J. Cardenas

发表

Overexpression of the Cell Cycle Inhibitor p16INK4a Promotes a Prothrombotic Phenotype Following Vascular Injury in Mice

N. Sharpless, J. Krishnamurthy, A. P. Owens, 2011, Arteriosclerosis, thrombosis, and vascular biology.

Post-translational oxidative modification of fibrinogen is associated with coagulopathy after traumatic injury.

K. Ward, C. Wade, D. Brophy, 2016, Free radical biology & medicine.

Thrombin Generation Following Severe Trauma: Mechanisms, Modulators, and Implications for Hemostasis and Thrombosis

J. Cardenas, 2021, Shock.

Establishment of Methods for Performing Thrombelastography and Calibrated Automated Thrombography in Rats

C. Wade, J. Holcomb, J. Salsbury, 2014, Shock.

Measuring thrombin generation as a tool for predicting hemostatic potential and transfusion requirements following trauma

C. Wade, J. Holcomb, B. Cotton, 2014, The journal of trauma and acute care surgery.

Endotheliopathy Is Associated With a 24-Hour Fibrinolysis Phenotype Described by Low TEG Lysis and High d-Dimer After Trauma

C. Wade, J. Holcomb, J. Kerby, 2022, Annals of surgery open : perspectives of surgical history, education, and clinical approaches.

Treating the endotheliopathy of SARS‐CoV‐2 infection with plasma: Lessons learned from optimized trauma resuscitation with blood products

J. Holcomb, M. Schreiber, R. Kozar, 2021, Transfusion.

TEG Lysis Shutdown Represents Coagulopathy in Bleeding Trauma Patients: Analysis of the PROPPR Cohort

C. Wade, J. Holcomb, M. Schreiber, 2019, Shock.

Older Blood Is Associated With Increased Mortality and Adverse Events in Massively Transfused Trauma Patients: Secondary Analysis of the PROPPR Trial.

Samuel M. Galvagno, Henry E. Wang, A. Arkin, 2019, Annals of emergency medicine.

Early versus late venous thromboembolism: A secondary analysis of data from the PROPPR trial.

M. Rosengart, C. Wade, J. Holcomb, 2019, Surgery.

Traumatic brain injury is associated with increased syndecan-1 shedding in severely injured patients

Ryan S. Kitagawa, J. Holcomb, C. Cox, 2018, Scandinavian Journal of Trauma, Resuscitation and Emergency Medicine.

Platelet transfusions improve hemostasis and survival in a substudy of the prospective, randomized PROPPR trial.

E. Fox, C. Wade, J. Holcomb, 2018, Blood advances.

Traumatic Endotheliopathy: A Prospective Observational Study of 424 Severely Injured Patients

C. Wade, J. Holcomb, B. Cotton, 2016, Annals of surgery.

Time to plasma transfusion: a patient centered approach and modifiable risk factor

J. Holcomb, J. Cardenas, 2017, Transfusion.

Impact of blood products on platelet function in patients with traumatic injuries: a translational study.

E. Fox, C. Wade, J. Holcomb, 2017, The Journal of surgical research.

Early Fibrinolysis Associated with Hemorrhagic Progression Following Traumatic Brain Injury

Ryan S. Kitagawa, C. Wade, J. Holcomb, 2017, Shock.

Reply to: is "Thrombin Burst" Now the Worst Option in Trauma?

C. Wade, A. Cap, J. Cardenas, 2017, Shock.

Plasma Resuscitation Promotes Coagulation Homeostasis Following Shock-Induced Hypercoagulability

C. Wade, J. Holcomb, M. Swartz, 2016, Shock.

Microvesicle phenotypes are associated with transfusion requirements and mortality in subjects with severe injuries

C. Wade, J. Holcomb, R. Kozar, 2015, Journal of extracellular vesicles.

Trauma, Time, and Transfusions: A Longitudinal Analysis of Coagulation Markers in Severely Injured Trauma Patients Receiving Modified Whole Blood or Component Blood Products

M. Cohen, C. Wade, J. Holcomb, 2015, Shock.

Antithrombin III Contributes to the Protective Effects of Fresh Frozen Plasma Following Hemorrhagic Shock by Preventing Syndecan-1 Shedding and Endothelial Barrier Disruption.

C. Wade, T. Ko, R. Kozar, 2020, Shock.

Causal relationship between hyperfibrinogenemia, thrombosis, and resistance to thrombolysis in mice.

A. Wolberg, F. Church, K. Machlus, 2011, Blood.

Protease-Activated Receptor 1 Contributes to Angiotensin II-Induced Cardiovascular Remodeling and Inflammation

N. Mackman, R. Pawlinski, F. Church, 2016, Cardiology.

Acquired antithrombin deficiency is a risk factor for venous thromboembolism after major trauma.

