W. Kirlin

发表

Cysteine/cystine couple is a newly recognized node in the circuitry for biologic redox signaling and control

Dean P Jones, Young-Mi Go, Thomas R Ziegler, 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Shift work influences the outcomes of Chlamydia infection and pathogenesis

Jason P. DeBruyne, Yang Fengxia, F. Eko, 2020, Scientific Reports.

Polymorphism of CYP2D6 in Black populations: implications for psychopharmacology.

L. D. Bradford, W. Kirlin, Dianne Bradford, 1998, The international journal of neuropsychopharmacology.

Variation in the response of T cells to concanavalin A after in vitro exposure to benzo[A]pyrene and 2-aminofluorene.

W. Kirlin, M. Lee, M. Sturrup, 1996, Immunopharmacology and immunotoxicology.

SELDI-TOF-MS Analysis of Transcriptional Activation Protein Binding to Response Elements Regulating Carcinogenesis Enzymes

M. Powell, W. Kirlin, S. Bischoff, 2002 .

Nanoparticle-linked antagonist for leptin signaling inhibition in breast cancer

Lily Yang, R. Gonzalez-Perez, W. Kirlin, 2017, World journal of clinical oncology.

Acetylator genotype-dependent expression of arylamine N-acetyltransferase in human colon cytosol from non-cancer and colorectal cancer patients.

D. Hein, W. Kirlin, R. J. Ferguson, 1991, Cancer research.

Maternal exposure to benzo[a]pyrene alters development of T lymphocytes in offspring.

T. Hodge, W. Kirlin, P. Urso, 1999, Immunopharmacology and immunotoxicology.

and Colorectal Cancer Patients N-Acetyltransferase in Human Colon Cytosol from Non-Cancer Acetylator Genotype-dependent Expression of Arylamine

W. Kirlin, F. Ogolla, A. F. Andrews, 2013 .

- Acetyltransferase O-Hydroxyarylamine-N Hamster : Identity with Polymorphic-Acetyltransferase from Homozygous Rapid Acetylator Inbred N Purification of Hepatic Polymorphic Arylamine Updated

T. Rustan, D. Hein, W. Kirlin, 2006 .

Purification of hepatic polymorphic arylamine N-acetyltransferase from homozygous rapid acetylator inbred hamster: identity with polymorphic N-hydroxyarylamine-O-acetyltransferase.

T. Rustan, D. Hein, W. Kirlin, 1990, Cancer research.

Human acetylator genotype: Relationship to colorectal cancer incidence and arylamine N-acetyltransferase expression in colon cytosol

T. Rustan, M. Doll, D. Hein, 2005, Archives of Toxicology.

Prostacyclin-induced hypothermia: involvement of central histamine H2-receptors.

P. N. Kaul, W. Kirlin, S. Kandasamy, 1981, Life sciences.

Phytochemical Induction of Cell Cycle Arrest by Glutathione Oxidation and Reversal by N-Acetylcysteine in Human Colon Carcinoma Cells

K. Jackson, W. Kirlin, M. Dansby, 2007, Nutrition and cancer.

Overexpression of heat shock factor 1 inhibits butyrate-induced differentiation in colon cancer cells

Dean P. Jones, Jiyang Cai, A. Sartorelli, 2006, Cell stress & chaperones.

Glutathione redox potential in response to differentiation and enzyme inducers.

Dean P. Jones, Jiyang Cai, S. Thompson, 1999, Free radical biology & medicine.

DETECTION OF DNA ADDUCTS IN DEVELOPING CD4+CD8+ THYMOCYTES AND SPLENOCYTES FOLLOWING IN UTERO EXPOSURE TO BENZO[a]PYRENE

W. Kirlin, L. King, M. Kohan, 2002, Immunopharmacology and immunotoxicology.

Polymorphic expression of acetyl coenzyme A-dependent arylamine N-acetyltransferase and acetyl coenzyme A-dependent O-acetyltransferase-mediated activation of N-hydroxyarylamines by human bladder cytosol.

P. K. Brady, D. Hein, W. Kirlin, 1989, Cancer research.

Dietary compounds that induce cancer preventive phase 2 enzymes activate apoptosis at comparable doses in HT29 colon carcinoma cells.

Dean P. Jones, Jiyang Cai, D. P. Jones, 1999, The Journal of nutrition.

Effect of Time of Day of Infection on Chlamydia Infectivity and Pathogenesis

Jason P. DeBruyne, Tarek Adnan Ahmad, F. Eko, 2019, Scientific Reports.

Oxidation of the glutathione/glutathione disulfide redox state is induced by cysteine deficiency in human colon carcinoma HT29 cells.

Dean P. Jones, W. Watson, T. Ziegler, 2002, The Journal of nutrition.

Acetylator genotype-dependent expression of arylamine N-acetyltransferase and N-hydroxyarylamine O-acetyltransferase in Syrian inbred hamster intestine and colon. Identity with the hepatic acetylation polymorphism.

D. Hein, W. Kirlin, R. J. Ferguson, 1990, Drug metabolism and disposition: the biological fate of chemicals.

Kinetic characterization of acetylator genotype-dependent and -independent N-acetyltransferase isozymes in homozygous rapid and slow acetylator inbred hamster liver cytosol.

P. K. Brady, D. Hein, W. Kirlin, 1989, Drug metabolism and disposition: the biological fate of chemicals.

Identification and kinetic characterization of acetylator genotype-dependent and -independent arylamine carcinogen N-acetyltransferases in hamster bladder cytosol.

P. K. Brady, D. Hein, W. Kirlin, 1989, Drug metabolism and disposition: the biological fate of chemicals.

Acetylator genotype-dependent metabolic activation of carcinogenic N-hydroxyarylamines by S-acetyl coenzyme A-dependent enzymes of inbred hamster tissue cytosols: relationship to arylamine N-acetyltransferase.

D. Hein, W. Kirlin, T. Flammang, 1987, Carcinogenesis.

Inheritance of acetylator genotype-dependent arylamine N-acetyltransferase in hamster bladder cytosol.

D. Hein, W. Kirlin, T. Yerokun, 1987, Carcinogenesis.

Extrahepatic expression of N-acetylator genotype in the inbred hamster.

D. Hein, W. Kirlin, F. Ogolla, 1987, Drug metabolism and disposition: the biological fate of chemicals.

Biochemical investigation of the basis for the genetic N-acetylation polymorphism in the inbred hamster.

W. Weber, D. Hein, W. Kirlin, 1985, The Journal of pharmacology and experimental therapeutics.

Toxicity Studies Related to Medicinal Plants

W. Kirlin, S. Bose, R. Datta, 2021, Evidence Based Validation of Traditional Medicines.

Exosomes derived from Induced Vascular Progenitor Cells Promote Angiogenesis in vitro and in an in vivo Rat Hindlimb Ischemia Model.

Dihan Zhu, W. Chilian, W. Kirlin, 2019, American journal of physiology. Heart and circulatory physiology.

Permeation of roxarsone and its metabolites increases caco-2 cell proliferation.

K. Jackson, L. Hammonds-Odie, G. S. Bayse, 2013, Advances in biological chemistry.

Genetic control of acetyl coenzyme A-dependent arylamine N-acetyltransferase, hydrazine N-acetyltransferase, and N-hydroxy-arylamine O-acetyltransferase enzymes in C57BL/6J, A/J, AC57F1, and the rapid and slow acetylator A.B6 and B6.A congenic inbred mouse.

W. Weber, D. Hein, W. Kirlin, 1988, Drug metabolism and disposition: the biological fate of chemicals.