O. Witte

PROMM and deafness: exclusion of ZNF9 as the disease gene in DFNA18 suggests a polygenic origin of the PROMM/DM2 phenotype

R. Lang-Roth, T. Deufel, A. Lamprecht-Dinnesen, 2002, Clinical genetics.

A gene for autosomal dominant paroxysmal choreoathetosis/spasticity (CSE) maps to the vicinity of a potassium channel gene cluster on chromosome 1p, probably within 2 cM between D1S443 and D1S197.

F. Binkofski, W. Heindel, H. Kugel, 1996, Genomics.

[Long QT syndrome causing grand mal epilepsy: case report, pedigree, therapeutic options, and review of the literature].

O. Witte, E. Schulze-Bahr, C. Fitzek, 2006, Der Nervenarzt.

High frequency of partial SPAST deletions in autosomal dominant hereditary spastic paraplegia

O. Witte, R. Schüle, L. Schöls, 2006, Neurology.

[Cortical dysgenesis: current classification, MRI diagnosis, and clinical review].

G. Hagemann, O. Witte, C. Redecker, 2000, Der Nervenarzt.

Distribution of glutamate receptor subunits in experimentally induced cortical malformations

G. Hagemann, O. Witte, H. Luhmann, 2003, Neuroscience.

Recurrent posterior reversible encephalopathy syndrome (PRES)

G. Hagemann, O. Witte, C. Fitzek, 2004, Journal of Human Hypertension.