G. Nowak

发表

Recovery of cellular functions following oxidant injury.

M. Aleo, R. Schnellmann, G. Nowak, 1998, American journal of physiology. Renal physiology.

Dependence of cisplatin-induced cell death in vitro and in vivo on cyclin-dependent kinase 2.

J. Megyesi, R. Safirstein, P. Price, 2006, Journal of the American Society of Nephrology : JASN.

Protein kinase B/Akt modulates nephrotoxicant-induced necrosis in renal cells.

G. Nowak, Zabeena P. Shaik, E. K. Fifer, 2007, American journal of physiology. Renal physiology.

Protein Kinase C- (cid:1) and ERK1/2 Mediate Mitochondrial Dysfunction, Decreases in Active Na (cid:2) Transport, and Cisplatin-induced Apoptosis in Renal Cells*

G. Nowak, 2022 .

Lack of a functional p21WAF1/CIP1 gene accelerates caspase-independent apoptosis induced by cisplatin in renal cells.

P. Price, R. Schnellmann, G. Nowak, 2003, American journal of physiology. Renal physiology.

Microenvironment‐induced PIM kinases promote CXCR4‐triggered mTOR pathway required for chronic lymphocytic leukaemia cell migration

M. Szydłowski, E. Jabłońska, P. Juszczyński, 2018, Journal of cellular and molecular medicine.

Differential effects of EGF on repair of cellular functions after dichlorovinyl-l-cysteine-induced injury.

R. Schnellmann, G. Nowak, K. B. Keasler, 1999, American journal of physiology. Renal physiology.

Akt activation improves oxidative phosphorylation in renal proximal tubular cells following nephrotoxicant injury.

G. Nowak, Zabeena P. Shaik, E. K. Fifer, 2008, American journal of physiology. Renal physiology.

γ-Tocotrienol Protects against Mitochondrial Dysfunction, Energy Deficits, Morphological Damage, and Decreases in Renal Functions after Renal Ischemia

J. Megyesi, G. Nowak, 2021, International journal of molecular sciences.

γ-Tocotrienol Protects against Mitochondrial Dysfunction and Renal Cell Death

M. Hauer-Jensen, C. Compadre, G. Nowak, 2012, Journal of Pharmacology and Experimental Therapeutics.

Protein kinase Cε targets respiratory chain and mitochondrial membrane potential but not F0F1‐ATPase in renal cells injured by oxidant

G. Nowak, Diana Bakajsova-Takacsova, 2018, Journal of cellular biochemistry.

Linoleic acid epoxide promotes the maintenance of mitochondrial function and active Na+ transport following hypoxia.

D. Grant, G. Nowak, J. Moran, 2004, Toxicology letters.

Protein Kinase C Mediates Repair of Mitochondrial and Transport Functions after Toxicant-Induced Injury in Renal Cells

G. Nowak, 2003, Journal of Pharmacology and Experimental Therapeutics.

Ascorbic acid promotes recovery of cellular functions following toxicant-induced injury.

R. Schnellmann, C. A. Carter, G. Nowak, 2000, Toxicology and applied pharmacology.

Differential effects of EGF on repair of cellular functions after dichlorovinyl-L-cysteine-induced injury

R. Schnellmann, G. Nowak, K. B. Keasler, 1999 .

Increased expression of E3 ubiquitin ligases targeting p53 in CLL patients with wild-type TP53 exhibits associations with clinical features of the disease

P. Juszczyński, P. Gorniak, K. Warzocha, 2016, Leukemia & lymphoma.

Integrative effects of EGF on metabolism and proliferation in renal proximal tubular cells.

R. Schnellmann, G. Nowak, 1995, The American journal of physiology.

Recovery of cellular functions following oxidant injury

M. Aleo, R. Schnellmann, G. Nowak, 1998 .

Deletion of VDAC1 Hinders Recovery of Mitochondrial and Renal Functions After Acute Kidney Injury

W. Craigen, J. Megyesi, G. Nowak, 2020, Biomolecules.

Autocrine production and TGF-beta 1-mediated effects on metabolism and viability in renal cells.

R. Schnellmann, G. Nowak, 1996, The American journal of physiology.

Succinate Ameliorates Energy Deficits and Prevents Dysfunction of Complex I in Injured Renal Proximal Tubular Cells

G. Nowak, Diana Bakajsova, G. L. Clifton, 2008, Journal of Pharmacology and Experimental Therapeutics.

Hypoxia and proliferation are primarily responsible for induction of lactate dehydrogenase activity in cultured cells.

R. Schnellmann, G. Nowak, J. Griffin, 1996, Journal of toxicology and environmental health.

Improved culture conditions stimulate gluconeogenesis in primary cultures of renal proximal tubule cells.

R. Schnellmann, G. Nowak, 1995, The American journal of physiology.

Frequency of BCR-ABL gene mutations in Polish patients with chronic myeloid leukemia treated with imatinib: a final report of the MAPTEST study.

M. Majewski, A. Skotnicki, K. Warzocha, 2009, Polskie Archiwum Medycyny Wewnetrznej.

Deletion of protein kinase C-ε attenuates mitochondrial dysfunction and ameliorates ischemic renal injury.

J. Megyesi, G. Nowak, Diana Takacsova-Bakajsova, 2017, American journal of physiology. Renal physiology.

Protein Kinase C-ε Activation Induces Mitochondrial Dysfunction and Fragmentation in Renal Proximal Tubules

A. Samarel, G. Nowak, Diana Bakajsova, 2022 .

Protein Kinases and Regulation of Mitochondrial Function in Ischemia/Reperfusion Injury Protein Kinases and Regulation of Mitochondrial Function in Ischemia/Reperfusion Injury

G. Nowak, 2018 .

Serum Tumor Necrosis Factor-α and Interleukin-10 Levels as Markers to Predict Outcome of Patients with Chronic Lymphocytic Leukemia in Different Risk Groups Defined by the IGHV Mutation Status

M. Borowiec, O. Grzybowska-Izydorczyk, W. Młynarski, 2012, Archivum Immunologiae et Therapiae Experimentalis.

L-ascorbic acid regulates growth and metabolism of renal cells: improvements in cell culture.

