J. Aguiar

SCA2 trinucleotide expansion in German SCA patients

S. Pulst, K. Wessel, L. Schöls, 1997, Neurogenetics.

Prophylactic use of epidermal growth factor reduces ischemia/reperfusion intestinal damage.

R. Playford, J. Boyle, P. López-Saura, 2002, The American journal of pathology.

Localization of the candidate gene D-amino acid oxidase outside the refined I-cM region of spinocerebellar ataxia 2.

A. Brice, S. Gispert, I. Torréns, 1995, American journal of human genetics.

Identification of the physiological promoter for spinocerebellar ataxia 2 gene reveals a CpG island for promoter activity situated into the exon 1 of this gene and provides data about the origin of the nonmethylated state of these types of islands.

W. Schulz, G. Auburger, C. Alexander, 1999, Biochemical and biophysical research communications.

Ubiquitous expression of human SCA2 gene under the regulation of the SCA2 self promoter cause specific Purkinje cell degeneration in transgenic mice

G. Guillén, J. Aguiar, N. Merino, 2006, Neuroscience Letters.

Characterization of a new transgenic mouse model of the spinocerebellar ataxia type 2 disease

J. Aguiar, J. Berlanga, J. Fernández, 2006 .

Bone marrow stromal cells produce nerve growth factor and glial cell line-derived neurotrophic factors.

N. Pavón, J. Aguiar, Rocío García, 2004, Biochemical and biophysical research communications.

Evaluation of the neurorestorative effects of the murine beta-nerve growth factor infusions in old rat with cognitive deficit.

J. Aguiar, Carmen Mejías, W. Almaguer, 2003, Biochemical and biophysical research communications.