T. Karonitsch

发表

The RNA‐binding protein HuR/ELAVL1 regulates IFN‐β mRNA abundance and the type I IFN response

G. Superti-Furga, K. Bennett, A. Stukalov, 2015, European journal of immunology.

Analysis of gene expression in rheumatoid arthritis and related conditions offers insights into sex-bias, gene biotypes and co-expression patterns

T. Nussbaumer, J. Smolen, A. Platzer, 2019, PloS one.

Reporting disease activity in clinical trials of patients with rheumatoid arthritis: EULAR/ACR collaborative recommendations

G. Koch, P. Tugwell, C. Bombardier, 2008, Annals of the rheumatic diseases.

Systemic lupus erythematosus patients exhibit functional deficiencies of endothelial progenitor cells.

J. Smolen, G. Weigel, I. Schwarzinger, 2008, Rheumatology.

Engulfed cell remnants, and not cells undergoing apoptosis, constitute the LE-cell phenomenon

M. Aringer, E. Feierl, J. Smolen, 2007, Autoimmunity.

Musculoskeletal disease burden of hereditary hemochromatosis.

C. Datz, J. Zwerina, R. Axmann, 2010, Arthritis and rheumatism.

CCL17/thymus and activation-related chemokine in Churg-Strauss syndrome.

F. Moosig, J. Zwerina, G. Schett, 2010, Arthritis and rheumatism.

Histone deacetylase 1 (HDAC1): A key player of T cell-mediated arthritis.

R. Holmdahl, L. Müller, W. Ellmeier, 2019, Journal of autoimmunity.

The immunobiology of mTOR in autoimmunity.

T. Karonitsch, T. Suto, 2019, Journal of autoimmunity.

CCL 17 / Thymus and Activation – Related Chemokine in Churg-Strauss Syndrome

F. Moosig, J. Zwerina, G. Schett, 2010 .

Eotaxin-3 is involved in Churg-Strauss syndrome--a serum marker closely correlating with disease activity.

J. Zwerina, G. Schett, K. Redlich, 2008, Rheumatology.

mTOR Senses Environmental Cues to Shape the Fibroblast-like Synoviocyte Response to Inflammation

G. Superti-Furga, T. Pap, T. Weichhart, 2018, Cell reports.

Abatacept (CTLA-4Ig) treatment reduces T cell apoptosis and regulatory T cell suppression in patients with rheumatoid arthritis.

J. Smolen, C. Scheinecker, S. Blüml, 2016, Rheumatology.

Hereditary hemochromatosis as a risk factor for joint replacement surgery.

C. Datz, S. Kiechl, J. Willeit, 2010, The American journal of medicine.

Targeted inhibition of Janus kinases abates interfon gamma-induced invasive behaviour of fibroblast-like synoviocytes

R. Byrne, T. Pap, J. Smolen, 2018, Rheumatology.

Response to SARS-CoV-2 vaccination in systemic autoimmune rheumatic disease depends on immunosuppressive regimen: a matched, prospective cohort study

P. Mandl, H. Haslacher, D. Aletaha, 2022, Annals of the Rheumatic Diseases.

Validation of a radiographic scoring system for haemochromatosis arthropathy

C. Datz, A. Cavallaro, J. Zwerina, 2010, Annals of the rheumatic diseases.

Interferon signals and monocytic sensitization of the interferon-γ signaling pathway in the peripheral blood of patients with rheumatoid arthritis.

M. Aringer, J. Smolen, S. Blüml, 2012, Arthritis and rheumatism.

The Lipid-Modifying Enzyme SMPDL3B Negatively Regulates Innate Immunity

G. Superti-Furga, K. Bennett, B. Snijder, 2015, Cell reports.

The synovium as a privileged site in rheumatoid arthritis: cadherin-11 as a dominant player in synovial pathology.

H. Kiener, T. Karonitsch, 2011, Best practice & research. Clinical rheumatology.

FOXO3 is involved in the tumor necrosis factor-driven inflammatory response in fibroblast-like synoviocytes

G. Superti-Furga, R. Byrne, T. Pap, 2019, Laboratory Investigation.

IRF1 is critical for the TNF-driven interferon response in rheumatoid fibroblast-like synoviocytes

G. Superti-Furga, T. Pap, J. Smolen, 2019, Experimental & Molecular Medicine.

IRF1 is critical for the TNF-driven interferon response in rheumatoid fibroblast-like synoviocytes

G. Superti-Furga, T. Pap, J. Smolen, 2019, Experimental & Molecular Medicine.

Nicotinic acetylcholine receptors modulate osteoclastogenesis

A. Mócsai, P. Mandl, J. Smolen, 2016, Arthritis Research & Therapy.

Activation of the interferon-gamma signaling pathway in systemic lupus erythematosus peripheral blood mononuclear cells.

M. Aringer, E. Feierl, J. Smolen, 2009, Arthritis and rheumatism.

Reporting disease activity in clinical trials of patients with rheumatoid arthritis: EULAR/ACR collaborative recommendations.

P Tugwell, B Combe, G. Koch, 2008, Arthritis and rheumatism.

TNFR2 is critical for TNF-induced rheumatoid arthritis fibroblast-like synoviocyte inflammation.

T. Pap, A. Platzer, D. Aletaha, 2022, Rheumatology.

Methods of deriving EULAR/ACR recommendations on reporting disease activity in clinical trials of patients with rheumatoid arthritis

M. Dougados, G. Wells, D. Felson, 2008, Annals of the rheumatic diseases.

Non-classical monocytes as mediators of tissue destruction in arthritis

Jörg Menche, J. O’Shea, Michael Caldera, 2018, Annals of the rheumatic diseases.

Cytokine-directed cellular cross-talk imprints synovial pathotypes in rheumatoid arthritis

G. Superti-Furga, G. Steiner, L. Müller, 2023, Annals of the Rheumatic Diseases.

CD4+CD25-Foxp3+ T cells: a marker for lupus nephritis?

J. Smolen, C. Scheinecker, S. Blüml, 2014, Arthritis Research & Therapy.

A4.6 Nicotinic acetylcholine receptor ligands inhibit osteoclastogenesis by blocking RANKL-induced calcium-oscillation and induction of NFATC1 and CFOS

A. Mócsai, J. Smolen, P. Mandl, 2014 .

RESEARCH ARTICLE Open Access CD4 + CD25 Foxp3 + T cells: a marker for lupus

J. Smolen, C. Scheinecker, S. Blüml, 2014 .

Abatacept (CTLA-4IG) treatment reduces the migratory capacity of monocytes in patients with rheumatoid arthritis.

R. Byrne, J. Smolen, M. Bergmann, 2013, Arthritis and rheumatism.

Focal adhesion protein Lasp1 links the Arp2/3 complex to adherens junctions and promotes motility of arthritic fibroblast-like synoviocytes.

S. Blüml, T. Karonitsch, A. Korb-Pap, 2024, Annals of the rheumatic diseases.

Amino acids fuel fibroblast-like synoviocyte activation and arthritis by regulating chemokine expression and leukocyte migration.

D. Aletaha, S. Blüml, F. Kartnig, 2023, Arthritis & rheumatology.