J. Paterson

发表

Metabolic syndrome without obesity: Hepatic overexpression of 11beta-hydroxysteroid dehydrogenase type 1 in transgenic mice.

J. Seckl, J. Mullins, B. Staels, 2004, Proceedings of the National Academy of Sciences of the United States of America.

Short-term plasticity of cyclic adenosine 3',5'-monophosphate signaling in anterior pituitary corticotrope cells: the role of adenylyl cyclase isotypes.

A. Sosunov, A. Haunsø, J. Paterson, 2003, Molecular endocrinology.

11β-Hydroxysteroid Dehydrogenase Type 1 Expression Is Increased in the Aged Mouse Hippocampus and Parietal Cortex and Causes Memory Impairments

J. Seckl, J. Mullins, M. Holmes, 2010, The Journal of Neuroscience.

Liver-selective transgene rescue of hypothalamic-pituitary-adrenal axis dysfunction in 11beta-hydroxysteroid dehydrogenase type 1-deficient mice.

J. Seckl, J. Mullins, M. Holmes, 2007, Endocrinology.

Edinburgh Research Explorer The mother or the fetus? 11 beta-hydroxysteroid dehydrogenase type 2 null mice provide evidence for direct fetal programming of behavior by endogenous glucocorticoids

J. Seckl, J. Mullins, Christian T. Abrahamsen, 2018 .

Hypothalamic-Pituitary-Adrenal Axis Abnormalities in Response to Deletion of 11β-HSD1 is Strain-Dependent

J. Seckl, J. Mullins, M. Holmes, 2009, Journal of neuroendocrinology.

Genetic manipulation of 11beta-hydroxysteroid dehydrogenases in mice.

J. Seckl, J. Mullins, J. Paterson, 2005, American journal of physiology. Regulatory, integrative and comparative physiology.

A Transgenic Model of Visceral Obesity and the Metabolic Syndrome

J. Flier, J. Seckl, J. Mullins, 2001, Science.

Calcineurin Feedback Inhibition of Agonist-evoked cAMP Formation (*)

R. J. Barnard, M. Shipston, J. Paterson, 1995, The Journal of Biological Chemistry.

11β-Hydroxysteroid dehydrogenase type 2 protects the neonatal cerebellum from deleterious effects of glucocorticoids

J. Seckl, I. Whittle, J. Mullins, 2006, Neuroscience.

The Mother or the Fetus? 11β-Hydroxysteroid Dehydrogenase Type 2 Null Mice Provide Evidence for Direct Fetal Programming of Behavior by Endogenous Glucocorticoids

J. Seckl, J. Mullins, M. Holmes, 2006, The Journal of Neuroscience.

Repression of preprotachykinin-A promoter activity is mediated by a proximal promoter element

J. Quinn, S. Dobson, C. Morrison, 1995, Neuroscience.

Novel adipose tissue-mediated resistance to diet-induced visceral obesity in 11 beta-hydroxysteroid dehydrogenase type 1-deficient mice.

J. Flier, B. Walker, J. Seckl, 2004, Diabetes.

11&bgr;-Hydroxysteroid Dehydrogenase Type 2 in Mouse Aorta: Localization and Influence on Response to Glucocorticoids

B. Walker, J. Seckl, J. Mullins, 2003, Hypertension.

An upstream stimulatory factor (USF) binding motif is critical for rat preprotachykinin-A promoter activity in PC12 cells.

J. Quinn, C. Morrison, J. Paterson, 1995, The Biochemical journal.

Characterisation of a functional E box motif in the proximal rat preprotachykinin-A promoter

J. Quinn, S. Dobson, C. Morrison, 1995, Neuroscience Letters.

Ca2+/Calcineurin-Inhibited Adenylyl Cyclase, Highly Abundant in Forebrain Regions, Is Important for Learning and Memory

M. Palkovits, G. Fink, R. Rosie, 1998, The Journal of Neuroscience.

Ca 2 1 / Calcineurin-Inhibited Adenylyl Cyclase , Highly Abundant in Forebrain Regions , Is Important for Learning and Memory

M. Palkovits, G. Fink, R. Rosie, 1998 .

Characterisation of Human Adenylyl Cyclase IX Reveals Inhibition by Ca2+/Calcineurin and Differential mRNA Plyadenylation

O. Grace, A. Sosunov, J. Paterson, 2000, Journal of neurochemistry.

Three immediate early gene response elements in the proximal preprotachykinin-A promoter in two functionally distinct domains

J. Quinn, S. Dobson, C. Morrison, 1995, Neuroscience.

Transgenic amplification of glucocorticoid action in adipose tissue causes high blood pressure in mice.

J. Flier, J. Elmquist, J. Seckl, 2003, The Journal of clinical investigation.

Calcium control of adenylyl cyclase: the calcineurin connection.

G. Fink, R. Rosie, S. Smith, 1998, Advances in second messenger and phosphoprotein research.

A switch in the mechanism of hypertension in the syndrome of apparent mineralocorticoid excess.

N. Wrobel, J. Seckl, J. Mullins, 2008, Journal of the American Society of Nephrology : JASN.

Hypothalamic-Pituitary-Adrenal Axis Abnormalities in Response to Deletion of 11β-HSD1 is Strain-Dependent

J. Seckl, J. Mullins, M. Holmes, 2009, Journal of neuroendocrinology.

Transcriptional control of neuropeptide gene expression in sensory neurons, using the preprotachykinin-A gene as a model.

J. Quinn, C. Morrison, J. Paterson, 1995, Canadian journal of physiology and pharmacology.

11&bgr;-Hydroxysteroid Dehydrogenase Type 2 in Mouse Aorta: Localization and Influence on Response to Glucocorticoids

B. Walker, J. Seckl, J. Mullins, 2003, Hypertension.

11beta-Hydroxysteroid dehydrogenase type 2 protects the neonatal cerebellum from deleterious effects of glucocorticoids.

J. Seckl, I. Whittle, J. Mullins, 2006, Neuroscience.