B. Omar

发表

Beta-Cell Function and Its Underlying Mechanism 2015

Li Chen, Yanbing Li, Chen Wang, 2015, Journal of diabetes research.

The phenotype of a knockout mouse identifies flavin-containing monooxygenase 5 (FMO5) as a regulator of metabolic ageing

J. Timms, I. Phillips, E. Shephard, 2015, Biochemical pharmacology.

Methods and Models for Metabolic Assessment in Mice

G. Pacini, B. Ahrén, B. Omar, 2013, Journal of diabetes research.

Conditional glucagon receptor overexpression has multi-faceted consequences for beta-cell function.

B. Ahrén, M. Sörhede-Winzell, B. Omar, 2014, Metabolism: clinical and experimental.

From PDE3B to the regulation of energy homeostasis.

E. Degerman, V. Manganiello, L. Stenson, 2011, Current opinion in pharmacology.

Link Between GIP and Osteopontin in Adipose Tissue and Insulin Resistance

L. Groop, M. Laakso, P. Poulsen, 2013, Diabetes.

Short running title: GIPR, adipose tissue and insulin resistance

L. Groop, M. Laakso, P. Poulsen, 2013 .

Incretin hormone receptors are required for normal beta cell development and function in female mice

Y. Seino, B. Ahrén, B. Omar, 2016, Peptides.

Regulation of the pro-inflammatory cytokine osteopontin by GIP in adipocytes--a role for the transcription factor NFAT and phosphodiesterase 3B.

L. Groop, V. Lyssenko, E. Degerman, 2012, Biochemical and biophysical research communications.

Dipeptidyl peptidase 4 (DPP-4) is expressed in mouse and human islets and its activity is decreased in human islets from individuals with type 2 diabetes

P. Marchetti, Y. Seino, B. Ahrén, 2014, Diabetologia.

Protein kinase B activity is required for the effects of insulin on lipid metabolism in adipocytes.

A. Gormand, E. Degerman, O. Göransson, 2009, American journal of physiology. Endocrinology and metabolism.

Fibroblast Growth Factor 21 (FGF21) and Glucagon-Like Peptide 1 Contribute to Diabetes Resistance in Glucagon Receptor–Deficient Mice

J. Hald, B. Ahrén, E. Nishimura, 2013, Diabetes.

Incretin and islet hormone responses to meals of increasing size in healthy subjects.

G. Pacini, A. Mari, R. Bizzotto, 2015, The Journal of clinical endocrinology and metabolism.

Pleiotropic Mechanisms for the Glucose-Lowering Action of DPP-4 Inhibitors

B. Ahrén, B. Omar, 2014, Diabetes.

Differential Development of Glucose Intolerance and Pancreatic Islet Adaptation in Multiple Diet Induced Obesity Models

G. Pacini, B. Ahrén, B. Omar, 2012, Nutrients.

Defective insulin secretion by chronic glucagon receptor activation in glucose intolerant mice.

K. Fosgerau, B. Ahrén, B. Omar, 2016, The Journal of endocrinology.

receptor activation in glucose intolerant mice

K. Fosgerau, B. Ahrén, B. Omar, 2015 .

Fibroblast Growth Factor 21 ( FGF 21 ) and Glucagon Like-Peptide 1 Contribute to Diabetes Resistance in Glucagon Receptor Deficient Mice

J. Hald, E. Nishimura, K. Raun, 2014 .

Mixed meal ingestion diminishes glucose excursion in comparison with glucose ingestion via several adaptive mechanisms in people with and without type 2 diabetes

A. Tura, G. Pacini, A. Mari, 2016, Diabetes, obesity & metabolism.

Evidence for neural contribution to islet effects of DPP-4 inhibition in mice.

G. Pacini, B. Ahrén, B. Omar, 2016, European journal of pharmacology.

Effect of Zn on the structural and electrical properties of high temperature HgBa2Ca2Cu3O8+δ superconductor

B. Omar, S. J. Fathi, K. A. Jassim, 2018 .

Alterations in cyclic nucleotide phosphodiesterase activities in omental and subcutaneous adipose tissues in human obesity

E. Degerman, E. Banke, B. Omar, 2011, Nutrition & Diabetes.

Fibroblast growth factor 21 prevents glycemic deterioration in insulin deficient mouse models of diabetes.

B. Ahrén, K. Raun, B. Omar, 2015, European journal of pharmacology.

Extrapancreatic contribution to glucose regulation by dipeptidyl peptidase 4 inhibition

B. Ahrén, B. Omar, 2016 .

Dipeptidyl peptidase-4 (DPP-4): Localization and activity in human and rodent islets.

P. Marchetti, B. Ahrén, B. Omar, 2014, Biochemical and biophysical research communications.

Regulation of AMP-activated protein kinase by cAMP in adipocytes: roles for phosphodiesterases, protein kinase B, protein kinase A, Epac and lipolysis.

E. Degerman, V. Manganiello, O. Göransson, 2009, Cellular signalling.

The phenotype of a flavin-containing monooyxgenase knockout mouse implicates the drug-metabolizing enzyme FMO1 as a novel regulator of energy balance.

I. Phillips, E. Shephard, B. Omar, 2014, Biochemical pharmacology.

Deletion of the mouse Fmo1 gene results in enhanced pharmacological behavioural responses to imipramine

I. Phillips, E. Shephard, D. Hernandez, 2009, Pharmacogenetics and genomics.

Enhanced beta cell function and anti-inflammatory effect after chronic treatment with the dipeptidyl peptidase-4 inhibitor vildagliptin in an advanced-aged diet-induced obesity mouse model

J. Foley, B. Ahrén, M. S. Winzell, 2013, Diabetologia.

DPP-4 is expressed in human pancreatic beta cells and its direct inhibition improves beta cell function and survival in type 2 diabetes

U. Boggi, M. Bugliani, P. Marchetti, 2018, Molecular and Cellular Endocrinology.

Enhanced insulin sensitivity mediated by adipose tissue browning perturbs islet morphology and hormone secretion in response to autonomic nervous activation in female mice.

S. Enerbäck, B. Ahrén, B. Omar, 2016, American journal of physiology. Endocrinology and metabolism.