel, 2005, Proceedings of the National Academy of Sciences of the United States of America.