R. Ayon

发表

Molecular and Functional Significance of Ca2+-Activated Cl− Channels in Pulmonary Arterial Smooth Muscle

N. Leblanc, C. Singer, F. Britton, 2015, Pulmonary circulation.

Endothelial platelet-derived growth factor-mediated activation of smooth muscle platelet-derived growth factor receptors in pulmonary arterial hypertension

D. Valdez-Jasso, P. Thistlethwaite, J. Garcia, 2020, Pulmonary circulation.

Endothelial HIF-2α contributes to severe pulmonary hypertension due to endothelial-to-mesenchymal transition.

T. Zhou, J. Yuan, Haiyang Tang, 2017, American journal of physiology. Lung cellular and molecular physiology.

Chloroquine is a potent pulmonary vasodilator that attenuates hypoxia‐induced pulmonary hypertension

J. Garcia, J. Yuan, Zhihao Liang, 2017, British journal of pharmacology.

-independent signaling mechanism 2+ a Camuscle cells contract in response to fluid flow via

J. Tarbell, Shanshan Song, A. Makino, 2015 .

Flow shear stress enhances intracellular Ca2+ signaling in pulmonary artery smooth muscle cells from patients with pulmonary arterial hypertension.

J. Yuan, Haiyang Tang, Shanshan Song, 2014, American journal of physiology. Cell physiology.

Notch Enhances Ca2+ Entry by Activating Calcium-sensing Receptors and Inhibiting Voltage-gated K+ Channels.

P. Thistlethwaite, J. Yuan, Shanshan Song, 2020, American journal of physiology. Cell physiology.

MicroRNA-mediated Downregulation of K+ Channels in Pulmonary Arterial Hypertension.

P. Thistlethwaite, J. Yuan, A. Makino, 2019, American journal of physiology. Lung cellular and molecular physiology.

Chronic hypoxia selectively enhances L- and T-type voltage-dependent Ca2+ channel activity in pulmonary artery by upregulating Cav1.2 and Cav3.2.

J. Yuan, Haiyang Tang, A. Makino, 2013, American journal of physiology. Lung cellular and molecular physiology.

Functional characterization of voltage-dependent Ca(2+) channels in mouse pulmonary arterial smooth muscle cells: divergent effect of ROS.

J. Yuan, Haiyang Tang, A. Makino, 2013, American journal of physiology. Cell physiology.

SGLT inhibitors attenuate NO-dependent vascular relaxation in the pulmonary artery but not in the coronary artery.

J. Yuan, A. Makino, Young-Eun Cho, 2015, American journal of physiology. Lung cellular and molecular physiology.

Increased TMEM16A-encoded calcium-activated chloride channel activity is associated with pulmonary hypertension.

N. Leblanc, C. Singer, D. Duan, 2012, American journal of physiology. Cell physiology.

Increased TMEM 16 A-encoded calcium-activated chloride channel activity is associated with pulmonary hypertension

N. Leblanc, C. Singer, D. Duan, 2012 .

Endothelial Upregulation of Mechanosensitive Channel Piezo1 in Pulmonary Hypertension.

D. Valdez-Jasso, P. Thistlethwaite, J. Garcia, 2021, American journal of physiology. Cell physiology.

Transcriptomic profiles in pulmonary arterial hypertension associate with disease severity and identify novel candidate genes

M. Gonzalez-Garay, J. Garcia, Xinshuai Qi, 2020, Pulmonary circulation.

Calcium-Sensing Receptor Regulates Cytosolic [Ca2+] and Plays a Major Role in the Development of Pulmonary Hypertension

J. Yuan, Haiyang Tang, A. Makino, 2016, Front. Physiol..

Pathogenic Role of mTORC1 and mTORC2 in Pulmonary Hypertension

J. Garcia, J. Yuan, Haiyang Tang, 2018, JACC. Basic to translational science.

Hypoxia selectively upregulates cation channels and increases cytosolic [Ca2+] in pulmonary, but not coronary, arterial smooth muscle cells.

W. Wier, J. Yuan, Shanshan Song, 2018, American journal of physiology. Cell physiology.

STIM2 (Stromal Interaction Molecule 2)-Mediated Increase in Resting Cytosolic Free Ca2+ Concentration Stimulates PASMC Proliferation in Pulmonary Arterial Hypertension.

