M. Mihalyo

发表

Effector CD4 cell tolerization is mediated through functional inactivation and involves preferential impairment of TNF-α and IFN-γ expression potentials

A. Adler, M. Long, A. Higgins, 2003 .

Self-Antigen Prevents CD8 T Cell Effector Differentiation by CD134 and CD137 Dual Costimulation1

A. Adler, Suman Bandyopadhyay, H. Aguila, 2008, The Journal of Immunology.

In Vivo Cyclophosphamide and IL-2 Treatment Impedes Self-Antigen-Induced Effector CD4 Cell Tolerization: Implications for Adoptive Immunotherapy1

A. Adler, Yi Yang, M. Long, 2004, The Journal of Immunology.

for Adoptive Immunotherapy Effector CD4 Cell Tolerization: Implications Treatment Impedes Self-Antigen-Induced In Vivo Cyclophosphamide and IL-2

A. Adler, M. Long, M. Mihalyo, 2004 .

Antigen Subsequent Immunization to the Same Expression during γ Increased IFN-Tolerized CD 4 Cells Is Associated with Promoter in Ifng Histone Acetylation at the

A. Adler, Suman Bandyopadhyay, A. Vella, 2007 .

Initial Engagement Differentially Programmed by APCs upon CD 4 Cell Priming and Tolerization Are and

A. Adler, A. Higgins, M. Mihalyo, 2002 .

Exposure to Parenchymal Self-Antigen Effector CD 4 Cells Are Tolerized Upon

A. Adler, A. Higgins, M. Mihalyo, 2002 .

CD4 Cell Priming and Tolerization Are Differentially Programmed by APCs upon Initial Engagement1

A. Adler, A. Higgins, M. Mihalyo, 2002, The Journal of Immunology.

Effector CD4 Cells Are Tolerized Upon Exposure to Parenchymal Self-Antigen1

A. Adler, A. Higgins, M. Mihalyo, 2002, The Journal of Immunology.

T-bet Down-Modulation in Tolerized Th1 Effector CD4 Cells Confers a TCR-Distal Signaling Defect That Selectively Impairs IFN- γ Expression.

A. Adler, S. Reiner, M. Long, 2005 .

Steady State Dendritic Cells Present Parenchymal Self-Antigen and Contribute to, but Are Not Essential for, Tolerization of Naive and Th1 Effector CD4 Cells1

A. Adler, L. Lefrançois, Harry Z. Qui, 2007, The Journal of Immunology.

Naive and Th 1 Effector CD 4 Cells but Are Not Essential for , Tolerization of Parenchymal Self-Antigen and Contribute to , Steady State Dendritic Cells Present

A. Adler, L. Lefrançois, Harry Z. Qui, 2007 .

Glycoprotein 96 Can Chaperone Both MHC Class I- and Class II-Restricted Epitopes for In Vivo Presentation, but Selectively Primes CD8+ T Cell Effector Function 1

C. Drake, A. Adler, M. Mihalyo, 2004, The Journal of Immunology.

Androgen ablation mitigates tolerance to a prostate/prostate cancer-restricted antigen.

C. Drake, D. Pardoll, W. Nelson, 2005, Cancer cell.

The E3 Ubiquitin Ligase Cbl-b Regulates Expansion but Not Functional Activity of Self-Reactive CD4 T Cells1

A. Adler, Suman Bandyopadhyay, Harry Z. Qui, 2009, The Journal of Immunology.

Self-Reactive CD 4 T Cells Expansion but Not Functional Activity of The E 3 Ubiquitin Ligase Cbl-b Regulates

A. Adler, Suman Bandyopadhyay, Harry Z. Qui, 2009 .

Histone Acetylation at the Ifng Promoter in Tolerized CD4 Cells Is Associated with Increased IFN-γ Expression during Subsequent Immunization to the Same Antigen1

A. Adler, Suman Bandyopadhyay, A. Vella, 2007, The Journal of Immunology.

T-bet Down-Modulation in Tolerized Th1 Effector CD4 Cells Confers a TCR-Distal Signaling Defect That Selectively Impairs IFN-γ Expression1

A. Adler, S. Reiner, M. Long, 2005, The Journal of Immunology.

IFN-Signaling Defect That Selectively Impairs Effector CD 4 Cells Confers a TCR-Distal T-bet Down-Modulation in Tolerized Th 1

A. Adler, M. Long, M. Mihalyo, 2005 .

Dendritic cells program non‐immunogenic prostate‐specific T cell responses beginning at early stages of prostate tumorigenesis

A. Adler, M. Mihalyo, A. Hagymasi, 2007, The Prostate.

Feeding by the tick, Ixodes scapularis, causes CD4+ T cells responding to cognate antigen to develop the capacity to express IL‐4

A. Adler, S. Wikel, A. Vella, 2007, Parasite immunology.

Effector CD4 cell tolerization is mediated through functional inactivation and involves preferential impairment of TNF-alpha and IFN-gamma expression potentials.

A. Adler, M. Long, A. Higgins, 2003, Cellular immunology.