E. Onesto

发表

From transcriptomic to protein level changes in TDP-43 and FUS loss-of-function cell models.

E. Buratti, F. Baralle, A. Ratti, 2015, Biochimica et biophysica acta.

TDP-43 and FUS RNA-binding Proteins Bind Distinct Sets of Cytoplasmic Messenger RNAs and Differently Regulate Their Post-transcriptional Fate in Motoneuron-like Cells*

Vincenzo Silani, A. Pizzuti, E. Buratti, 2012, The Journal of Biological Chemistry.

The anabolic/androgenic steroid nandrolone exacerbates gene expression modifications induced by mutant SOD1 in muscles of mice models of amyotrophic lateral sclerosis

M. Bentivoglio, C. Bendotti, V. Crippa, 2012, Pharmacological research.

ALS-related misfolded protein management in motor neurons and muscle cells

E. Giorgetti, V. Crippa, A. Poletti, 2014, Neurochemistry International.

Neuritin (cpg15) enhances the differentiating effect of NGF on neuronal PC12 cells

C. Ronchi, A. Poletti, E. Onesto, 2007, Journal of neuroscience research.

The small heat shock protein B8 (HspB8) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis (ALS).

C. Bendotti, Marianna Marino, E. Fontana, 2010, Human molecular genetics.

The small heat shock protein B 8 ( HspB 8 ) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis ( ALS )

C. Bendotti, Marianna Marino, E. Fontana, 2010 .

Gene-specific mitochondria dysfunctions in human TARDBP and C9ORF72 fibroblasts

V. Tiranti, A. Ratti, V. Silani, 2016, Acta neuropathologica communications.

Inter-Species Differences in Regulation of the Progranulin–Sortilin Axis in TDP-43 Cell Models of Neurodegeneration

A. Ratti, V. Silani, V. Gumina, 2019, International journal of molecular sciences.

Gene-specific mitochondria dysfunctions in human TARDBP and C9ORF72 fibroblasts

V. Tiranti, A. Ratti, V. Silani, 2016, Acta neuropathologica communications.

RNA-binding proteins and RNA metabolism: a new scenario in the pathogenesis of Amyotrophic lateral sclerosis.

N. Ticozzi, C. Tiloca, A. Ratti, 2011, Archives italiennes de biologie.

Dysregulation of axonal transport and motorneuron diseases

A. Ratti, V. Crippa, A. Poletti, 2011, Biology of the cell.

Motoneuronal and muscle-selective removal of ALS-related misfolded proteins.

E. Giorgetti, V. Crippa, A. Poletti, 2013, Biochemical Society transactions.

Muscle cells and motoneurons differentially remove mutant SOD1 causing familial amyotrophic lateral sclerosis

V. Crippa, A. Poletti, N. Ferri, 2011, Journal of neurochemistry.

17-AAG increases autophagic removal of mutant androgen receptor in spinal and bulbar muscular atrophy

V. Crippa, A. Poletti, N. Ferri, 2011, Neurobiology of Disease.

The role of the polyglutamine tract in androgen receptor

A. Gliozzi, V. Crippa, A. Poletti, 2008, The Journal of Steroid Biochemistry and Molecular Biology.

Mutation of SOD1 in ALS: a gain of a loss of function.

P. Riso, M. Porrini, C. Bendotti, 2007, Human molecular genetics.

Proteasomal and autophagic degradative activities in spinal and bulbar muscular atrophy

V. Crippa, A. Poletti, M. Piccolella, 2010, Neurobiology of Disease.

Androgens inhibit androgen receptor promoter activation in motor neurons

L. Martini, A. Poletti, E. Onesto, 2009, Neurobiology of Disease.

Aggregation and proteasome: The case of elongated polyglutamine aggregation in spinal and bulbar muscular atrophy

L. Martini, V. Crippa, A. Poletti, 2007, Neurobiology of Aging.

ALS-related misfolded protein management in motor neurons and muscle cells

E. Giorgetti, V. Crippa, A. Poletti, 2014, Neurochemistry International.

The small heat shock protein B8 (HspB8) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis (ALS).

C. Bendotti, Marianna Marino, E. Fontana, 2010, Human molecular genetics.

Proteasomal and autophagic degradative activities in spinal and bulbar muscular atrophy

V. Crippa, A. Poletti, M. Piccolella, 2010, Neurobiology of Disease.