J. Wang

发表

Fibrilization in mouse senile amyloidosis is fibril conformation-dependent.

T. Chiba, M. Hosokawa, T. Matsushita, 1998, Laboratory investigation; a journal of technical methods and pathology.

Body mass index and hepatic lipase gene (LIPC) polymorphism jointly influence postheparin plasma hepatic lipase activity.

S. Grundy, G. Vega, J. Cohen, 1998, Journal of lipid research.

Overexpression of human apolipoprotein C-III in transgenic mice results in an accumulation of apolipoprotein B48 remnants that is corrected by excess apolipoprotein E.

J. Taylor, R. Mahley, K. Weisgraber, 1994, The Journal of biological chemistry.

High plasma levels of apo(a) fragments in Caucasians and African‐Americans with end‐stage renal disease: implications for plasma Lp(a) assay

H. Hobbs, V. Mooser, S. Marcovina, 1997, Clinical genetics.

Accelerated senile amyloidosis induced by amyloidogenic Apoa-II gene shortens the life span of mice but does not accelerate the rate of senescence.

K. Kitagawa, M. Hosokawa, T. Matsushita, 1996, The journals of gerontology. Series A, Biological sciences and medical sciences.

Mouse senile amyloid deposition is suppressed by adenovirus-mediated overexpression of amyloid-resistant apolipoprotein A-II.

J. Miyazaki, T. Chiba, M. Hosokawa, 1999, The American journal of pathology.

The Apo(a) gene is the major determinant of variation in plasma Lp(a) levels in African Americans.

H. Hobbs, V. Mooser, S. Marcovina, 1997, American journal of human genetics.

Wild type ApoA-II gene does not rescue senescence-accelerated mouse (SAMP1) from short life span and accelerated mortality.

T. Chiba, M. Hosokawa, T. Matsushita, 2000, The journals of gerontology. Series A, Biological sciences and medical sciences.

Regulation of the metabolism of plasma lipoproteins by apolipoprotein A-II.

K. Kitagawa, M. Hosokawa, T. Matsushita, 1997, Biochimica et biophysica acta.

Overexpression of hepatic lipase in transgenic rabbits leads to a marked reduction of plasma high density lipoproteins and intermediate density lipoproteins.

J. Taylor, R. Mahley, Jianglin Fan, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Low density lipoprotein receptor-negative mice expressing human apolipoprotein B-100 develop complex atherosclerotic lesions on a chow diet: no accentuation by apolipoprotein(a).

R. Hammer, H. Hobbs, V. Mooser, 1998, Proceedings of the National Academy of Sciences of the United States of America.

A hepatic lipase (LIPC) allele associated with high plasma concentrations of high density lipoprotein cholesterol.

S. Grundy, J. Cohen, J. Wang, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Accumulation of pro-apolipoprotein A-II in mouse senile amyloid fibrils.

T. Chiba, M. Hosokawa, T. Matsushita, 1997, The Biochemical journal.