B. Harris

发表

Acamprosate inhibits the binding and neurotoxic effects of trans-ACPD, suggesting a novel site of action at metabotropic glutamate receptors.

N. W. Pedigo, M. Prendergast, B. Harris, 2002, Alcoholism, clinical and experimental research.

Acamprosate, MK-801, and ifenprodil inhibit neurotoxicity and calcium entry induced by ethanol withdrawal in organotypic slice cultures from neonatal rat hippocampus.

M. Prendergast, B. Harris, D. Gibson, 2002 .

Acamprosate, MK-801, and ifenprodil inhibit neurotoxicity and calcium entry induced by ethanol withdrawal in organotypic slice cultures from neonatal rat hippocampus.

M. Prendergast, B. Harris, D. Gibson, 2002, Alcoholism, clinical and experimental research.

Corticosterone and dexamethasone potentiate cytotoxicity associated with oxygen-glucose deprivation in organotypic cerebellar slice cultures

P. Mulholland, M. Prendergast, B. Harris, 2005, Neuroscience.

Acamprosate is neuroprotective against glutamate-induced excitotoxicity when enhanced by ethanol withdrawal in neocortical cultures of fetal rat brain.

B. Harris, J. Littleton, M. al Qatari, 2001, Alcoholism, clinical and experimental research.

(−)-nicotine ameliorates corticosterone's potentiation of N-methyl-d-aspartate receptor-mediated cornu ammonis 1 toxicity

P. Mulholland, M. Prendergast, B. Harris, 2004, Neuroscience.

Chronic nicotine exposure reduces N-methyl-d-aspartate receptor-mediated damage in the hippocampus without altering calcium accumulation or extrusion: evidence of calbindin-D28K overexpression

M. Prendergast, B. Harris, R. Holley, 2001, Neuroscience.

Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression.

P. Mulholland, M. Prendergast, B. Harris, 2003, Alcohol.

Hippocampal CA1 region neurodegeneration produced by ethanol withdrawal requires activation of intrinsic polysynaptic hippocampal pathways and function of N-methyl-d-aspartate receptors

M. Prendergast, B. Harris, D. Gibson, 2004, Neuroscience.

Chronic, but not acute, nicotine exposure attenuates ethanol withdrawal-induced hippocampal damage in vitro.

M. Prendergast, B. Harris, J. Littleton, 2000, Alcoholism, clinical and experimental research.

The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca2+ that require function of an N-methyl-d-aspartate receptor polyamine-sensitive site

P. Mulholland, A. Nath, M. Prendergast, 2004, Brain Research.

The neurotoxicity induced by ethanol withdrawal in mature organotypic hippocampal slices might involve cross-talk between metabotropic glutamate type 5 receptors and N-methyl-D-aspartate receptors.

N. W. Pedigo, M. Prendergast, B. Harris, 2003, Alcoholism, clinical and experimental research.

Choline exposure reduces potentiation of N-methyl-D-aspartate toxicity by corticosterone in the developing hippocampus.

P. Mulholland, M. Prendergast, B. Harris, 2004, Brain research. Developmental brain research.

Acamprosate has no effect on NMDA-induced toxicity but reduces toxicity induced by spermidine or by changing the medium in organotypic hippocampal slice cultures from rat.

M. Prendergast, B. Harris, D. Gibson, 2002, Alcoholism, clinical and experimental research.

Acamprosate is neuroprotective against glutamate-induced excitotoxicity when enhanced by ethanol withdrawal in neocortical cultures of fetal rat brain.

B. Harris, J. Littleton, M. al Qatari, 2001, Alcoholism, clinical and experimental research.

Corticosterone increases damage and cytosolic calcium accumulation associated with ethanol withdrawal in rat hippocampal slice cultures.

P. Mulholland, M. Prendergast, B. Harris, 2005, Alcoholism, clinical and experimental research.

Cytotoxic effects of exposure to the human immunodeficiency virus type 1 protein Tat in the hippocampus are enhanced by prior ethanol treatment.

Avindra Nath, P. Mulholland, A. Nath, 2004, Alcoholism, clinical and experimental research.

Polyamines contribute to ethanol withdrawal-induced neurotoxicity in rat hippocampal slice cultures through interactions with the NMDA receptor.

N. W. Pedigo, M. Prendergast, B. Harris, 2003, Alcoholism, clinical and experimental research.

In vitro effects of ethanol withdrawal and spermidine on viability of hippocampus from male and female rat.

M. Prendergast, B. Harris, D. Gibson, 2000, Alcoholism, clinical and experimental research.

Radioligand binding studies reveal agmatine is a more selective antagonist for a polyamine-site on the NMDA receptor than arcaine or ifenprodil

B. Harris, D. Gibson, D. Rogers, 2002, Brain Research.

Nicotine exposure reduces N-methyl-D-aspartate toxicity in the hippocampus: relation to distribution of the alpha7 nicotinic acetylcholine receptor subunit.

J. R. Pauly, M. Prendergast, B. Harris, 2001, Medical science monitor : international medical journal of experimental and clinical research.