A. Nair

Cyclosporin A inhibits growth of autocrine tumour cell lines by destabilizing interleukin-3 mRNA

H. Hirsch, C. Moroni, S. Hahn, 1994, Nature.

Tumor suppression involves down-regulation of interleukin 3 expression in hybrids between autocrine mastocytoma and interleukin 3-dependent parental mast cells.

H. Hirsch, C. Moroni, I. Diamantis, 1989, Proceedings of the National Academy of Sciences of the United States of America.

A v-H-ras-dependent hemopoietic tumor model involving progression from a clonal stage of transformation competence to autocrine interleukin 3 production

V. Kindler, C. Moroni, I. Diamantis, 1989, Molecular and cellular biology.

Mast cells sensitive to v-H-ras transformation are hyperinducible for interleukin 3 expression and have lost tumor-suppressor activity.

H. Hirsch, C. Moroni, A. Nair, 1992, Oncogene.

Suppressible and nonsuppressible autocrine mast cell tumors are distinguished by insertion of an endogenous retroviral element (IAP) into the interleukin 3 gene

H. Hirsch, C. Moroni, A. Nair, 1993, The Journal of experimental medicine.

Interleukin-3 mRNA Stabilization by a trans-Acting Mechanism in Autocrine Tumors Lacking Interleukin-3 Gene Rearrangements (*)

H. Hirsch, C. Moroni, A. Nair, 1995, The Journal of Biological Chemistry.

Rapamycin destabilizes interleukin-3 mRNA in autocrine tumor cells by a mechanism requiring an intact 3' untranslated region

H. Hirsch, C. Moroni, X. Ming, 1997, Molecular and cellular biology.