C. Wade, B. Cotton, Elaheh Rahbar, 2021, Thrombosis research.

THE PREHOSPITAL USE OF YOUNGER AGE WHOLE BLOOD IS ASSOCIATED WITH AN IMPROVED ARRIVAL COAGULATION PROFILE.

C. Wade, B. Cotton, J. Cardenas, 2020, The journal of trauma and acute care surgery.

Hemostatic potential of cold-stored non-leukoreduced whole blood over time: An assessment of platelet function and thrombin generation for optimal shelf life

C. Wade, B. Cotton, J. Cardenas, 2020, The journal of trauma and acute care surgery.

Overview of Blood Coagulation and the Pathophysiology of Blood Coagulation Disorders

F. Church, C. M. Rein-Smith, J. Cardenas, 2016 .

Multiplate and TEG platelet mapping in a population of severely injured trauma patients

C. Wade, B. Gill, N. White, 2018, Transfusion medicine.

Absences of Endothelial Microvesicle Changes in the Presence of the Endotheliopathy of Trauma

C. Wade, J. Holcomb, B. Cotton, 2019, Shock.

The controversial role of the urokinase system in abdominal aortic aneurysm formation and rupture.

F. Church, Chantelle M. Rein, J. Cardenas, 2011, Arteriosclerosis, thrombosis, and vascular biology.

Murine models do not recapitulate the pathophysiology of age‐related venous thrombosis in humans

A. Wolberg, F. Church, H. Whinna, 2013, Journal of thrombosis and haemostasis : JTH.

Association of Changes in Antithrombin Activity Over Time With Responsiveness to Enoxaparin Prophylaxis and Risk of Trauma-Related Venous Thromboembolism.

E. Fox, C. Wade, B. Cotton, 2022, JAMA surgery.

Supplementation with antithrombin III ex vivo optimizes enoxaparin responses in critically injured patients.

C. Wade, B. Cotton, A. Cap, 2020, Thrombosis research.

Early diagnosis of clinically significant hyperfibrinolysis using thrombelastography velocity curves.

C. Wade, J. Holcomb, M. Goodman, 2014, Journal of the American College of Surgeons.

Obesity and the balancing act of endothelial damage and repair: Commentary on an article by Maria-Victoria Noci, MD, et al.: "Changes in endothelial microparticles and endothelial progenitor cells in obese patients in response to surgical stress".

C. Wade, J. Cardenas, 2015, The Journal of bone and joint surgery. American volume.

Commentary & Perspective Obesity and the Balancing Act of Endothelial Damage and Repair

C. Wade, J. Cardenas, M. Noci, 2015 .

Hypercoagulability after energy drink consumption.

C. Wade, J. Holcomb, B. Cotton, 2015, The Journal of surgical research.

The Impact of Rapid Infuser Use on the Platelet Count, Platelet Function, and Hemostatic Potential of Whole Blood.

B. Cotton, J. Cardenas, D. Meyer, 2020, The Journal of surgical research.

Alterations in heparan sulfate proteoglycan synthesis and sulfation and the impact on vascular endothelial function

J. Cardenas, J. R. Richter, Robert P. Richter, 2022, Matrix biology plus.

Mechanisms of Traumatic Hyperfibrinolysis and Implications for Antifibrinolytic Therapy

J. Cardenas, 2019, Current Trauma Reports.

Green Plasma Has a Superior Hemostatic Profile Compared With Standard Color Plasma

C. Wade, B. Cotton, J. Cardenas, 2022, The American surgeon.

Upon admission coagulation and platelet function in patients with thermal and electrical injuries.

C. Wade, J. Holcomb, B. Cotton, 2016, Burns : journal of the International Society for Burn Injuries.

In vitro efficacy of RiaSTAP after rapid reconstitution.

C. Wade, J. Holcomb, B. Cotton, 2014, The Journal of surgical research.

Sympathoadrenal activation and endotheliopathy are drivers of hypocoagulability and hyperfibrinolysis in trauma: A prospective observational study of 404 severely injured patients

C. Wade, J. Holcomb, B. Cotton, 2017, The journal of trauma and acute care surgery.

Mechanisms of trauma-induced coagulopathy

C. Wade, J. Holcomb, J. Cardenas, 2014, Current opinion in hematology.

Hypocoagulability in Traumatic Brain Injury as Measured by Traditional Means and Thrombelastography

Ryan S. Kitagawa, C. Wade, J. Holcomb, 2015 .

Syndecan-1: A Quantitative Marker for the Endotheliopathy of Trauma.