R. Schnellmann, G. Nowak, 1996, The American journal of physiology.

Nephrotoxicant inhibition of renal proximal tubule cell regeneration.

R. Schnellmann, R. Wyatt, G. Nowak, 1995, The American journal of physiology.

Protein kinase C-alpha inhibits the repair of oxidative phosphorylation after S-(1,2-dichlorovinyl)-L-cysteine injury in renal cells.

G. Nowak, Xiuli Liu, Malinda L Godwin, 2004, American journal of physiology. Renal physiology.

Collagen IV promotes repair of renal cell physiological functions after toxicant injury.

R. Schnellmann, P. Nony, G. Nowak, 2001, American journal of physiology. Renal physiology.

Protein kinase C-epsilon activation induces mitochondrial dysfunction and fragmentation in renal proximal tubules.

A. Samarel, G. Nowak, Diana Bakajsova, 2011, American journal of physiology. Renal physiology.

Direct Interaction of All-trans-retinoic Acid with Protein Kinase C (PKC)

C. A. Carter, G. Nowak, P. Czernik, 2000, The Journal of Biological Chemistry.

High incidence of ancestral HLA haplotype 8.1 and monoclonal incomplete DH–JH immunoglobulin heavy chain gene rearrangement in persistent polyclonal B-cell lymphocytosis

M. Majewski, K. Kuliczkowski, J. Nowak, 2008, Annals of Hematology.

Protein kinase C-α interaction with iHSP70 in mitochondria promotes recovery of mitochondrial function after injury in renal proximal tubular cells.

R. Mestril, G. Nowak, S. Soundararajan, 2013, American journal of physiology. Renal physiology.

Protein kinase C-interaction with iHSP 70 in mitochondria promotes recovery of mitochondrial function after injury in renal proximal tubular cells

R. Mestril, G. Nowak, S. Soundararajan, 2013 .

Activation of ERK1/2 pathway mediates oxidant-induced decreases in mitochondrial function in renal cells.

G. Nowak, Diana Bakajsova, G. L. Clifton, 2006, American journal of physiology. Renal physiology.

Comparison of high‐resolution melting analysis with direct sequencing for the detection of recurrent mutations in DNA methyltransferase 3A and isocitrate dehydrogenase 1 and 2 genes in acute myeloid leukemia patients

P. Juszczyński, P. Gorniak, K. Warzocha, 2016, European journal of haematology.

Predictive significance of selected gene mutations in relapsed and refractory chronic lymphocytic leukemia patients treated with ibrutinib

R. Płoski, M. Rydzanicz, P. Juszczyński, 2020, European journal of haematology.

Ochratoxin A secretion in primary cultures of rabbit renal proximal tubule cells.

M. Morales, G. Nowak, C. E. Groves, 1999, Journal of the American Society of Nephrology : JASN.

Radiation-Induced Alterations in Mitochondria of the Rat Heart

E. Moros, M. Boerma, V. Sridharan, 2014, Radiation research.

Protein Kinases and Regulation of Mitochondrial Function in Ischemia/Reperfusion Injury

G. Nowak, 2017, Mitochondrial Diseases.

Protein kinase Cα mediates recovery of renal and mitochondrial functions following acute injury

J. Megyesi, G. Nowak, 2020, The FEBS journal.

Protein kinase C-ϵ modulates mitochondrial function and active Na+ transport after oxidant injury in renal cells

G. Nowak, Diana Bakajsova, G. L. Clifton, 2004 .

Protein Kinase C-α Interaction with F0F1-ATPase Promotes F0F1-ATPase Activity and Reduces Energy Deficits in Injured Renal Cells*

G. Nowak, Diana Bakajsova, 2015, The Journal of Biological Chemistry.

Protein kinase C-α activation promotes recovery of mitochondrial function and cell survival following oxidant injury in renal cells.

G. Nowak, Diana Bakajsova, 2012, American journal of physiology. Renal physiology.

Renal cell regeneration following oxidant exposure: inhibition by TGF-beta1 and stimulation by ascorbic acid.

R. Schnellmann, G. Nowak, Rick G. Schnellmann, 1997, Toxicology and applied pharmacology.

Stress Response in a Leporine Renal Cell Model

R. Schnellmann, K. Henle, G. Nolen, 1997, Nephron.

Assessment of mitochondrial functions and cell viability in renal cells overexpressing protein kinase C isozymes.

G. Nowak, Diana Bakajsova, 2013, Journal of visualized experiments : JoVE.

Protein kinase C-epsilon modulates mitochondrial function and active Na+ transport after oxidant injury in renal cells.

G. Nowak, Diana Bakajsova, G. L. Clifton, 2004, AJP - Renal Physiology.

P. Juszczyński, P. Gorniak, K. Warzocha, 2016, European journal of haematology.

γ-Tocotrienol Protects against Mitochondrial Dysfunction and Renal Cell Death

M. Hauer-Jensen, C. Compadre, G. Nowak, 2012, Journal of Pharmacology and Experimental Therapeutics.

PROTEIN KINASE C MEDIATES REPAIR OF MITOCHONDRIAL AND TRANSPORT FUNCTIONS FOLLOWING TOXICANT-INDUCED INJURY IN RENAL CELLS

G. Nowak, 2003 .