J. Ratelade

发表

Involvement of antibody-dependent cell-mediated cytotoxicity in inflammatory demyelination in a mouse model of neuromyelitis optica

A. Verkman, J. Bennett, A. Ritchie, 2013, Acta Neuropathologica.

Experimental mouse model of optic neuritis with inflammatory demyelination produced by passive transfer of neuromyelitis optica-immunoglobulin G

M. Papadopoulos, A. Verkman, J. Bennett, 2014, Journal of Neuroinflammation.

A murine model of Denys-Drash syndrome reveals novel transcriptional targets of WT1 in podocytes.

N. Hastie, C. Antignac, M. Pontoglio, 2010, Human molecular genetics.

Severity of arterial defects in the retina correlates with the burden of intracerebral haemorrhage in COL4A1‐related stroke

A. Joutel, Valérie Domenga-Denier, E. Plaisier, 2018, The Journal of pathology.

Intravenous Neuromyelitis Optica Autoantibody in Mice Targets Aquaporin-4 in Peripheral Organs and Area Postrema

A. Verkman, J. Bennett, J. Ratelade, 2011, PloS one.

Podocin inactivation in mature kidneys causes focal segmental glomerulosclerosis and nephrotic syndrome.

M. Dallman, G. Mollet, C. Antignac, 2009, Journal of the American Society of Nephrology : JASN.

Neuroprotective effect of aquaporin-4 deficiency in a mouse model of severe global cerebral ischemia produced by transient 4-vessel occlusion

A. Verkman, G. Akdemir, J. Ratelade, 2014, Neuroscience Letters.

Complement-dependent Cytotoxicity in Neuromyelitis Optica Requires Aquaporin-4 Protein Assembly in Orthogonal Arrays*

A. Verkman, J. Ratelade, Lukmanee Tradtrantip, 2012, The Journal of Biological Chemistry.

Transient hyperckemia in the setting of neuromyelitis optica (NMO)

A. Verkman, A. Bollen, B. Cree, 2014, Muscle & nerve.

Notch3ECD immunotherapy improves cerebrovascular responses in CADASIL mice

J. T. Pedersen, A. Joutel, C. Capone, 2018, Annals of neurology.

A missense mutation in podocin leads to early and severe renal disease in mice.

C. Antignac, C. Antignac, M. Gubler, 2008, Kidney international.

Reducing Hypermuscularization of the Transitional Segment Between Arterioles and Capillaries Protects Against Spontaneous Intracerebral Hemorrhage

Jean-Frédéric Gerbeau, D. Lombardi, Colin Smith, 2020, Circulation.

Aggregation state determines the localization and function of M1– and M23–aquaporin-4 in astrocytes

Alex J. Smith, A. Verkman, Byung-Ju Jin, 2014, The Journal of cell biology.

Greatly improved survival and neuroprotection in aquaporin‐4‐knockout mice following global cerebral ischemia

A S Verkman, A. Verkman, A. Verkman, 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Enzymatic deglycosylation converts pathogenic neuromyelitis optica anti–aquaporin‐4 immunoglobulin G into therapeutic antibody

A. Verkman, J. Ratelade, Lukmanee Tradtrantip, 2013, Annals of neurology.

Neuromyelitis optica IgG does not alter aquaporin‐4 water permeability, plasma membrane M1/M23 isoform content, or supramolecular assembly

M. Papadopoulos, A. Verkman, J. Bennett, 2012, Glia.

Super-resolution imaging of aquaporin-4 orthogonal arrays of particles in cell membranes

A. Verkman, T. Moritz, J. Ratelade, 2012, Journal of Cell Science.

Neuromyelitis optica: aquaporin-4 based pathogenesis mechanisms and new therapies.

A. Verkman, J. Ratelade, 2012, The international journal of biochemistry & cell biology.

Neuromyelitis optica IgG and natural killer cells produce NMO lesions in mice without myelin loss

M. Papadopoulos, A. Verkman, S. Saadoun, 2012, Acta Neuropathologica.

Aquaporin-4: orthogonal array assembly, CNS functions, and role in neuromyelitis optica

A. Verkman, J. Ratelade, Lukmanee Tradtrantip, 2011, Acta Pharmacologica Sinica.

Blood brain barrier leakage is not a consistent feature of white matter lesions in CADASIL

Rikesh M. Rajani, R. Kalaria, H. Kalimo, 2019, Acta Neuropathologica Communications.

Perturbations of the cerebrovascular matrisome: A convergent mechanism in small vessel disease of the brain?

M. Nelson, A. Joutel, J. Ratelade, 2016, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

Maternal environment interacts with modifier genes to influence progression of nephrotic syndrome.

M. Kretzler, G. Mollet, C. Antignac, 2008, Journal of the American Society of Nephrology : JASN.

Evidence against Cellular Internalization in Vivo of NMO-IgG, Aquaporin-4, and Excitatory Amino Acid Transporter 2 in Neuromyelitis Optica*

A. Verkman, J. Bennett, J. Ratelade, 2011, The Journal of Biological Chemistry.

Blood brain barrier leakage is not a consistent feature of white matter lesions in CADASIL

Rikesh M. Rajani, R. Kalaria, H. Kalimo, 2019, Acta Neuropathologica Communications.

Inhibitor(s) of the classical complement pathway in mouse serum limit the utility of mice as experimental models of neuromyelitis optica.

A. Verkman, J. Ratelade, A. S. Verkman, 2014, Molecular immunology.

Unique neuromyelitis optica pathology produced in naïve rats by intracerebral administration of NMO-IgG

A. Verkman, J. Ratelade, Nithi Asavapanumas, 2014, Acta Neuropathologica.

Human immunoglobulin G reduces the pathogenicity of aquaporin-4 autoantibodies in neuromyelitis optica

Alex J. Smith, A. Verkman, J. Ratelade, 2014, Experimental Neurology.

Consequences of NMO-IgG binding to aquaporin-4 in neuromyelitis optica

M. Papadopoulos, A. Verkman, J. Bennett, 2012, Proceedings of the National Academy of Sciences.

Production of Recombinant Proteins in the lon-Deficient BL21(DE3) Strain of Escherichia coli in the Absence of the DnaK Chaperone

J. Betton, P. Mazodier, N. Benaroudj, 2009, Applied and Environmental Microbiology.

Production of Recombinant Proteins in the lon -Deﬁcient BL21(DE3) Strain of Escherichia coli in the Absence of the DnaK Chaperone (cid:1) †

J. Betton, P. Mazodier, N. Benaroudj, 2009 .

Experimental mouse model of optic neuritis with inflammatory demyelination produced by passive transfer of neuromyelitis optica-immunoglobulin G

M. Papadopoulos, A. Verkman, J. Bennett, 2014, Journal of Neuroinflammation.

Enzymatic deglycosylation converts pathogenic neuromyelitis optica anti–aquaporin‐4 immunoglobulin G into therapeutic antibody

A. Verkman, J. Ratelade, Lukmanee Tradtrantip, 2013, Annals of neurology.

Podocin inactivation in mature kidneys causes focal segmental glomerulosclerosis and nephrotic syndrome.

M. Dallman, G. Mollet, C. Antignac, 2009, Journal of the American Society of Nephrology : JASN.

Neuromyelitis optica IgG does not alter aquaporin‐4 water permeability, plasma membrane M1/M23 isoform content, or supramolecular assembly

A. Verkman, J. Ratelade, A. Rossi, 2012, Glia.

Neuromyelitis optica: aquaporin-4 based pathogenesis mechanisms and new therapies.

A. Verkman, J. Ratelade, 2012, The international journal of biochemistry & cell biology.