T. Kakita

发表

Calcineurin Pathway Is Required for Endothelin-1—Mediated Protection Against Oxidant Stress—Induced Apoptosis in Cardiac Myocytes

S. Sasayama, S. Adachi, H. Wada, 2001, Circulation research.

p300 Protein as a Coactivator of GATA-5 in the Transcription of Cardiac-restricted Atrial Natriuretic Factor Gene*

S. Sasayama, H. Wada, K. Hasegawa, 1999, The Journal of Biological Chemistry.

GATA-5 Is Involved in Leukemia Inhibitory Factor-responsive Transcription of the β-Myosin Heavy Chain Gene in Cardiac Myocytes*

A. Matsumori, S. Sasayama, R. Kitsis, 1999, The Journal of Biological Chemistry.

Phosphorylation of GATA-4 Is Involved in α1-Adrenergic Agonist-responsive Transcription of the Endothelin-1 Gene in Cardiac Myocytes*

S. Sasayama, H. Wada, K. Hasegawa, 2000, The Journal of Biological Chemistry.

α- and β-Adrenergic Pathways Differentially Regulate Cell Type–Specific Apoptosis in Rat Cardiac Myocytes

S. Sasayama, K. Hasegawa, E. Iwai-Kanai, 1999 .

Calcineurin-GATA-6 pathway is involved in smooth muscle–specific transcription

S. Sasayama, M. Abe, H. Wada, 2002, The Journal of cell biology.

A p300 Protein as a Coactivator of GATA-6 in the Transcription of the Smooth Muscle-Myosin Heavy Chain Gene*

S. Sasayama, H. Wada, K. Hasegawa, 2000, The Journal of Biological Chemistry.

a-and b-Adrenergic Pathways Differentially Regulate Cell Type – Specific Apoptosis in Rat Cardiac Myocytes

S. Sasayama, K. Hasegawa, E. Iwai-Kanai, 1999 .

Endothelin-1 as a protective factor against beta-adrenergic agonist-induced apoptosis in cardiac myocytes.

M. Fujita, S. Sasayama, T. Sawamura, 2000, Journal of the American College of Cardiology.

alpha- and beta-adrenergic pathways differentially regulate cell type-specific apoptosis in rat cardiac myocytes.

S. Sasayama, K. Hasegawa, E. Iwai-Kanai, 1999, Circulation.

Calcineurin-GATA4 Pathway Is Involved in β-Adrenergic Agonist-responsive Endothelin-1 Transcription in Cardiac Myocytes*

S. Sasayama, H. Wada, K. Hasegawa, 2001, The Journal of Biological Chemistry.