Hongyan Wang

发表

Low-level overexpression of wild type TDP-43 causes late-onset, progressive neurodegeneration and paralysis in mice

H. Cai, Jia Yu, Chunxing Yang, 2022, PloS one.

Low-level overexpression of wild type TDP-43 causes late-onset, progressive neurodegeneration and paralysis in mice

Robert H. Brown, H. Cai, Jia Yu, 2021, bioRxiv.

Several rAAV vectors efficiently cross the blood-brain barrier and transduce neurons and astrocytes in the neonatal mouse central nervous system.

C. Mueller, M. Sena-Esteves, G. Gao, 2011, Molecular therapy : the journal of the American Society of Gene Therapy.

A construct with fluorescent indicators for conditional expression of miRNA

Zuoshang Xu, Hongyan Wang, Hongxia Zhou, 2008, BMC biotechnology.

Asymmetrically designed siRNAs and shRNAs enhance the strand specificity and efficacy in RNAi

Hongyan Wang, Hongliu Ding, Yejin Zhou, 2007, Journal of RNAi and gene silencing : an international journal of RNA and gene targeting research.

Low-level overexpression of wild type TDP-43 causes late-onset, progressive neurodegeneration and paralysis in mice

Robert H. Brown, H. Cai, Jia Yu, 2021, bioRxiv.

Widespread aggregation of mutant VAPB associated with ALS does not cause motor neuron degeneration or modulate mutant SOD1 aggregation and toxicity in mice

Zuoshang Xu, Tao Qiao, Hongyan Wang, 2013, Molecular Neurodegeneration.

Identification of Human Monoclonal Antibodies Specific for Human SOD1 Recognizing Distinct Epitopes and Forms of SOD1

D. A. Bosco, C. Matthews, Can Kayatekin, 2013, PloS one.

Partial loss of TDP-43 function causes phenotypes of amyotrophic lateral sclerosis

Robert H. Brown, H. Cai, M. Moore, 2014, Proceedings of the National Academy of Sciences.

Nerve injection of viral vectors efficiently transfers transgenes into motor neurons and delivers RNAi therapy against ALS.

Chunyan Liu, M. Castro, Zuoshang Xu, 2009, Antioxidants & redox signaling.

Therapeutic Gene Silencing Delivered by a Chemically Modified Small Interfering RNA against Mutant SOD1 Slows Amyotrophic Lateral Sclerosis Progression*♦

T. Rana, Huricha Baigude, Zuoshang Xu, 2008, Journal of Biological Chemistry.

Global CNS transduction of adult mice by intravenously delivered rAAVrh.8 and rAAVrh.10 and nonhuman primates by rAAVrh.10.

R. Desrosiers, G. Gao, L. Zhong, 2014, Molecular therapy : the journal of the American Society of Gene Therapy.

Widespread spinal cord transduction by intrathecal injection of rAAV delivers efficacious RNAi therapy for amyotrophic lateral sclerosis.

Robert H. Brown, Chunxing Yang, G. Gao, 2014, Human molecular genetics.

The Analysis of Progranulin Mutations as a Causative Agent in Frontotemporal Dementia

Hongyan Wang, A. Major, Linghua Qiu, 2008 .

A construct with fluorescent indicators for conditional expression of miRNA

Zuoshang Xu, Hongyan Wang, Hongxia Zhou, 2008 .

Development of Excipient-Free Freeze-Dryable Unimolecular Hyperstar Polymers for Efficient siRNA Silencing.

Amol D. Punjabi, Xiaofeng Wang, Gang Han, 2017, ACS macro letters.

Several rAAV vectors efficiently cross the blood-brain barrier and transduce neurons and astrocytes in the neonatal mouse central nervous system.

M. Sena-Esteves, G. Gao, Zuoshang Xu, 2011, Molecular therapy : the journal of the American Society of Gene Therapy.

Therapeutic Gene Silencing Delivered by a Chemically Modified Small Interfering RNA against Mutant SOD1 Slows Amyotrophic Lateral Sclerosis Progression*♦

T. Rana, Huricha Baigude, Zuoshang Xu, 2008, Journal of Biological Chemistry.