J. Quoyer

Common Structural Requirements for Heptahelical Domain Function in Class A and Class C G Protein-coupled Receptors*

Gilles Labesse, G. Labesse, L. Prézeau, 2007, Journal of Biological Chemistry.

β-Arrestin 1 Is Required for PAC1 Receptor-mediated Potentiation of Long-lasting ERK1/2 Activation by Glucose in Pancreatic β-Cells*

J. Bockaert, S. Dalle, G. Bertrand, 2009, Journal of Biological Chemistry.

The chemokine CXC4 and CC2 receptors form homo‐ and heterooligomers that can engage their signaling G‐protein effectors and βarrestin

L. Prézeau, J. Pin, N. Heveker, 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Roles and regulation of the transcription factor CREB in pancreatic β -cells.

S. Dalle, S. Costes, J. Quoyer, 2011, Current molecular pharmacology.

GLP-1 Mediates Antiapoptotic Effect by Phosphorylating Bad through a β-Arrestin 1-mediated ERK1/2 Activation in Pancreatic β-Cells

J. Bockaert, S. Dalle, G. Bertrand, 2009, The Journal of Biological Chemistry.

1 GLP-1 MEDIATES ANTI-APOPTOTIC EFFECT BY PHOSPHORYLATING BAD THROUGH A BETA-ARRESTIN 1-MEDIATED ERK 1 / 2 ACTIVATION IN PANCREATIC β-CELLS

J. Bockaert, S. Dalle, G. Bertrand, 2009 .

Roles and regulation of the transcription factor CREB in pancreatic β -cells.

S. Dalle, S. Costes, J. Quoyer, 2011, Current molecular pharmacology.

Development and Characterization of Pepducins as Gs-biased Allosteric Agonists*♦

Michel Bouvier, Reynold A Panettieri, B. Kobilka, 2014, The Journal of Biological Chemistry.

Pepducin targeting the C-X-C chemokine receptor type 4 acts as a biased agonist favoring activation of the inhibitory G protein

J. Benovic, M. Bouvier, V. Lukashova, 2013, Proceedings of the National Academy of Sciences.