C. Wade, J. Holcomb, B. Cotton, 2017, Journal of the American College of Surgeons.

Endothelial glycocalyx shedding and vascular permeability in severely injured trauma patients

J. Holcomb, S. Ostrowski, P. Johansson, 2015, Journal of Translational Medicine.

Cellular microparticle and thrombogram phenotypes in the Prospective Observational Multicenter Major Trauma Transfusion (PROMMTT) study: correlation with coagulopathy.

E. Fox, M. Cohen, C. Wade, 2014, Thrombosis research.

A Reply to "Carriage Before the Horse", a Letter to the Editor in Reference to "TEG Lysis Shutdown Represents Coagulopathy In Bleeding Trauma Patients: Analysis of the PROPPR Cohort" (2019; 51 (3): 273: 283).

C. Wade, N. White, J. Cardenas, 2019, Shock.

Tumor-derived tissue factor activates coagulation and enhances thrombosis in a mouse xenograft model of human pancreatic cancer.

J. Yeh, J. Rak, D. Kirchhofer, 2012, Blood.

Platelet-derived- Extracellular Vesicles Promote Hemostasis and Prevent the Development of Hemorrhagic Shock

C. Wade, J. Holcomb, A. Srivastava, 2019, Scientific Reports.

Early Identification of the Patient with Endotheliopathy of Trauma by Arrival Serum Albumin

C. Wade, J. Holcomb, B. Cotton, 2017, Shock.

Traumatic Endotheliopathy

J. Holcomb, B. Cotton, L. Baer, 2017 .

Prevalence and impact of admission hyperfibrinolysis in severely injured pediatric trauma patients.

B. Cotton, M. Harting, Ioannis N. Liras, 2015, Surgery.

Unique Contribution of Haptoglobin and Haptoglobin Genotype in Aneurysmal Subarachnoid Hemorrhage

J. Grotta, Peeyush Kumar, S. Blackburn, 2018, Front. Physiol..

Antithrombin Activity Is Associated with Persistent Thromboinflammation and Mortality in Patients with Severe COVID-19 Illness

B. Cotton, J. Podbielski, J. Cardenas, 2022, Acta Haematologica.

Advances in the understanding of trauma-induced coagulopathy.

C. Wade, J. Holcomb, J. Cardenas, 2016, Blood.

Elevated Tissue Plasminogen Activator and Reduced Plasminogen Activator Inhibitor Promote Hyperfibrinolysis in Trauma Patients

C. Wade, J. Holcomb, B. Cotton, 2014, Shock.

Acute Inflammation in Traumatic Brain Injury and Polytrauma Patients Using Network Analysis.

C. Wade, J. Holcomb, Jay Karri, 2019, Shock.

PLATELET FUNCTION IN TRAUMA: IS CURRENT TECHNOLOGY IN FUNCTION TESTING MISSING THE MARK IN INJURED PATIENTS?

B. Gill, S. Olson, J. Cardenas, 2022, Shock.

Tranexamic Acid and Safety in the Right Patient.

A. Sauaia, J. Holcomb, J. Cardenas, 2021, JAMA surgery.

Endothelial glycocalyx shedding and vascular permeability in severely injured trauma patients

C. Wade, J. Holcomb, R. Bruhn, 2015, Journal of Translational Medicine.

A 3-O-sulfated heparan sulfate dodecasaccharide (12-mer) suppresses thromboinflammation and attenuates early organ injury following trauma and hemorrhagic shock

K. Arnold, Yongmei Xu, Jian Liu, 2023, Frontiers in Immunology.

Hydrogen Sulfide Ameliorates SARS-CoV-2-Associated Lung Endothelial Barrier Disruption

Alexander N. Freiberg, J. Cardenas, Katalin Módis, 2023, Biomedicines.

Soluble endothelial protein C receptor is an independent predictor of venous thromboembolism after severe injury: Secondary analysis of a prospective cohort study.

B. Cotton, J. Cardenas, Kelly E. Sanders, 2023, Surgery.

Radiofrequency Ablation Remodels the Tumor Microenvironment and Promotes Neutrophil-Mediated Abscopal Immunomodulation in Pancreatic Cancer

N. Thosani, J. Rowe, C. Wray, 2022, Cancer immunology research.

Predicting Arterial Thrombotic Events Following Peripheral Revascularization Using Objective Viscoelastic Data

Natalie Sumetsky, Samuel Jessula, M. Eagleton, 2022, Journal of the American Heart Association : Cardiovascular and Cerebrovascular Disease.

What's New in Shock, July 2021?

J. Cardenas, J. Krocker, 2021, Shock.

Predicting Futility in Severely Injured Patients: Using Arrival Lab Values and Physiology to Support Evidence-Based Resource Stewardship.

L. Kao, B. Cotton, J. Cardenas, 2023, Journal of the American College of Surgeons.