J. Yuan, Shanshan Song, A. Makino, 2018, Hypertension.

RESEARCH ARTICLE Ion Channels and Transporters in Lung Function and Disease Capsaicin-induced Ca 2 (cid:2) signaling is enhanced via upregulated TRPV1 channels in pulmonary artery smooth muscle cells from patients with idiopathic PAH

J. Yuan, Haiyang Tang, Shanshan Song, 2017 .

Closeup New insights into the pathology of pulmonary hypertension : implication of the miR-210 / ISCU 1 / 2 / FeS Axis

J. Yuan, Haiyang Tang, R. Ayon, 2015 .

Gasping for answers. Focus on "Calpain activation by ROS mediates human ether-a-go-go-related gene protein degradation by intermittent hypoxia".

J. Yuan, Haiyang Tang, R. Ayon, 2016, American journal of physiology. Cell physiology.

Mutant hERG channel traffic jam. Focus on "Pharmacological correction of long QT-linked mutations in KCNH2 (hERG) increases the trafficking of Kv11.1 channels stored in the transitional endoplasmic reticulum".

J. Yuan, R. Ayon, Ruby A. Fernandez, 2013, American journal of physiology. Cell physiology.

Phosphorylation alters the pharmacology of Ca2+‐activated Cl‐ channels in rabbit pulmonary arterial smooth muscle cells

N. Leblanc, R. Ayon, M. Wiwchar, 2009, British journal of pharmacology.

Ryanodine receptor-2: a necessity for gating store-operated Ca2+ channels.

J. Yuan, Shanshan Song, R. Ayon, 2016, Cardiovascular research.

Capsaicin-induced Ca2+ signaling is enhanced via upregulated TRPV1 channels in pulmonary artery smooth muscle cells from patients with idiopathic PAH.

J. Yuan, Haiyang Tang, Shanshan Song, 2017, American journal of physiology. Lung cellular and molecular physiology.

Divergent changes of p53 in pulmonary arterial endothelial and smooth muscle cells involved in the development of pulmonary hypertension.

J. Yuan, Haiyang Tang, Shanshan Song, 2019, American journal of physiology. Lung cellular and molecular physiology.

Endothelial dysfunction in pulmonary arterial hypertension: an evolving landscape (2017 Grover Conference Series)

J. Yuan, S. Bonnet, S. Chan, 2017, Pulmonary circulation.

Pathogenic role of calcium-sensing receptors in the development and progression of pulmonary hypertension.

T. Zhou, D. Fraidenburg, J. Yuan, 2016, American journal of physiology. Lung cellular and molecular physiology.

PFKFB3-mediated endothelial glycolysis promotes pulmonary hypertension

M. Zhang, Yunchao Su, S. Barman, 2019, Proceedings of the National Academy of Sciences.

Smooth Muscle Cell Ion Channels in Pulmonary Arterial Hypertension: Pathogenic Role in Pulmonary Vasoconstriction and Vascular Remodeling

J. Yuan, Haiyang Tang, A. Makino, 2016 .

Molecular Mechanism of TMEM16A Regulation: Role of CaMKII and PP1/PP2A.

N. Leblanc, C. Singer, I. Greenwood, 2019, American journal of physiology. Cell physiology.

New insights into the pathology of pulmonary hypertension: implication of the miR-210/ISCU1/2/Fe-S axis

J. Yuan, Haiyang Tang, R. Ayon, 2015, EMBO molecular medicine.

SGLT inhibitors attenuate NO‐dependent vascular relaxation in mouse pulmonary artery (677.3)

J. Yuan, A. Makino, Young-Eun Cho, 2014 .

development and progression of pulmonary hypertension Pathogenic role of calcium-sensing receptors in the

T. Zhou, J. Yuan, Haiyang Tang, 2016 .

Complex Phosphatase Regulation of Ca2+-activated Cl− Currents in Pulmonary Arterial Smooth Muscle Cells*

N. Leblanc, William R. Sones, I. Greenwood, 2009, The Journal of Biological Chemistry.

Endothelial platelet-derived growth factor-mediated activation of smooth muscle platelet-derived growth factor receptors in pulmonary arterial hypertension

D. Valdez-Jasso, P. Thistlethwaite, J. Yuan, 2020, Pulmonary circulation.

Endothelial HIF-2α contributes to severe pulmonary hypertension due to endothelial-to-mesenchymal transition.

T. Zhou, J. Yuan, Haiyang Tang, 2017, American journal of physiology. Lung cellular and molecular physiology.