C. Thaw

发表

Hydrogen bonding interaction of thyrotropin-releasing hormone (TRH) with transmembrane tyrosine 106 of the TRH receptor.

R. Osman, M. Gershengorn, C. Bowers, 1994, The Journal of biological chemistry.

Benzodiazepines modulate voltage-sensitive calcium channels in GH3 pituitary cells at sites distinct from thyrotropin-releasing hormone receptors.

E. Geras-Raaka, M. Gershengorn, C. Thaw, 1988, Endocrinology.

Acetylcholine inhibits the release of somatostatin from rat hypothalamus in vitro.

C. Thaw, C. Hollander, S. Richardson, 1980, Endocrinology.

Human calcitonin receptor is directly targeted to and retained in the basolateral surface of MDCK cells.

C. Thaw, D. Nussenzveig, M. Matos, 1998, The American journal of physiology.

Human calcitonin receptor is directly targeted to and retained in the basolateral surface of MDCK cells.

C. Thaw, D. Nussenzveig, M. Matos, 1998, American journal of physiology. Cell physiology.

Human calcitonin receptors exhibit agonist-independent (constitutive) signaling activity.

M. Gershengorn, D. P. Cohen, C. Thaw, 1997, Endocrinology.

Thyrotropin (TSH)-releasing hormone decreases phosphatidylinositol and increases unesterified arachidonic acid in thyrotropic cells: possible early events in stimulation of TSH secretion.

R. Kolesnick, M. Gershengorn, C. Thaw, 1984, Endocrinology.

Tetracaine, propranolol and trifluoperazine inhibit thyrotropin releasing hormone-induced prolactin secretion from GH3 cells by displacing membrane calcium: further evidence that TRH acts to mobilize cellular calcium.

M. Gershengorn, S. Wittlin, C. Thaw, 1982, Endocrinology.

Thyrotropin-releasing hormone (TRH) stimulates biphasic elevation of cytoplasmic free calcium in GH3 cells. Further evidence that TRH mobilizes cellular and extracellular Ca2+.

M. Gershengorn, C. Thaw, Marvin C. Gershengorn, 1985, Endocrinology.

Characterization of the calcium response to thyrotropin-releasing hormone (TRH) in cells transfected with TRH receptor complementary DNA: importance of voltage-sensitive calcium channels.

M. Gershengorn, G. Sempowski, P. M. Hinkle, 1992, Molecular endocrinology.

Calcium influx is not required for TRH to elevate free cytoplasmic calcium in GH3 cells.

M. Gershengorn, C. Thaw, Marvin C. Gershengorn, 1983, Endocrinology.

Inhibition of inositol phosphate second messenger formation by intracellular loop one of a human calcitonin receptor. Expression and mutational analysis of synthetic receptor genes.

M. Gershengorn, C. Thaw, D. Nussenzveig, 1994, The Journal of biological chemistry.

TRH mobilizes membrane calcium in thyrotropic cells as monitored by chlortetracycline.

M. Gershengorn, C. Thaw, 1982, American Journal of Physiology.

Thyrotropin-Releasing Hormone (TRH) Stimulates Biphasic Elevation of Cytoplasmic Free Calcium in GH 3 Cells. Further Evidence that TRH Mobilizes Cellular and

C. Thaw, Marvin C. GERSHENGORNt, 1985 .

Regulation of thyrotropin-releasing hormone receptors is cell type specific: comparison of endogenous pituitary receptors and receptors transfected into non-pituitary cells.

M. Gershengorn, C. Thaw, 1991, Endocrinology.

Evidence that cobalt ion inhibition of prolactin secretion occurs at an intracellular locus.

M. Gershengorn, C. Thaw, E. G. Raaka, 1984, The American journal of physiology.

Intrarenal volumes of distribution and relative diffusion coefficients of monohydric alcohols.

W. Perl, F. Chinard, C. Thaw, 1969, Circulation research.

Calcium influx is not required for thyrotropin-releasing hormone stimulation of prolactin release from GH3 cells.

C. Thaw, R. Gerry, Marvin C. Gershengorn, 1983, Cell calcium.

Arachidonic acid mobilizes calcium and stimulates prolactin secretion from GH3 cells.

R. Kolesnick, M. Gershengorn, C. Thaw, 1984, The American journal of physiology.