A. Wright

Riluzole does not ameliorate disease caused by cytoplasmic TDP‐43 in a mouse model of amyotrophic lateral sclerosis

B. Shi, P. D. Della Gatta, A. Walker, 2021, The European journal of neuroscience.

Early activation of cellular stress and death pathways caused by cytoplasmic TDP-43 in the rNLS8 mouse model of ALS/FTD

F. Rigo, P. Jafar-Nejad, A. Walker, 2022, bioRxiv.

Splicing factor proline and glutamine rich intron retention, reduced expression and aggregate formation are pathological features of amyotrophic lateral sclerosis

Amy K. Cain, L. Henden, Zong-Hong Zhang, 2021, Neuropathology and applied neurobiology.

SFPQ intron retention, reduced expression and aggregate formation in central nervous system tissue are pathological features of amyotrophic lateral sclerosis

Amy K. Cain, L. Henden, Zong-Hong Zhang, 2020, bioRxiv.

Human and mouse ISLR (immunoglobulin superfamily containing leucine-rich repeat) genes: genomic structure and tissue expression.

N. Shimizu, S. Noda, J. Kudoh, 1999, Genomics.

A transient protein folding response targets aggregation in the early phase of TDP-43-mediated disease

M. Mirzaei, Adekunle T. Bademosi, D. Pascovici, 2023, bioRxiv.

Early activation of cellular stress and death pathways caused by cytoplasmic TDP-43 in the rNLS8 mouse model of ALS and FTD

F. Rigo, P. Jafar-Nejad, A. Wright, 2023, Molecular Psychiatry.

A transient protein folding response targets aggregation in the early phase of TDP-43-mediated neurodegeneration

Emma L. Scotter, Adekunle T. Bademosi, John D. Lee, 2024, Nature communications.