W. Bao

Selective mGluR5 receptor antagonist or agonist provides neuroprotection in a rat model of focal cerebral ischemia

F. Tortella, A. Faden, A. Williams, 2001, Brain Research.

Small shifts in craniotomy position in the lateral fluid percussion injury model are associated with differential lesion development.

R. Vink, A. Faden, P. Mullins, 2001, Journal of neurotrauma.

Role of vascular endothelial growth factor in neuronal DNA damage and repair in rat brain following a transient cerebral ischemia

M. Qiu, F. Sun, Ling-Mei Zhang, 2002, Journal of neuroscience research.

Differential Expression of Apoptotic Protease-Activating Factor-1 and Caspase-3 Genes and Susceptibility to Apoptosis during Brain Development and after Traumatic Brain Injury

A. Yakovlev, Geping Wang, A. Faden, 2001, The Journal of Neuroscience.

Selective Blockade of the mGluR1 Receptor Reduces Traumatic Neuronal Injury in Vitro and Improves Outcome after Brain Trauma

A. Faden, P. Mullins, W. Bao, 2001, Experimental Neurology.

mGluR5 antagonists 2-methyl-6-(phenylethynyl)-pyridine and (E)-2-methyl-6-(2-phenylethenyl)-pyridine reduce traumatic neuronal injury in vitro and in vivo by antagonizing N-methyl-D-aspartate receptors.

A. Faden, P. Mullins, S. Knoblach, 2001, The Journal of pharmacology and experimental therapeutics.

Intraventricular vascular endothelial growth factor antibody increases infarct volume following transient cerebral ischemia.

F. Sun, W. Bao, Huan Wang, 1999, Zhongguo yao li xue bao = Acta pharmacologica Sinica.