Michael Martin

发表

Sphingosine Kinase-1 Is Required for Toll Mediated β-Defensin 2 Induction in Human Oral Keratinocytes

D. Kinane, Jiawei Zhao, M. Benakanakere, 2010, PloS one.

Toll-Like Receptor-Mediated Production of IL-1Ra Is Negatively Regulated by GSK3 via the MAPK ERK1/21

Carlos A. Garcia, D. Kinane, Michael Martin, 2009, The Journal of Immunology.

Mammalian Target of Rapamycin Complex 2 (mTORC2) Negatively Regulates Toll-like Receptor 4-mediated Inflammatory Response via FoxO1*

J. Suttles, D. Graves, Michael Martin, 2011, The Journal of Biological Chemistry.

Mammalian target of rapamycin (mTOR) regulates TLR3 induced cytokines in human oral keratinocytes.

D. Kinane, K. Hosur, Jiawei Zhao, 2010, Molecular immunology.

Convergence of the Mammalian Target of Rapamycin Complex 1- and Glycogen Synthase Kinase 3-β–Signaling Pathways Regulates the Innate Inflammatory Response

Carlos A. Garcia, R. Jope, Ruqiang Liang, 2011, The Journal of Immunology.

IFN-β Production by TLR4-Stimulated Innate Immune Cells Is Negatively Regulated by GSK3-β1

Carlos A. Garcia, T. Mitchell, D. Kinane, 2008, The Journal of Immunology.

Antigenic Experience Dictates Functional Role of Glycogen Synthase Kinase-3 in Human CD4+ T Cell Responses1

Carlos A. Garcia, D. Kinane, M. Benakanakere, 2008, The Journal of Immunology.

b by GSK3-Innate Immune Cells Is Negatively Regulated Production by TLR4-Stimulated b IFN-Michael

Carlos A. Garcia, T. Mitchell, D. Kinane, 2008 .

c-Jun Controls the Ability of IL-12 to Induce IL-10 Production from Human Memory CD4+ T Cells1

Carlos A. Garcia, D. Kinane, M. Benakanakere, 2009, The Journal of Immunology.

This information is current as the MAPK ERK 1 / 2 IL-1 Ra Is Negatively Regulated by GSK 3 via Toll-Like Receptor-Mediated Production of Kinane and

Carlos A. Garcia, D. Kinane, Michael Martin, 2008 .

Significant upregulation of U1 and U4 spliceosomal snRNAs by ATP nanoliposomes explains acceleration of wound healing, due to increased pre-mRNA processing to functional mRNA.

G. Kotwal, Michael Martin, S. Chien, 2018, Nanomedicine : nanotechnology, biology, and medicine.

Characterization of ATP Nanoliposome Treatment for Regeneration of Injured Spinal Cord

G. Kotwal, E. Hattab, Michael Martin, 2019, Nanotechnology Characterization Tools for Tissue Engineering and Medical Therapy.

IFN-beta production by TLR4-stimulated innate immune cells is negatively regulated by GSK3-beta.

Caglar Cekic, T. Mitchell, D. Kinane, 2008, Journal of immunology.

Glycogen synthase kinase 3: a point of convergence for the host inflammatory response.

Michael Martin, Huizhi Wang, Jonathan Brown, 2011, Cytokine.

Toll-like receptor–mediated cytokine production is differentially regulated by glycogen synthase kinase 3

R. Jope, S. Michalek, Michael Martin, 2005, Nature Immunology.

The Role of Glycogen Synthase Kinase 3 in Regulating IFN-β–Mediated IL-10 Production

Yunan Tang, D. Kinane, M. Benakanakere, 2011, The Journal of Immunology.

Toll-Like Receptor-Mediated Production of IL-1Ra Is Negatively Regulated by GSK3 via the MAPK ERK1/2

D. Kinane, Michael Martin, Huizhi Wang, 2008 .

Cotinine-induced convergence of the cholinergic and PI3 kinase-dependent anti-inflammatory pathways in innate immune cells.

D. Renaud, D. Scott, Michael Martin, 2008, Biochimica et biophysica acta.

Treponema denticola Activates Mitogen-Activated Protein Kinase Signal Pathways through Toll-Like Receptor 2

Michael Martin, J. Ruby, Kunal Rehani, 2007, Infection and Immunity.

TLR-signaling Networks

Hong Wang, G. Hajishengallis, Michael Martin, 2011, Journal of dental research.

The role of glycogen synthase kinase 3-beta in interferon beta biology

J. Suttles, T. Mitchell, D. Scott, 2018 .

Critical Reviews in Oral Biology & Medicine: TLR-signaling Networks: An Integration of Adaptor Molecules, Kinases, and Cross-talk

Hong Wang, G. Hajishengallis, Michael Martin, 2011 .

TLR-signaling Networks

Michael Martin, Jonathan Brown, Huizhi Wang, 2011, Journal of dental research.

Cotinine-induced convergence of the cholinergic and PI3 kinase-dependent anti-inflammatory pathways in innate immune cells.

D. Renaud, D. Scott, Michael Martin, 2008, Biochimica et biophysica acta.